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Targeting DNA damage and repair: embracing the pharmacological era for successful cancer therapy

Pharmacology & therapeutics (Oxford), 2012-03, Vol.133 (3), p.334-350

2012

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Activating Akt1 mutations alter DNA double strand break repair and radiosensitivity

Scientific reports, 2017-02, Vol.7 (1), p.42700-42700, Article 42700

2017

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Ultra-short laser-accelerated proton pulses have similar DNA-damaging effectiveness but produce less immediate nitroxidative stress than conventional proton beams

Scientific reports, 2016-08, Vol.6 (1), p.32441-32441, Article 32441

2016

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Linear Non-Modal Growth of Planar Perturbations in a Layered Couette Flow

Fluids (Basel), 2021-12, Vol.6 (12), p.442

2021

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Mechanisms of DNA double strand break repair and chromosome aberration formation

Cytogenetic and Genome Research, 2004-01, Vol.104 (1-4), p.14-20

2004

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An experimental platform for studying the radiation effects of laser accelerated protons on mammalian cells

AIP advances, 2021-06, Vol.11 (6), p.065208-065208-9

2021

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DNA double-strand-break complexity levels and their possible contributions to the probability for error-prone processing and repair pathway choice

Nucleic acids research, 2013-09, Vol.41 (16), p.7589-7605

2013

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Histone H1 functions as a stimulatory factor in backup pathways of NHEJ

Nucleic acids research, 2008-03, Vol.36 (5), p.1610-1623

2008

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Protein kinase Bα (AKT1) plays a role in the cellular response to ionizing radiation

European journal of cancer (1990), 2016-07, Vol.61, p.S159-S159

2016

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Cell cycle regulation in irradiated and nonirradiated cells

Seminars in oncology, 1997-12, Vol.24 (6), p.602-615

Iliakis, G

1997

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Results of a Pilot Study Involving the Use of an Antisense Oligodeoxynucleotide Directed Against the Insulin-Like Growth Factor Type I Receptor in Malignant Astrocytomas

Journal of clinical oncology, 2001-04, Vol.19 (8), p.2189-2200

2001

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Robust Radiosensitization by Combined Treatment of Cancer Cells with Talazoparib and Polθ Inhibitors

International journal of radiation oncology, biology, physics, 2023-10, Vol.117 (2), p.e245-e245

2023

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Homologous recombination as a potential target for caffeine radiosensitization in mammalian cells: reduced caffeine radiosensitization in XRCC2 and XRCC3 mutants

Oncogene, 2000-11, Vol.19 (50), p.5788-5800

2000

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Break-Induced Replication Repair of Damaged Forks Induces Genomic Duplications in Human Cells

Science (American Association for the Advancement of Science), 2014-01, Vol.343 (6166), p.88-91

2014

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28 What is new in DNA repair of radiation damage?

European journal of cancer supplements, 2009, Vol.7 (2), p.10-10

Iliakis, G

2009

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PO-133 Inhibition of HIF-1α enhances radiation sensitivity in tumour cells and mediates tumour growth decrease in mice

ESMO open, 2018-07, Vol.3 (Suppl 2), p.A278-A278

2018

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Investigation of Bystander Effects in Hybrid Cells by Means of Cell Fusion and Premature Chromosome Condensation Induction

Radiation research, 2010-06, Vol.173 (6), p.789-801

2010

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The Radioresistance to Killing of A1–5 Cells Derives from Activation of the Chk1 Pathway

The Journal of biological chemistry, 2001-05, Vol.276 (21), p.17693-17698

2001

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SP-0841 DSB complexity: A key to cell lethality

Radiotherapy and oncology, 2022-05, Vol.170, p.S754-S754

2022

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Session 1: DNA repair

Journal of radiation research, 2014-03, Vol.55 (suppl_1), p.i9-i11

Iliakis, George

2014

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DNA damage checkpoint control in cells exposed to ionizing radiation

Oncogene, 2003-09, Vol.22 (37), p.5834-5847

2003

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H2AX in recognition and signaling of DNA double-strand breaks in the context of chromatin

Nucleic acids research, 2008-08, Vol.36 (17), p.5678-5694

2008

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An ATM-independent S-phase checkpoint response involves CHK1 pathway

Cancer research (Chicago, Ill.), 2002-03, Vol.62 (6), p.1598-1603

2002

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DNA ligase III as a candidate component of backup pathways of nonhomologous end joining

Cancer research (Chicago, Ill.), 2005-05, Vol.65 (10), p.4020-4030

2005

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Ku affects the ataxia and rad 3-related/CHK1-dependent S phase checkpoint response after camptothecin treatment

Cancer research (Chicago, Ill.), 2002-05, Vol.62 (9), p.2483-2487

2002

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Targeting DNA damage and repair: embracing the pharmacological era for successful cancer therapy

Activating Akt1 mutations alter DNA double strand break repair and radiosensitivity

Ultra-short laser-accelerated proton pulses have similar DNA-damaging effectiveness but produce less immediate nitroxidative stress than conventional proton beams

Linear Non-Modal Growth of Planar Perturbations in a Layered Couette Flow

Mechanisms of DNA double strand break repair and chromosome aberration formation

An experimental platform for studying the radiation effects of laser accelerated protons on mammalian cells

DNA double-strand-break complexity levels and their possible contributions to the probability for error-prone processing and repair pathway choice

Histone H1 functions as a stimulatory factor in backup pathways of NHEJ

Protein kinase Bα (AKT1) plays a role in the cellular response to ionizing radiation

Cell cycle regulation in irradiated and nonirradiated cells

Results of a Pilot Study Involving the Use of an Antisense Oligodeoxynucleotide Directed Against the Insulin-Like Growth Factor Type I Receptor in Malignant Astrocytomas

Robust Radiosensitization by Combined Treatment of Cancer Cells with Talazoparib and Polθ Inhibitors

Homologous recombination as a potential target for caffeine radiosensitization in mammalian cells: reduced caffeine radiosensitization in XRCC2 and XRCC3 mutants

Break-Induced Replication Repair of Damaged Forks Induces Genomic Duplications in Human Cells

28 What is new in DNA repair of radiation damage?

PO-133 Inhibition of HIF-1α enhances radiation sensitivity in tumour cells and mediates tumour growth decrease in mice

Investigation of Bystander Effects in Hybrid Cells by Means of Cell Fusion and Premature Chromosome Condensation Induction

The Radioresistance to Killing of A1–5 Cells Derives from Activation of the Chk1 Pathway

SP-0841 DSB complexity: A key to cell lethality

Session 1: DNA repair

DNA damage checkpoint control in cells exposed to ionizing radiation

H2AX in recognition and signaling of DNA double-strand breaks in the context of chromatin

An ATM-independent S-phase checkpoint response involves CHK1 pathway

DNA ligase III as a candidate component of backup pathways of nonhomologous end joining

Ku affects the ataxia and rad 3-related/CHK1-dependent S phase checkpoint response after camptothecin treatment

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