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Selective inhibitory effects of zinc on cell proliferation in esophageal squamous cell carcinoma through Orai1

The FASEB journal, 2018-01, Vol.32 (1), p.404-416

2018

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Omeprazole blocks STAT6 binding to the eotaxin-3 promoter in eosinophilic esophagitis cells

PloS one, 2012-11, Vol.7 (11), p.e50037

2012

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Gastroesophageal Reflux Might Cause Esophagitis Through a Cytokine-Mediated Mechanism Rather Than Caustic Acid Injury

Gastroenterology (New York, N.Y. 1943), 2009-11, Vol.137 (5), p.1776-1784

2009

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JAK-STAT6 Pathway Inhibitors Block Eotaxin-3 Secretion by Epithelial Cells and Fibroblasts from Esophageal Eosinophilia Patients: Promising Agents to Improve Inflammation and Prevent Fibrosis in EoE

PloS one, 2016-06, Vol.11 (6), p.e0157376-e0157376

2016

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In Barrett's esophagus patients and Barrett's cell lines, ursodeoxycholic acid increases antioxidant expression and prevents DNA damage by bile acids

American journal of physiology: Gastrointestinal and liver physiology, 2014-07, Vol.307 (2), p.G129-G139

2014

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Acid burn or cytokine sizzle in the pathogenesis of heartburn?

Journal of gastroenterology and hepatology, 2013-03, Vol.28 (3), p.385-387

2013

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Cancer-related inflammation and Barrett's carcinogenesis: interleukin-6 and STAT3 mediate apoptotic resistance in transformed Barrett's cells

American journal of physiology: Gastrointestinal and liver physiology, 2011-03, Vol.300 (3), p.G454-G460

2011

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Targeting the intrinsic inflammatory pathway: honokiol exerts proapoptotic effects through STAT3 inhibition in transformed Barrett's cells

American journal of physiology: Gastrointestinal and liver physiology, 2012-09, Vol.303 (5), p.G561-G569

2012

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Important roles of reversible acetylation in the function of hematopoietic transcription factors

Journal of cellular and molecular medicine, 2005-01, Vol.9 (1), p.103-112

2005

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NOD-Like Receptor Protein 3 Inflammasome Priming and Activation in Barrett’s Epithelial Cells

Cellular and molecular gastroenterology and hepatology, 2016-07, Vol.2 (4), p.439-453

2016

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Malignant transformation of non-neoplastic Barrett's epithelial cells through well-defined genetic manipulations

PloS one, 2010-09, Vol.5 (9), p.e13093

2010

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Omeprazole blocks eotaxin-3 expression by oesophageal squamous cells from patients with eosinophilic oesophagitis and GORD

Gut, 2013-06, Vol.62 (6), p.824-832

2013

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Genomic mechanisms of RET inhibitor sensitivity and resistance in RET-fusion positive cancer cells

Journal of clinical oncology, 2023-06, Vol.41 (16_suppl), p.e21029-e21029

2023

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Ablation of EWS-FLI1 by USP9X inhibition suppresses cancer cell growth in Ewing sarcoma

Cancer letters, 2023-01, Vol.552, p.215984, Article 215984

2023

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Association of Acute Gastroesophageal Reflux Disease With Esophageal Histologic Changes

JAMA : the journal of the American Medical Association, 2016-05, Vol.315 (19), p.2104

2016

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Reflux-Induced Inflammation and the Development of Barrett's Esophagus

Digestive diseases and sciences, 2022-05, Vol.55 (10), p.2699

2022

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Discovery of novel cell proliferation-enhancing gene by random siRNA library based combinatorial screening

Combinatorial chemistry & high throughput screening, 2010-11, Vol.13 (9), p.798

2010

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528 – In Barret's Epithelial Cells, Nucleophosmin 1 Facilitates Repair of Oxidative Dna Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair Pathway

Gastroenterology (New York, N.Y. 1943), 2019-05, Vol.156 (6), p.S-107-S-107

2019

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Acidic Bile Salts Induce Epithelial to Mesenchymal Transition via VEGF Signaling in Non-Neoplastic Barrett’s Cells

Gastroenterology (New York, N.Y. 1943), 2019-01, Vol.156 (1), p.130-144.e10

2019

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In Barrett's epithelial cells, weakly acidic bile salt solutions cause oxidative DNA damage with response and repair mediated by p38

American journal of physiology: Gastrointestinal and liver physiology, 2020-03, Vol.318 (3), p.G464-G478

2020

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Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H 2 -Receptor Antagonist

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-260

2018

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Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H2-Receptor Antagonist

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-260-S-260

2018

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Su1054 - in Barrett's Epithelial Cells, Ursodeoxycholic Acid Hastens Repair of Oxidative DNA Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-469-S-469

2018

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Mithramycin suppresses DNA damage repair via targeting androgen receptor in prostate cancer

Cancer letters, 2020-09, Vol.488, p.40-49

2020

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In oesophageal squamous cells exposed to acidic bile salt medium, omeprazole inhibits IL-8 expression through effects on nuclear factor-κB and activator protein-1

Gut, 2014-07, Vol.63 (7), p.1042-1052

2014

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Selective inhibitory effects of zinc on cell proliferation in esophageal squamous cell carcinoma through Orai1

Omeprazole blocks STAT6 binding to the eotaxin-3 promoter in eosinophilic esophagitis cells

Gastroesophageal Reflux Might Cause Esophagitis Through a Cytokine-Mediated Mechanism Rather Than Caustic Acid Injury

JAK-STAT6 Pathway Inhibitors Block Eotaxin-3 Secretion by Epithelial Cells and Fibroblasts from Esophageal Eosinophilia Patients: Promising Agents to Improve Inflammation and Prevent Fibrosis in EoE

In Barrett's esophagus patients and Barrett's cell lines, ursodeoxycholic acid increases antioxidant expression and prevents DNA damage by bile acids

Acid burn or cytokine sizzle in the pathogenesis of heartburn?

Cancer-related inflammation and Barrett's carcinogenesis: interleukin-6 and STAT3 mediate apoptotic resistance in transformed Barrett's cells

Targeting the intrinsic inflammatory pathway: honokiol exerts proapoptotic effects through STAT3 inhibition in transformed Barrett's cells

Important roles of reversible acetylation in the function of hematopoietic transcription factors

NOD-Like Receptor Protein 3 Inflammasome Priming and Activation in Barrett’s Epithelial Cells

Malignant transformation of non-neoplastic Barrett's epithelial cells through well-defined genetic manipulations

Omeprazole blocks eotaxin-3 expression by oesophageal squamous cells from patients with eosinophilic oesophagitis and GORD

Genomic mechanisms of RET inhibitor sensitivity and resistance in RET-fusion positive cancer cells

Ablation of EWS-FLI1 by USP9X inhibition suppresses cancer cell growth in Ewing sarcoma

Association of Acute Gastroesophageal Reflux Disease With Esophageal Histologic Changes

Reflux-Induced Inflammation and the Development of Barrett's Esophagus

Discovery of novel cell proliferation-enhancing gene by random siRNA library based combinatorial screening

528 – In Barret's Epithelial Cells, Nucleophosmin 1 Facilitates Repair of Oxidative Dna Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair Pathway

Acidic Bile Salts Induce Epithelial to Mesenchymal Transition via VEGF Signaling in Non-Neoplastic Barrett’s Cells

In Barrett's epithelial cells, weakly acidic bile salt solutions cause oxidative DNA damage with response and repair mediated by p38

Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H 2 -Receptor Antagonist

Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H2-Receptor Antagonist

Su1054 - in Barrett's Epithelial Cells, Ursodeoxycholic Acid Hastens Repair of Oxidative DNA Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair

Mithramycin suppresses DNA damage repair via targeting androgen receptor in prostate cancer

In oesophageal squamous cells exposed to acidic bile salt medium, omeprazole inhibits IL-8 expression through effects on nuclear factor-κB and activator protein-1

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