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Gefitinib-induced killing of NSCLC cell lines expressing mutant EGFR requires BIM and can be enhanced by BH3 mimetics

PLoS medicine, 2007-10, Vol.4 (10), p.1681-89; discussion 1690

2007

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Substantial Susceptibility of Chronic Lymphocytic Leukemia to BCL2 Inhibition: Results of a Phase I Study of Navitoclax in Patients With Relapsed or Refractory Disease

Journal of clinical oncology, 2012-02, Vol.30 (5), p.488-496

2012

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XIAP discriminates between type I and type II FAS-induced apoptosis

Nature (London), 2009-08, Vol.460 (7258), p.1035-1039

2009

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PRMT1-mediated H4R3me2a recruits SMARCA4 to promote colorectal cancer progression by enhancing EGFR signaling

Genome medicine, 2021-04, Vol.13 (1), p.58-58, Article 58

2021

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Sensitization of BCL-2–expressing breast tumors to chemotherapy by the BH3 mimetic ABT-737

Proceedings of the National Academy of Sciences - PNAS, 2012-02, Vol.109 (8), p.2766-2771

2012

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Replication stress induces mitotic death through parallel pathways regulated by WAPL and telomere deprotection

Nature communications, 2019-09, Vol.10 (1), p.4224-15, Article 4224

2019

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Key residues in the VDAC2-BAK complex can be targeted to modulate apoptosis

PLoS biology, 2024-05, Vol.22 (5), p.e3002617

2024

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Apoptosis Initiated When BH3 Ligands Engage Multiple Bcl-2 Homologs, Not Bax or Bak

Science (American Association for the Advancement of Science), 2007-02, Vol.315 (5813), p.856-859

2007

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Current Advances and Future Strategies for BCL-2 Inhibitors: Potent Weapons against Cancers

Cancers, 2023-10, Vol.15 (20), p.4957

2023

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Two distinct pathways regulate platelet phosphatidylserine exposure and procoagulant function

Blood, 2009-07, Vol.114 (3), p.663-666

2009

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BCL2 and MCL1 inhibitors for hematologic malignancies

Blood, 2021-09, Vol.138 (13), p.1120-1136

2021

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Proapoptotic Bcl-2 Relative Bim Required for Certain Apoptotic Responses, Leukocyte Homeostasis, and to Preclude Autoimmunity

Science (American Association for the Advancement of Science), 1999-11, Vol.286 (5445), p.1735-1738

1999

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Structural basis for apoptosis inhibition by Epstein-Barr virus BHRF1

PLoS pathogens, 2010-12, Vol.6 (12), p.e1001236

2010

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Synthesis of Biotinylated Episilvestrol: Highly Selective Targeting of the Translation Factors eIF4AI/II

Organic letters, 2013-03, Vol.15 (6), p.1406-1409

2013

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Prosurvival Bcl-2 family members affect autophagy only indirectly, by inhibiting Bax and Bak

Proceedings of the National Academy of Sciences - PNAS, 2014-06, Vol.111 (23), p.8512-8517

2014

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Mesenchymal stromal cell apoptosis is required for their therapeutic function

Nature communications, 2021-11, Vol.12 (1), p.6495-6495, Article 6495

2021

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Deubiquitinase USP9X stabilizes MCL1 and promotes tumour cell survival

Nature (London), 2010-01, Vol.463 (7277), p.103-107

2010

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Sheeppox Virus SPPV14 Encodes a Bcl-2-Like Cell Death Inhibitor That Counters a Distinct Set of Mammalian Proapoptotic Proteins

Journal of Virology, 2012-11, Vol.86 (21), p.11501-11511

2012

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In vivo Efficacy of the Bcl-2 Antagonist ABT-737 against Aggressive Myc-Driven Lymphomas

Proceedings of the National Academy of Sciences - PNAS, 2008-11, Vol.105 (46), p.17961-17966

2008

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Structures of BCL-2 in complex with venetoclax reveal the molecular basis of resistance mutations

Nature communications, 2019-06, Vol.10 (1), p.2385-2385, Article 2385

2019

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Apoptosis initiated by Bcl-2-regulated caspase activation independently of the cytochrome c/Apaf-1/caspase-9 apoptosome

Nature (London), 2002-10, Vol.419 (6907), p.634-637

2002

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The BCL2 selective inhibitor venetoclax induces rapid onset apoptosis of CLL cells in patients via a TP53-independent mechanism

Blood, 2016-06, Vol.127 (25), p.3215-3224

2016

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Enhanced stability of Mcl1, a prosurvival Bcl2 relative, blunts stress-induced apoptosis, causes male sterility, and promotes tumorigenesis

Proceedings of the National Academy of Sciences - PNAS, 2014-01, Vol.111 (1), p.261-266

2014

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BH3 mimetics antagonizing restricted prosurvival Bcl-2 proteins represent another class of selective immune modulatory drugs

Proceedings of the National Academy of Sciences - PNAS, 2010-06, Vol.107 (24), p.10967-10971

2010

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De-Novo Designed Library of Benzoylureas as Inhibitors of BCL‑XL: Synthesis, Structural and Biochemical Characterization

Journal of medicinal chemistry, 2014-02, Vol.57 (4), p.1323-1343

2014

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Gefitinib-induced killing of NSCLC cell lines expressing mutant EGFR requires BIM and can be enhanced by BH3 mimetics

Substantial Susceptibility of Chronic Lymphocytic Leukemia to BCL2 Inhibition: Results of a Phase I Study of Navitoclax in Patients With Relapsed or Refractory Disease

XIAP discriminates between type I and type II FAS-induced apoptosis

PRMT1-mediated H4R3me2a recruits SMARCA4 to promote colorectal cancer progression by enhancing EGFR signaling

Sensitization of BCL-2–expressing breast tumors to chemotherapy by the BH3 mimetic ABT-737

Replication stress induces mitotic death through parallel pathways regulated by WAPL and telomere deprotection

Key residues in the VDAC2-BAK complex can be targeted to modulate apoptosis

Apoptosis Initiated When BH3 Ligands Engage Multiple Bcl-2 Homologs, Not Bax or Bak

Current Advances and Future Strategies for BCL-2 Inhibitors: Potent Weapons against Cancers

Two distinct pathways regulate platelet phosphatidylserine exposure and procoagulant function

BCL2 and MCL1 inhibitors for hematologic malignancies

Proapoptotic Bcl-2 Relative Bim Required for Certain Apoptotic Responses, Leukocyte Homeostasis, and to Preclude Autoimmunity

Structural basis for apoptosis inhibition by Epstein-Barr virus BHRF1

Synthesis of Biotinylated Episilvestrol: Highly Selective Targeting of the Translation Factors eIF4AI/II

Prosurvival Bcl-2 family members affect autophagy only indirectly, by inhibiting Bax and Bak

Mesenchymal stromal cell apoptosis is required for their therapeutic function

Deubiquitinase USP9X stabilizes MCL1 and promotes tumour cell survival

Sheeppox Virus SPPV14 Encodes a Bcl-2-Like Cell Death Inhibitor That Counters a Distinct Set of Mammalian Proapoptotic Proteins

In vivo Efficacy of the Bcl-2 Antagonist ABT-737 against Aggressive Myc-Driven Lymphomas

Structures of BCL-2 in complex with venetoclax reveal the molecular basis of resistance mutations

Apoptosis initiated by Bcl-2-regulated caspase activation independently of the cytochrome c/Apaf-1/caspase-9 apoptosome

The BCL2 selective inhibitor venetoclax induces rapid onset apoptosis of CLL cells in patients via a TP53-independent mechanism

Enhanced stability of Mcl1, a prosurvival Bcl2 relative, blunts stress-induced apoptosis, causes male sterility, and promotes tumorigenesis

BH3 mimetics antagonizing restricted prosurvival Bcl-2 proteins represent another class of selective immune modulatory drugs

De-Novo Designed Library of Benzoylureas as Inhibitors of BCL‑XL: Synthesis, Structural and Biochemical Characterization

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