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Extensive 5′-surveillance guards against non-canonical NAD-caps of nuclear mRNAs in yeast

Nature communications, 2020-11, Vol.11 (1), p.5508-5508, Article 5508

2020

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PCNA and Msh2-Msh6 Activate an Mlh1-Pms1 Endonuclease Pathway Required for Exo1-Independent Mismatch Repair

Molecular cell, 2014-07, Vol.55 (2), p.291-304

2014

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Dominant mutations in S. cerevisiae PMS1 identify the Mlh1-Pms1 endonuclease active site and an exonuclease 1-independent mismatch repair pathway

PLoS genetics, 2013-10, Vol.9 (10), p.e1003869-e1003869

2013

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Mlh2 is an accessory factor for DNA mismatch repair in Saccharomyces cerevisiae

PLoS genetics, 2014-05, Vol.10 (5), p.e1004327-e1004327

2014

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Identification of MLH2/hPMS1 dominant mutations that prevent DNA mismatch repair function

Communications biology, 2020-12, Vol.3 (1), p.751-751, Article 751

2020

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Checkpoint Kinases Regulate a Global Network of Transcription Factors in Response to DNA Damage

Cell reports (Cambridge), 2013-07, Vol.4 (1), p.174-188

2013

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Generation of active protein phosphatase 2A is coupled to holoenzyme assembly

PLoS biology, 2007-06, Vol.5 (6), p.e155-e155

2007

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A novel and essential mechanism determining specificity and activity of protein phosphatase 2A (PP2A) in vivo

Genes & development, 2003-09, Vol.17 (17), p.2138-2150

2003

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New insights into the mechanism of DNA mismatch repair

Chromosoma, 2015-12, Vol.124 (4), p.443-462

2015

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Mismatch Repair, But Not Heteroduplex Rejection, Is Temporally Coupled to DNA Replication

Science (American Association for the Advancement of Science), 2011-12, Vol.334 (6063), p.1713-1716

2011

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Ligation of newly replicated DNA controls the timing of DNA mismatch repair

Current biology, 2021-03, Vol.31 (6), p.1268-1276.e6

2021

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Inactivation of folylpolyglutamate synthetase Met7 results in genome instability driven by an increased dUTP/dTTP ratio

Nucleic acids research, 2020-01, Vol.48 (1), p.264

2020

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Visualization of mismatch repair complexes using fluorescence microscopy

DNA repair, 2016-02, Vol.38, p.58-67

2016

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Alterations in cellular metabolism triggered by URA7 or GLN3 inactivation cause imbalanced dNTP pools and increased mutagenesis

Proceedings of the National Academy of Sciences - PNAS, 2017-05, Vol.114 (22), p.E4442-E4451

2017

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A genetic screen pinpoints ribonucleotide reductase residues that sustain dNTP homeostasis and specifies a highly mutagenic type of dNTP imbalance

Nucleic acids research, 2019-01, Vol.47 (1), p.237

2019

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A genome wide screen to identify genes that prevent the accumulation of mutations

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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Cdc28/Cdk1 positively and negatively affects genome stability in S. cerevisiae

The Journal of cell biology, 2009-05, Vol.185 (3), p.423-437

2009

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Mlh2 Is an Accessory Factor for DNA Mismatch Repair in Saccharomyces cerevisiae: e1004327

PLoS genetics, 2014-05, Vol.10 (5)

2014

Volltextzugriff (PDF)

Dominant mutations in S. cerevisiae PMS1 identify the Mhl-PMS1 endonuclease active site and an exonuclease 1-independent mismatch repair pathway

PLoS genetics, 2013-10, Vol.9 (10)

2013

Volltextzugriff (PDF)

Dominant Mutations in S. cerevisiae PMS1 Identify the Mlh1-Pms1 Endonuclease Active Site and an Exonuclease 1-Independent Mismatch Repair Pathway: e1003869

PLoS genetics, 2013-10, Vol.9 (10)

2013

Volltextzugriff (PDF)

Identification of a Subunit of a Novel Kleisin-β/SMC Complex as a Potential Substrate of Protein Phosphatase 2A

Current biology, 2003-12, Vol.13 (23), p.2058-2064

2003

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Extensive 5′-surveillance guards against non-canonical NAD-caps of nuclear mRNAs in yeast

PCNA and Msh2-Msh6 Activate an Mlh1-Pms1 Endonuclease Pathway Required for Exo1-Independent Mismatch Repair

Dominant mutations in S. cerevisiae PMS1 identify the Mlh1-Pms1 endonuclease active site and an exonuclease 1-independent mismatch repair pathway

Mlh2 is an accessory factor for DNA mismatch repair in Saccharomyces cerevisiae

Identification of MLH2/hPMS1 dominant mutations that prevent DNA mismatch repair function

Checkpoint Kinases Regulate a Global Network of Transcription Factors in Response to DNA Damage

Generation of active protein phosphatase 2A is coupled to holoenzyme assembly

A novel and essential mechanism determining specificity and activity of protein phosphatase 2A (PP2A) in vivo

New insights into the mechanism of DNA mismatch repair

Mismatch Repair, But Not Heteroduplex Rejection, Is Temporally Coupled to DNA Replication

Ligation of newly replicated DNA controls the timing of DNA mismatch repair

Inactivation of folylpolyglutamate synthetase Met7 results in genome instability driven by an increased dUTP/dTTP ratio

Visualization of mismatch repair complexes using fluorescence microscopy

Alterations in cellular metabolism triggered by URA7 or GLN3 inactivation cause imbalanced dNTP pools and increased mutagenesis

A genetic screen pinpoints ribonucleotide reductase residues that sustain dNTP homeostasis and specifies a highly mutagenic type of dNTP imbalance

SRD5A3 Is Required for Converting Polyprenol to Dolichol and Is Mutated in a Congenital Glycosylation Disorder

A genome wide screen to identify genes that prevent the accumulation of mutations

Cdc28/Cdk1 positively and negatively affects genome stability in S. cerevisiae

Mlh2 Is an Accessory Factor for DNA Mismatch Repair in Saccharomyces cerevisiae: e1004327

Dominant mutations in S. cerevisiae PMS1 identify the Mhl-PMS1 endonuclease active site and an exonuclease 1-independent mismatch repair pathway

Dominant Mutations in S. cerevisiae PMS1 Identify the Mlh1-Pms1 Endonuclease Active Site and an Exonuclease 1-Independent Mismatch Repair Pathway: e1003869

Identification of a Subunit of a Novel Kleisin-β/SMC Complex as a Potential Substrate of Protein Phosphatase 2A

SRD5A3 is required for the conversion of polyprenol to dolichol, essential for N-linked protein glycosylation

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