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Renal cell apoptosis and proliferation may be linked to nuclear factor– κB activation and expression of inducible nitric oxide synthase in patients with lupus nephritis

Human pathology, 2006-06, Vol.37 (6), p.637-647

2006

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CRM1-mediated Nuclear Export Is Required for 26 S Proteasome-dependent Degradation of the TRIP-Br2 Proto-oncoprotein

The Journal of biological chemistry, 2008-04, Vol.283 (17), p.11661-11676

2008

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Racial and Genetic Factors in IgA Nephropathy

Seminars in nephrology, 2008, Vol.28 (1), p.48-57

Hsu, Stephen I.-Hong, MD, PhD

2008

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Ablation of TRIP-Br2, a regulator of fat lipolysis, thermogenesis and oxidative metabolism, prevents diet-induced obesity and insulin resistance

Nature medicine, 2013-02, Vol.19 (2), p.217-226

2013

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Exploiting the TRIP-Br family of cell cycle regulatory proteins as chemotherapeutic drug targets in human cancer

Cancer biology & therapy, 2007-05, Vol.6 (5), p.712-718

2007

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Risk factors for proteinuria in a large, multiracial, southeast Asian population

Journal of the American Society of Nephrology, 2002-07, Vol.13 (7), p.1907-1917

2002

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In situ glomerular expression of activated NF-κB in human lupus nephritis and other non-proliferative proteinuric glomerulopathy

Virchows Archiv : an international journal of pathology, 2006-02, Vol.448 (2), p.172-183

2006

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Evidence for genetic factors in the development and progression of IgA nephropathy

Kidney international, 2000-05, Vol.57 (5), p.1818-1835

2000

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TRIP-Br: a novel family of PHD zinc finger- and bromodomain-interacting proteins that regulate the transcriptional activity of E2F-1/DP-1

The EMBO journal, 2001-05, Vol.20 (9), p.2273-2285

2001

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Downregulation of Bcl-2 by podocytes is associated with progressive glomerular injury and clinical indices of poor renal prognosis in human IgA nephropathy

Journal of the American Society of Nephrology, 2004, Vol.15 (1), p.79-90

2004

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Towards the application of proteomics in renal disease diagnosis

Clinical science (1979), 2005-11, Vol.109 (5), p.421-430

2005

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The CtBP2 co-repressor is regulated by NADH-dependent dimerization and possesses a novel N-terminal repression domain

Nucleic acids research, 2004, Vol.32 (5), p.1836-1847

2004

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Biopsy‐Proved Acute Tubulointerstitial Nephritis and Toxic Epidermal Necrolysis Associated with Vancomycin

Pharmacotherapy, 2001-10, Vol.21 (10), p.1233-1239

Hsu, Stephen I‐Hong

2001

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The TRIP-Br Family of Transcriptional Regulators is Essential for the Execution of Cyclin E-Mediated Cell Cycle Progression

Cell cycle (Georgetown, Tex.), 2006-05, Vol.5 (10), p.1111-1115

2006

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The human papillomavirus type 11 and 16 E6 proteins modulate the cell-cycle regulator and transcription cofactor TRIP-Br1

Virology (New York, N.Y.), 2003-12, Vol.317 (1), p.155-164

2003

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Low body weight is a risk factor for proteinuria in multiracial Southeast Asian pediatric population

American journal of kidney diseases, 2001-11, Vol.38 (5), p.1045-1054

2001

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TRIP-Br Links E2F to Novel Functions in the Regulation of Cyclin E Expression During Cell Cycle Progression and in the Maintenance of Genomic Stability

Cell cycle (Georgetown, Tex.), 2004-10, Vol.3 (10), p.1296-1304

2004

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When One Plus One Equals 10: Unique Rewards and Daunting Challenges of Being an MD‐PhD Clinician‐Scientist

The FASEB journal, 2012-04, Vol.26 (S1)

Hsu, Stephen I-Hong

2012

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Pathogenic Role of NF-κB Activation in Tubulointerstitial Inflammatory Lesions in Human Lupus Nephritis

The journal of histochemistry and cytochemistry, 2008-05, Vol.56 (5), p.517-529

2008

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Derailing the UPS of Protein Turnover in Cancer and other Human Diseases

Journal of Cancer Research, 2013-08, Vol.2013 (2013), p.1-11

2013

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Chronic progression of tubulointerstitial damage in proteinuric renal disease is mediated by complement activation: A therapeutic role for complement inhibitors?

Journal of the American Society of Nephrology, 2003-07, Vol.14 (7 Suppl 2), p.S186-S191

2003

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Abstract A48: Cost-effective topical delivery of chemotherapy to treat cervical dysplasia and prevent progression to HPV-associated cervical cancer in the low-resource setting

Cancer epidemiology, biomarkers & prevention, 2011-09, Vol.20 (10_Supplement), p.A48-A48

2011

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79: Idiopathic Nodular Glomerulosclerosis in a Non-Diabetic Young Caucasian Male With Metabolic Syndrome: A Case Report

American journal of kidney diseases, 2010, Vol.55 (4), p.B51-B51

2010

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NGF inhibits human leukemia proliferation by downregulating cyclin A1 expression through promoting acinus CtBP2 association

Oncogene, 2009-10, Vol.28 (43), p.3825-3836

2009

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Ablation of TRIP-Br2, a novel regulator of fat lipolysis, thermogenesis and oxidative metabolism, prevents diet-induced obesity and insulin resistance

Nature medicine, 2013-01, Vol.19 (2), p.217-226

2013

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Renal cell apoptosis and proliferation may be linked to nuclear factor– κB activation and expression of inducible nitric oxide synthase in patients with lupus nephritis

CRM1-mediated Nuclear Export Is Required for 26 S Proteasome-dependent Degradation of the TRIP-Br2 Proto-oncoprotein

Racial and Genetic Factors in IgA Nephropathy

Ablation of TRIP-Br2, a regulator of fat lipolysis, thermogenesis and oxidative metabolism, prevents diet-induced obesity and insulin resistance

Exploiting the TRIP-Br family of cell cycle regulatory proteins as chemotherapeutic drug targets in human cancer

Risk factors for proteinuria in a large, multiracial, southeast Asian population

In situ glomerular expression of activated NF-κB in human lupus nephritis and other non-proliferative proteinuric glomerulopathy

Evidence for genetic factors in the development and progression of IgA nephropathy

TRIP-Br: a novel family of PHD zinc finger- and bromodomain-interacting proteins that regulate the transcriptional activity of E2F-1/DP-1

Downregulation of Bcl-2 by podocytes is associated with progressive glomerular injury and clinical indices of poor renal prognosis in human IgA nephropathy

Towards the application of proteomics in renal disease diagnosis

The CtBP2 co-repressor is regulated by NADH-dependent dimerization and possesses a novel N-terminal repression domain

Biopsy‐Proved Acute Tubulointerstitial Nephritis and Toxic Epidermal Necrolysis Associated with Vancomycin

The TRIP-Br Family of Transcriptional Regulators is Essential for the Execution of Cyclin E-Mediated Cell Cycle Progression

The human papillomavirus type 11 and 16 E6 proteins modulate the cell-cycle regulator and transcription cofactor TRIP-Br1

Low body weight is a risk factor for proteinuria in multiracial Southeast Asian pediatric population

TRIP-Br Links E2F to Novel Functions in the Regulation of Cyclin E Expression During Cell Cycle Progression and in the Maintenance of Genomic Stability

When One Plus One Equals 10: Unique Rewards and Daunting Challenges of Being an MD‐PhD Clinician‐Scientist

Pathogenic Role of NF-κB Activation in Tubulointerstitial Inflammatory Lesions in Human Lupus Nephritis

Derailing the UPS of Protein Turnover in Cancer and other Human Diseases

Chronic progression of tubulointerstitial damage in proteinuric renal disease is mediated by complement activation: A therapeutic role for complement inhibitors?

Abstract A48: Cost-effective topical delivery of chemotherapy to treat cervical dysplasia and prevent progression to HPV-associated cervical cancer in the low-resource setting

79: Idiopathic Nodular Glomerulosclerosis in a Non-Diabetic Young Caucasian Male With Metabolic Syndrome: A Case Report

NGF inhibits human leukemia proliferation by downregulating cyclin A1 expression through promoting acinus CtBP2 association

Ablation of TRIP-Br2, a novel regulator of fat lipolysis, thermogenesis and oxidative metabolism, prevents diet-induced obesity and insulin resistance

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