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The L-Type Ca2+ Channel as a Therapeutic Target in Heart Disease

Current medicinal chemistry, 2009-09, Vol.16 (26), p.3341-3358

2009

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The L-type Ca(2+) channel as a therapeutic target in heart disease

Current medicinal chemistry, 2009, Vol.16 (26), p.3341

2009

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AlGaN/GaN-based biosensor for label-free detection of biological activity

Sensors and actuators. B, Chemical, 2013-02, Vol.177, p.577-582

2013

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Effect of osmotic swelling and shrinkage on Na(+)-K+ pump activity in mammalian cardiac myocytes

American Journal of Physiology: Cell Physiology, 1993-11, Vol.265 (5), p.C1201-C1210

1993

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Genistein Increases the Sensitivity of Cardiac Ion Channels to beta-Adrenergic Receptor Stimulation

Circulation research, 1998-07, Vol.83 (1), p.33-42

1998

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Angiotensin-converting enzyme inhibition, intracellular Na+, and Na(+)-K+ pumping in cardiac myocytes

American Journal of Physiology: Cell Physiology, 1995-02, Vol.268 (2), p.C366-C375

1995

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Angiotensin-converting enzyme inhibitors regulate the Na(+)-K+ pump via effects on angiotensin metabolism

American Journal of Physiology: Cell Physiology, 1996-07, Vol.271 (1), p.C172-C180

1996

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Dietary cholesterol affects Na+-K+ pump function in rabbit cardiac myocytes

American journal of physiology. Heart and circulatory physiology, 1997-04, Vol.272 (4), p.H1680-H1689

1997

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Loss of the RNA-binding protein TACO1 causes late-onset mitochondrial dysfunction in mice

Nature communications, 2016-06, Vol.7 (1), p.11884-11884, Article 11884

2016

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Thyroid status and regulation of intracellular sodium in rabbit heart

American journal of physiology. Heart and circulatory physiology, 1997-04, Vol.272 (4), p.H1589-H1597

1997

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Thyroid status and Na(+)-K+ pump current, intracellular sodium, and action potential duration in rabbit heart

American journal of physiology. Heart and circulatory physiology, 1995-05, Vol.268 (5), p.H1838-H1846

1995

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PTCD1 Is Required for 16S rRNA Maturation Complex Stability and Mitochondrial Ribosome Assembly

Cell reports (Cambridge), 2018-04, Vol.23 (1), p.127-142

2018

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Hypoxia increases the sensitivity of the L-type Ca(2+) current to beta-adrenergic receptor stimulation via a C2 region-containing protein kinase C isoform

Circulation research, 2000-12, Vol.87 (12), p.1164-1171

Hool, L C

2000

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REACTIVE OXYGEN SPECIES IN CARDIAC SIGNALLING: FROM MITOCHONDRIA TO PLASMA MEMBRANE ION CHANNELS

Clinical and experimental pharmacology & physiology, 2006-01, Vol.33 (1-2), p.146-151

Hool, Livia C

2006

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Hypoxia alters the sensitivity of the L-type Ca(2+) channel to alpha-adrenergic receptor stimulation in the presence of beta-adrenergic receptor stimulation

Circulation research, 2001-05, Vol.88 (10), p.1036-1043

Hool, L C

2001

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Hypoxia Increases the Sensitivity of the L-Type Ca2+ Current to β-Adrenergic Receptor Stimulation via a C2 Region–Containing Protein Kinase C Isoform

Circulation research, 2000-12, Vol.87 (12), p.1164-1171

Hool, Livia C

2000

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Decreasing Cellular Hydrogen Peroxide With Catalase Mimics the Effects of Hypoxia on the Sensitivity of the L-Type Ca2+ Channel to β-Adrenergic Receptor Stimulation in Cardiac Myocytes

Circulation research, 2002-10, Vol.91 (7), p.601-609

2002

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Role of NAD(P)H oxidase in the regulation of cardiac L-type Ca2+ channel function during acute hypoxia

Cardiovascular research, 2005-09, Vol.67 (4), p.624-635

2005

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Hypoxia Alters the Sensitivity of the L-Type Ca2+ Channel to α-Adrenergic Receptor Stimulation in the Presence of β-Adrenergic Receptor Stimulation

Circulation research, 2001-05, Vol.88 (10), p.1036-1043

Hool, Livia C

2001

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Differential regulation of the slow and rapid components of guinea-pig cardiac delayed rectifier K+ channels by hypoxia

The Journal of physiology, 2004-02, Vol.554 (3), p.743-754

Hool, Livia C.

2004

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Acute hypoxia differentially regulates K(+) channels. Implications with respect to cardiac arrhythmia

European biophysics journal, 2005-07, Vol.34 (5), p.369-376

Hool, Livia C

2005

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Protein kinase C isozyme selective peptides--a current view of what they tell us about location and function of isozymes in the heart

Current pharmaceutical design, 2005-01, Vol.11 (4), p.549-559

Hool, Livia C

2005

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Can Integrins Integrate Vascular Myogenic Responses?

Circulation research, 2002-03, Vol.90 (4), p.371-373

Hool, Livia C

2002

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α1-Adrenergic inhibition of the β-adrenergically activated Cl- current in guinea pig ventricular myocytes

Circulation research, 1996, Vol.78 (6), p.1090-1099

1996

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Role of β1- and β2-adrenergic receptors in regulation of Cl- and Ca2+ channels in guinea pig ventricular myocytes

American journal of physiology. Heart and circulatory physiology, 1997, Vol.42 (4), p.H1669-H1676

1997

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The L-Type Ca2+ Channel as a Therapeutic Target in Heart Disease

The L-type Ca(2+) channel as a therapeutic target in heart disease

AlGaN/GaN-based biosensor for label-free detection of biological activity

Effect of osmotic swelling and shrinkage on Na(+)-K+ pump activity in mammalian cardiac myocytes

Genistein Increases the Sensitivity of Cardiac Ion Channels to beta-Adrenergic Receptor Stimulation

Angiotensin-converting enzyme inhibition, intracellular Na+, and Na(+)-K+ pumping in cardiac myocytes

Angiotensin-converting enzyme inhibitors regulate the Na(+)-K+ pump via effects on angiotensin metabolism

Dietary cholesterol affects Na+-K+ pump function in rabbit cardiac myocytes

Loss of the RNA-binding protein TACO1 causes late-onset mitochondrial dysfunction in mice

Thyroid status and regulation of intracellular sodium in rabbit heart

Thyroid status and Na(+)-K+ pump current, intracellular sodium, and action potential duration in rabbit heart

PTCD1 Is Required for 16S rRNA Maturation Complex Stability and Mitochondrial Ribosome Assembly

Hypoxia increases the sensitivity of the L-type Ca(2+) current to beta-adrenergic receptor stimulation via a C2 region-containing protein kinase C isoform

REACTIVE OXYGEN SPECIES IN CARDIAC SIGNALLING: FROM MITOCHONDRIA TO PLASMA MEMBRANE ION CHANNELS

Hypoxia alters the sensitivity of the L-type Ca(2+) channel to alpha-adrenergic receptor stimulation in the presence of beta-adrenergic receptor stimulation

Hypoxia Increases the Sensitivity of the L-Type Ca2+ Current to β-Adrenergic Receptor Stimulation via a C2 Region–Containing Protein Kinase C Isoform

Decreasing Cellular Hydrogen Peroxide With Catalase Mimics the Effects of Hypoxia on the Sensitivity of the L-Type Ca2+ Channel to β-Adrenergic Receptor Stimulation in Cardiac Myocytes

Role of NAD(P)H oxidase in the regulation of cardiac L-type Ca2+ channel function during acute hypoxia

Hypoxia Alters the Sensitivity of the L-Type Ca2+ Channel to α-Adrenergic Receptor Stimulation in the Presence of β-Adrenergic Receptor Stimulation

Differential regulation of the slow and rapid components of guinea-pig cardiac delayed rectifier K+ channels by hypoxia

Acute hypoxia differentially regulates K(+) channels. Implications with respect to cardiac arrhythmia

Protein kinase C isozyme selective peptides--a current view of what they tell us about location and function of isozymes in the heart

Can Integrins Integrate Vascular Myogenic Responses?

α1-Adrenergic inhibition of the β-adrenergically activated Cl- current in guinea pig ventricular myocytes

Role of β1- and β2-adrenergic receptors in regulation of Cl- and Ca2+ channels in guinea pig ventricular myocytes

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