UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Altered cardiac electrophysiology and SUDEP in a model of Dravet syndrome

PloS one, 2013-10, Vol.8 (10), p.e77843

2013

Volltextzugriff (PDF)

Na Channel β Subunits: Overachievers of the Ion Channel Family

Frontiers in pharmacology, 2011-01, Vol.2, p.53-53

2011

Volltextzugriff (PDF)

More than a pore: ion channel signaling complexes

The Journal of neuroscience, 2014-11, Vol.34 (46), p.15159-15169

2014

Volltextzugriff (PDF)

Voltage-Gated Sodium Channel β1/β1B Subunits Regulate Cardiac Physiology and Pathophysiology

Frontiers in physiology, 2018-04, Vol.9, p.351-351

2018

Volltextzugriff (PDF)

Loss of Plakophilin-2 Expression Leads to Decreased Sodium Current and Slower Conduction Velocity in Cultured Cardiac Myocytes

Circulation research, 2009-09, Vol.105 (6), p.523-526

2009

Volltextzugriff (PDF)

Therapeutic potential for phenytoin: targeting Nav1.5 sodium channels to reduce migration and invasion in metastatic breast cancer

Breast cancer research and treatment, 2012-07, Vol.134 (2), p.603-615

2012

Volltextzugriff (PDF)

DSCAM gene triplication causes excessive GABAergic synapses in the neocortex in Down syndrome mouse models

PLoS biology, 2023-04, Vol.21 (4), p.e3002078-e3002078

2023

Volltextzugriff (PDF)

Dravet syndrome patient-derived neurons suggest a novel epilepsy mechanism

Annals of neurology, 2013-07, Vol.74 (1), p.128-139

2013

Volltextzugriff (PDF)

Heterogeneity of voltage gated sodium current density between neurons decorrelates spiking and suppresses network synchronization in Scn1b null mouse models

Scientific reports, 2023-06, Vol.13 (1), p.8887-8887, Article 8887

2023

Volltextzugriff (PDF)

Biocatalytic Detoxification of Paralytic Shellfish Toxins

ACS chemical biology, 2019-05, Vol.14 (5), p.941-948

2019

Volltextzugriff (PDF)

The adhesion function of the sodium channel beta subunit (β1) contributes to cardiac action potential propagation

eLife, 2018-08, Vol.7

2018

Volltextzugriff (PDF)

Dravet Syndrome: A Developmental and Epileptic Encephalopathy

Epilepsy currents, 2019-01, Vol.19 (1), p.51-53

2019

Volltextzugriff (PDF)

Delayed maturation of GABAergic signaling in the Scn1a and Scn1b mouse models of Dravet Syndrome

Scientific reports, 2019-04, Vol.9 (1), p.6210-6210, Article 6210

2019

Volltextzugriff (PDF)

SUMOylation of Neuronal K+ Channels: A Potential Therapeutic Pathway for Epilepsy and SUDEP?

Neuron (Cambridge, Mass.), 2014-09, Vol.83 (5), p.996-998

2014

Volltextzugriff (PDF)

Physiologic biomechanics enhance reproducible contractile development in a stem cell derived cardiac muscle platform

Nature communications, 2021-10, Vol.12 (1), p.6167-6167, Article 6167

2021

Volltextzugriff (PDF)

Input-Output Relationship of CA1 Pyramidal Neurons Reveals Intact Homeostatic Mechanisms in a Mouse Model of Fragile X Syndrome

Cell reports (Cambridge), 2020-08, Vol.32 (6), p.107988-107988, Article 107988

2020

Volltextzugriff (PDF)

Mitochondrial oxidative stress contributes to diastolic dysfunction through impaired mitochondrial dynamics

Redox biology, 2022-11, Vol.57, p.102474-102474, Article 102474

2022

Volltextzugriff (PDF)

Abnormal neuronal patterning occurs during early postnatal brain development of Scn1b-null mice and precedes hyperexcitability

Proceedings of the National Academy of Sciences - PNAS, 2013-01, Vol.110 (3), p.1089-1094

2013

Volltextzugriff (PDF)

Subcellular heterogeneity of sodium current properties in adult cardiac ventricular myocytes

Heart rhythm, 2011-12, Vol.8 (12), p.1923-1930

2011

Volltextzugriff (PDF)

The sodium channel accessory subunit Navβ1 regulates neuronal excitability through modulation of repolarizing voltage-gated K⁺ channels

The Journal of neuroscience, 2012-04, Vol.32 (17), p.5716-5727

2012

Volltextzugriff (PDF)

Sodium Channel β Subunits: Emerging Targets in Channelopathies

Annual review of physiology, 2015-01, Vol.77 (1), p.481-504

2015

Volltextzugriff (PDF)

Late Na(+) current and protracted electrical recovery are critical determinants of the aging myopathy

Nature communications, 2015-11, Vol.6 (1), p.8803-8803, Article 8803

2015

Volltextzugriff (PDF)

Voltage-gated Na+ channel β1B: a secreted cell adhesion molecule involved in human epilepsy

The Journal of neuroscience, 2011-10, Vol.31 (41), p.14577-14591

2011

Volltextzugriff (PDF)

Early Painful Diabetic Neuropathy Is Associated with Differential Changes in Tetrodotoxin-sensitive and -resistant Sodium Channels in Dorsal Root Ganglion Neurons in the Rat

The Journal of biological chemistry, 2004-07, Vol.279 (28), p.29341-29350

2004

Volltextzugriff (PDF)

Antisense oligonucleotides increase Scn1a expression and reduce seizures and SUDEP incidence in a mouse model of Dravet syndrome

Science translational medicine, 2020-08, Vol.12 (558)

2020

Volltextzugriff (PDF)

1 – 25 von 237

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(225)
Mit Volltextzugriff
(221)

Publikationsform

Sprache

Englisch
(237)
Japanisch
(38)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 30 Tagen
(1)

Menü

Weitere Dienste

Einstellungen

Altered cardiac electrophysiology and SUDEP in a model of Dravet syndrome

Na Channel β Subunits: Overachievers of the Ion Channel Family

More than a pore: ion channel signaling complexes

Voltage-Gated Sodium Channel β1/β1B Subunits Regulate Cardiac Physiology and Pathophysiology

Loss of Plakophilin-2 Expression Leads to Decreased Sodium Current and Slower Conduction Velocity in Cultured Cardiac Myocytes

Therapeutic potential for phenytoin: targeting Nav1.5 sodium channels to reduce migration and invasion in metastatic breast cancer

DSCAM gene triplication causes excessive GABAergic synapses in the neocortex in Down syndrome mouse models

Dravet syndrome patient-derived neurons suggest a novel epilepsy mechanism

Heterogeneity of voltage gated sodium current density between neurons decorrelates spiking and suppresses network synchronization in Scn1b null mouse models

Biocatalytic Detoxification of Paralytic Shellfish Toxins

The adhesion function of the sodium channel beta subunit (β1) contributes to cardiac action potential propagation

Dravet Syndrome: A Developmental and Epileptic Encephalopathy

Delayed maturation of GABAergic signaling in the Scn1a and Scn1b mouse models of Dravet Syndrome

SUMOylation of Neuronal K+ Channels: A Potential Therapeutic Pathway for Epilepsy and SUDEP?

Physiologic biomechanics enhance reproducible contractile development in a stem cell derived cardiac muscle platform

Input-Output Relationship of CA1 Pyramidal Neurons Reveals Intact Homeostatic Mechanisms in a Mouse Model of Fragile X Syndrome

Mitochondrial oxidative stress contributes to diastolic dysfunction through impaired mitochondrial dynamics

Abnormal neuronal patterning occurs during early postnatal brain development of Scn1b-null mice and precedes hyperexcitability

Subcellular heterogeneity of sodium current properties in adult cardiac ventricular myocytes

The sodium channel accessory subunit Navβ1 regulates neuronal excitability through modulation of repolarizing voltage-gated K⁺ channels

Sodium Channel β Subunits: Emerging Targets in Channelopathies

Late Na(+) current and protracted electrical recovery are critical determinants of the aging myopathy

Voltage-gated Na+ channel β1B: a secreted cell adhesion molecule involved in human epilepsy

Early Painful Diabetic Neuropathy Is Associated with Differential Changes in Tetrodotoxin-sensitive and -resistant Sodium Channels in Dorsal Root Ganglion Neurons in the Rat

Antisense oligonucleotides increase Scn1a expression and reduce seizures and SUDEP incidence in a mouse model of Dravet syndrome

Suchergebnisse filtern