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A highly-sensitive anti-Müllerian hormone assay improves analysis of ovarian function following chemotherapy for early breast cancer

European journal of cancer (1990), 2014-09, Vol.50 (14), p.2367-2374

2014

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Placental hormone profiles as predictors of preterm birth in twin pregnancy: A prospective cohort study

PloS one, 2017-03, Vol.12 (3), p.e0173732-e0173732

2017

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Urinary specific gravity as an alternative for the normalisation of endocrine metabolite concentrations in giant panda (Ailuropoda melanoleuca) reproductive monitoring

PloS one, 2018-07, Vol.13 (7), p.e0201420-e0201420

2018

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Ischemic preconditioning in the liver is independent of regulatory T cell activity

PloS one, 2012-11, Vol.7 (11), p.e49647-e49647

2012

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Sulforaphane, Erucin, and Iberin Up-Regulate Thioredoxin Reductase 1 Expression in Human MCF-7 Cells

Journal of agricultural and food chemistry, 2005-03, Vol.53 (5), p.1417-1421

2005

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Synergy between sulforaphane and selenium in the induction of thioredoxin reductase 1 requires both transcriptional and translational modulation

Carcinogenesis (New York), 2003-03, Vol.24 (3), p.497-503

2003

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Dual Action of Sulforaphane in the Regulation of Thioredoxin Reductase and Thioredoxin in Human HepG2 and Caco-2 Cells

Journal of agricultural and food chemistry, 2007-02, Vol.55 (4), p.1170-1176

2007

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Reproductive and metabolic adaptation to multistressor training in women

American journal of physiology: endocrinology and metabolism, 2021-08, Vol.321 (2), p.E281-E291

2021

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Complement C5a is present in CSF of human newborns and is elevated in association with preterm birth

The journal of maternal-fetal & neonatal medicine, 2017-10, Vol.30 (20), p.2413-2416

2017

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Functional characterisation of the H365Y mutation of the 21-hydroxylase gene in congenital adrenal hyperplasia

The Journal of steroid biochemistry and molecular biology, 2011-02, Vol.123 (3), p.109-114

2011

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Profiling the impact of medium formulation on morphology and functionality of primary hepatocytes in vitro

Scientific reports, 2013-09, Vol.3 (1), p.2735, Article 2735

2013

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Synergy between broccoli sprout extract and selenium in the upregulation of thioredoxin reductase in human hepatocytes

Food chemistry, 2008-09, Vol.110 (1), p.193-198

2008

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Comparison of Bioenergetic Activity of Primary Porcine Hepatocytes Cultured in Four Different Media

Cell transplantation, 2004-01, Vol.13 (3), p.213-229

2004

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Differential Effects of Doses and Forms of Dietary Selenium on Immune Cell Numbers in the Skin of Ultraviolet-irradiated and Unirradiated Mice

Biological trace element research, 2008-12, Vol.125 (3), p.255-267

2008

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Differential expression of selenoproteins by human skin cells and protection by selenium from UVB-radiation-induced cell death

Biochemical journal, 1998-05, Vol.332 ( Pt 1) (1), p.231-236

1998

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Thioredoxin reductase is the major selenoprotein expressed in human umbilical-vein endothelial cells and is regulated by protein kinase C

Biochemical journal, 1999-08, Vol.342 ( Pt 1) (1), p.111-117

1999

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Glutathione S-transferase mu locus: use of genotyping and phenotyping assays to assess association with lung cancer susceptibility

Carcinogenesis (New York), 1991-09, Vol.12 (9), p.1533-1537

1991

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An improved ex vivo method of primary porcine hepatocyte isolation for use in bioartificial liver systems

European journal of gastroenterology & hepatology, 2000-08, Vol.12 (8), p.923-930

2000

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Effect of sevoflurane anaesthesia on plasma concentrations of glutathione S-transferase

British journal of anaesthesia : BJA, 1996-09, Vol.77 (3), p.404-407

1996

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Soluble adhesion molecules and interleukin-2 receptor concentrations in patients with autoimmune chronic hepatitis

European journal of gastroenterology & hepatology, 1995-05, Vol.7 (5), p.455-460

1995

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Selenite protects human endothelial cells from oxidative damage and induces thioredoxin reductase

Clinical science (1979), 2001-05, Vol.100 (5), p.543

2001

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Identification of a 57-kilodalton selenoprotein in human thyrocytes as thioredoxin reductase and evidence that its expression is regulated through the calcium-phosphoinositol signaling pathway

The journal of clinical endocrinology and metabolism, 1998-06, Vol.83 (6), p.2052-2058

1998

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A novel nonstop mutation in the stop codon and a novel missense mutation in the type II 3beta-hydroxysteroid dehydrogenase (3beta-HSD) gene causing, respectively, nonclassic and classic 3beta-HSD deficiency congenital adrenal hyperplasia

The journal of clinical endocrinology and metabolism, 2002-06, Vol.87 (6), p.2556-2563

2002

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A novel nonstop mutation in the stop codon and a novel missense mutation in the type II 3β-hydroxysteroid dehydrogenase (3β-HSD) gene causing, respectively, nonclassic and classic 3β-HSD deficiency congenital adrenal hyperplasia : The impact of the human genome on endocrinology: Special features

The journal of clinical endocrinology and metabolism, 2002, Vol.87 (6), p.2556-2563

2002

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A Novel Nonstop Mutation in the Stop Codon and a Novel Missense Mutation in the Type II 3β-Hydroxysteroid Dehydrogenase (3β-HSD) Gene Causing, Respectively, Nonclassic and Classic 3β-HSD Deficiency Congenital Adrenal Hyperplasia

The journal of clinical endocrinology and metabolism, 2002-06, Vol.87 (6), p.2556-2563

2002

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A highly-sensitive anti-Müllerian hormone assay improves analysis of ovarian function following chemotherapy for early breast cancer

Placental hormone profiles as predictors of preterm birth in twin pregnancy: A prospective cohort study

Urinary specific gravity as an alternative for the normalisation of endocrine metabolite concentrations in giant panda (Ailuropoda melanoleuca) reproductive monitoring

Ischemic preconditioning in the liver is independent of regulatory T cell activity

Sulforaphane, Erucin, and Iberin Up-Regulate Thioredoxin Reductase 1 Expression in Human MCF-7 Cells

Synergy between sulforaphane and selenium in the induction of thioredoxin reductase 1 requires both transcriptional and translational modulation

Dual Action of Sulforaphane in the Regulation of Thioredoxin Reductase and Thioredoxin in Human HepG2 and Caco-2 Cells

Reproductive and metabolic adaptation to multistressor training in women

Complement C5a is present in CSF of human newborns and is elevated in association with preterm birth

Functional characterisation of the H365Y mutation of the 21-hydroxylase gene in congenital adrenal hyperplasia

Profiling the impact of medium formulation on morphology and functionality of primary hepatocytes in vitro

Synergy between broccoli sprout extract and selenium in the upregulation of thioredoxin reductase in human hepatocytes

Comparison of Bioenergetic Activity of Primary Porcine Hepatocytes Cultured in Four Different Media

Differential Effects of Doses and Forms of Dietary Selenium on Immune Cell Numbers in the Skin of Ultraviolet-irradiated and Unirradiated Mice

Differential expression of selenoproteins by human skin cells and protection by selenium from UVB-radiation-induced cell death

Thioredoxin reductase is the major selenoprotein expressed in human umbilical-vein endothelial cells and is regulated by protein kinase C

Glutathione S-transferase mu locus: use of genotyping and phenotyping assays to assess association with lung cancer susceptibility

An improved ex vivo method of primary porcine hepatocyte isolation for use in bioartificial liver systems

Effect of sevoflurane anaesthesia on plasma concentrations of glutathione S-transferase

Soluble adhesion molecules and interleukin-2 receptor concentrations in patients with autoimmune chronic hepatitis

Selenite protects human endothelial cells from oxidative damage and induces thioredoxin reductase

Identification of a 57-kilodalton selenoprotein in human thyrocytes as thioredoxin reductase and evidence that its expression is regulated through the calcium-phosphoinositol signaling pathway

A novel nonstop mutation in the stop codon and a novel missense mutation in the type II 3beta-hydroxysteroid dehydrogenase (3beta-HSD) gene causing, respectively, nonclassic and classic 3beta-HSD deficiency congenital adrenal hyperplasia

A novel nonstop mutation in the stop codon and a novel missense mutation in the type II 3β-hydroxysteroid dehydrogenase (3β-HSD) gene causing, respectively, nonclassic and classic 3β-HSD deficiency congenital adrenal hyperplasia : The impact of the human genome on endocrinology: Special features

A Novel Nonstop Mutation in the Stop Codon and a Novel Missense Mutation in the Type II 3β-Hydroxysteroid Dehydrogenase (3β-HSD) Gene Causing, Respectively, Nonclassic and Classic 3β-HSD Deficiency Congenital Adrenal Hyperplasia

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