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Recent advances in acute myeloid leukemia stem cell biology

Haematologica (Roma), 2012-07, Vol.97 (7), p.966-974

2012

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Early loss of Crebbp confers malignant stem cell properties on lymphoid progenitors

Nature cell biology, 2017-09, Vol.19 (9), p.1093-1104

2017

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BRD3/4 inhibition and FLT3-ligand deprivation target pathways that are essential for the survival of human MLL-AF9+ leukemic cells

PloS one, 2017-12, Vol.12 (12), p.e0189102-e0189102

2017

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Glutaminolysis is a metabolic dependency in FLT3ITD acute myeloid leukemia unmasked by FLT3 tyrosine kinase inhibition

Blood, 2018-04, Vol.131 (15), p.1639-1653

2018

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Zinc finger protein 521 antagonizes early B-cell factor 1 and modulates the B-lymphoid differentiation of primary hematopoietic progenitors

Cell cycle (Georgetown, Tex.), 2011-07, Vol.10 (13), p.2129-2139

2011

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HOXA9 forms a repressive complex with nuclear matrix–associated protein SAFB to maintain acute myeloid leukemia

Blood, 2023-04, Vol.141 (14), p.1737-1754

2023

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Continuous MLL-ENL expression is necessary to establish a hox code and maintain immortalization of hematopoietic progenitor cells

Cancer research (Chicago, Ill.), 2005-10, Vol.65 (20), p.9245-9252

2005

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Genetically engineered mesenchymal stromal cells produce IL-3 and TPO to further improve human scaffold-based xenograft models

Experimental hematology, 2017-07, Vol.51, p.36-46

2017

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A novel mouse model identifies cooperating mutations and therapeutic targets critical for chronic myeloid leukemia progression

The Journal of experimental medicine, 2015-09, Vol.212 (10), p.1551-1569

2015

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BMI1 collaborates with BCR-ABL in leukemic transformation of human CD34+ cells

Blood, 2010-11, Vol.116 (22), p.4621-4630

2010

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The Mechanism of Hematopoietic Progenitor Cell immortalization by MLL-ENL

Cell cycle (Georgetown, Tex.), 2006-02, Vol.5 (4), p.360-362

2006

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Frat2 mediates the oncogenic activation of Rac by MLL fusions

Blood, 2012-12, Vol.120 (24), p.4819-4828

2012

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Regulatory T-Cell Response to Low-Dose Interleukin-2 in Ischemic Heart Disease

NEJM evidence, 2022-01, Vol.1 (1), p.EVIDoa2100009

2022

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Acute myeloid leukemia induced by MLL-ENL is cured by oncogene ablation despite acquisition of complex genetic abnormalities

Blood, 2009-05, Vol.113 (20), p.4922-4929

2009

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Genetic and epigenetic alterations that drive leukemic stem cell self-renewal

Journal of stem cells, 2012, Vol.7 (3), p.155

2012

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HOXA9 Nucleates a Repressive Chromatin Complex with Nuclear Matrix-Associated Protein- Safb to Maintain Acute Myeloid Leukemia

Blood, 2022-11, Vol.140 (Supplement 1), p.720-721

2022

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Record of the Transmission of Illumination: Volume 1; an Annotated Translation of Zen Master Keizan's Denkōroku

1, 2021

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BRD3/4 inhibition and FLT3-ligand deprivation target pathways that are essential for the survival of human MLL-AF9.sup.+ leukemic cells

PloS one, 2017-12, Vol.12 (12), p.e0189102

2017

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Mutational Synergy Coordinately Remodels Chromatin Accessibility, Enhancer Landscape and 3-Dimensional DNA Topology to Alter Gene Expression during Leukemia Induction

Blood, 2019-11, Vol.134 (Supplement_1), p.278-278

2019

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Abstract 363: Genetic or pharmacological inactivation of CREBBP sensitizes B-cell acute lymphoblastic leukemia to ferroptotic cell death upon BCL2 inhibition

Cancer research (Chicago, Ill.), 2024-03, Vol.84 (6_Supplement), p.363-363

2024

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Abstract 5026: Targeting non-canonical Wnt signaling in acute myeloid leukemia

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.5026-5026

2013

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Glutaminolysis is a metabolic dependency in FLT3 ITD acute myeloid leukemia unmasked by FLT3 tyrosine kinase inhibition

Blood, 2018-04, Vol.131 (15), p.1639

2018

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Acute Myeloid Leukemia Induced by MLL-ENL Is Cured by Oncogene Ablation despite Acquisition of Complex Genetic Abnormalities

Blood, 2008-11, Vol.112 (11), p.3109-3109

2008

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Recent advances in acute myeloid leukemia stem cell biology

Early loss of Crebbp confers malignant stem cell properties on lymphoid progenitors

BRD3/4 inhibition and FLT3-ligand deprivation target pathways that are essential for the survival of human MLL-AF9+ leukemic cells

Glutaminolysis is a metabolic dependency in FLT3ITD acute myeloid leukemia unmasked by FLT3 tyrosine kinase inhibition

Zinc finger protein 521 antagonizes early B-cell factor 1 and modulates the B-lymphoid differentiation of primary hematopoietic progenitors

Mutational synergy during leukemia induction remodels chromatin accessibility, histone modifications and three-dimensional DNA topology to alter gene expression

HOXA9 forms a repressive complex with nuclear matrix–associated protein SAFB to maintain acute myeloid leukemia

Continuous MLL-ENL expression is necessary to establish a hox code and maintain immortalization of hematopoietic progenitor cells

Genetically engineered mesenchymal stromal cells produce IL-3 and TPO to further improve human scaffold-based xenograft models

A novel mouse model identifies cooperating mutations and therapeutic targets critical for chronic myeloid leukemia progression

BMI1 collaborates with BCR-ABL in leukemic transformation of human CD34+ cells

The Mechanism of Hematopoietic Progenitor Cell immortalization by MLL-ENL

Frat2 mediates the oncogenic activation of Rac by MLL fusions

Regulatory T-Cell Response to Low-Dose Interleukin-2 in Ischemic Heart Disease

Acute myeloid leukemia induced by MLL-ENL is cured by oncogene ablation despite acquisition of complex genetic abnormalities

Genetic and epigenetic alterations that drive leukemic stem cell self-renewal

HOXA9 Nucleates a Repressive Chromatin Complex with Nuclear Matrix-Associated Protein- Safb to Maintain Acute Myeloid Leukemia

Record of the Transmission of Illumination: Volume 1; an Annotated Translation of Zen Master Keizan's Denkōroku

Mutational synergy during leukemia induction remodels chromatin accessibility, modification and 3-Dimensional DNA topology to alter gene expression

BRD3/4 inhibition and FLT3-ligand deprivation target pathways that are essential for the survival of human MLL-AF9.sup.+ leukemic cells

Mutational Synergy Coordinately Remodels Chromatin Accessibility, Enhancer Landscape and 3-Dimensional DNA Topology to Alter Gene Expression during Leukemia Induction

Abstract 363: Genetic or pharmacological inactivation of CREBBP sensitizes B-cell acute lymphoblastic leukemia to ferroptotic cell death upon BCL2 inhibition

Abstract 5026: Targeting non-canonical Wnt signaling in acute myeloid leukemia

Glutaminolysis is a metabolic dependency in FLT3 ITD acute myeloid leukemia unmasked by FLT3 tyrosine kinase inhibition

Acute Myeloid Leukemia Induced by MLL-ENL Is Cured by Oncogene Ablation despite Acquisition of Complex Genetic Abnormalities

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