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Measuring In Vivo Mitophagy

Molecular cell, 2015-11, Vol.60 (4), p.685-696

2015

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PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1

Nature cell biology, 2010-02, Vol.12 (2), p.119-131

2010

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The Ins and Outs of Mitochondrial Calcium

Circulation research, 2015-05, Vol.116 (11), p.1810-1819

2015

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MICU1 Serves as a Molecular Gatekeeper to Prevent In Vivo Mitochondrial Calcium Overload

Cell reports (Cambridge), 2016-08, Vol.16 (6), p.1561-1573

2016

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Mutations in ANO3 Cause Dominant Craniocervical Dystonia: Ion Channel Implicated in Pathogenesis

American journal of human genetics, 2012-12, Vol.91 (6), p.1041-1050

2012

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The multifaceted role of Nrf2 in mitochondrial function

Current opinion in toxicology, 2016-12, Vol.1, p.80-91

2016

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Nrf2 impacts cellular bioenergetics by controlling substrate availability for mitochondrial respiration

Biology open, 2013-08, Vol.2 (8), p.761-770

2013

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Rare Individual Amyloid‑β Oligomers Act on Astrocytes to Initiate Neuronal Damage

Biochemistry (Easton), 2014-04, Vol.53 (15), p.2442-2453

2014

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Cellular mechanisms and physiological consequences of redox-dependent signalling

Nature reviews. Molecular cell biology, 2014-06, Vol.15 (6), p.411-421

2014

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Assessment of cardiac function in mice lacking the mitochondrial calcium uniporter

Journal of molecular and cellular cardiology, 2015-08, Vol.85, p.178-182

2015

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The PINK1/Parkin-mediated mitophagy is compromised by PD-associated mutations

Autophagy, 2010-10, Vol.6 (7), p.871-878

2010

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Brain-penetrating neurotrophic factor mimetics: HER-096 as a disease-modifying therapy for Parkinson’s disease

Neural regeneration research, 2025-04, Vol.20 (4), p.1094-1095

2025

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The spatiotemporal regulation of the Keap1-Nrf2 pathway and its importance in cellular bioenergetics

Biochemical Society transactions, 2015-08, Vol.43 (4), p.602-610

2015

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Broad AOX expression in a genetically tractable mouse model does not disturb normal physiology

Disease models & mechanisms, 2017-02, Vol.10 (2), p.163-171

2017

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EMRE is essential for mitochondrial calcium uniporter activity in a mouse model

JCI insight, 2020-02, Vol.5 (4)

2020

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Nrf2 regulates ROS production by mitochondria and NADPH oxidase

Biochimica et biophysica acta, 2015-04, Vol.1850 (4), p.794-801

2015

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Cyclophilin D-mediated regulation of the permeability transition pore is altered in mice lacking the mitochondrial calcium uniporter

Cardiovascular research, 2019-02, Vol.115 (2), p.385-394

2019

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Acetylcholine and antibodies against the acetylcholine receptor protect neurons and astrocytes against beta-amyloid toxicity

The international journal of biochemistry & cell biology, 2013-04, Vol.45 (4), p.899-907

2013

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Unresolved questions from the analysis of mice lacking MCU expression

Biochemical and biophysical research communications, 2014-07, Vol.449 (4), p.384-385

2014

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Signalling properties of inorganic polyphosphate in the mammalian brain

Nature communications, 2013-01, Vol.4 (1), p.1362-1362, Article 1362

2013

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HER-096 is a CDNF-derived brain-penetrating peptidomimetic that protects dopaminergic neurons in a mouse synucleinopathy model of Parkinson’s disease

Cell chemical biology, 2024-03, Vol.31 (3), p.593-606.e9

2024

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Glucocorticoids reduce intracellular calcium concentration and protects neurons against glutamate toxicity

Cell calcium (Edinburgh), 2013-04, Vol.53 (4), p.256-263

2013

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Modulation of mitochondrial function and morphology by interaction of Omi/HtrA2 with the mitochondrial fusion factor OPA1

Experimental cell research, 2010-04, Vol.316 (7), p.1213-1224

2010

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Abstract 513: Emre Regulates the Mitochondrial Calcium Uniporter in vivo

Circulation research, 2020-07, Vol.127 (Suppl_1)

2020

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Role of Polyphosphate in Mitochondria: From Modification of Energy Metabolism to Induction of the Cell Death

Biophysical journal, 2013-01, Vol.104 (2), p.660a-660a

2013

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Mice, Knockout
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Measuring In Vivo Mitophagy

PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1

The Ins and Outs of Mitochondrial Calcium

MICU1 Serves as a Molecular Gatekeeper to Prevent In Vivo Mitochondrial Calcium Overload

Mutations in ANO3 Cause Dominant Craniocervical Dystonia: Ion Channel Implicated in Pathogenesis

The multifaceted role of Nrf2 in mitochondrial function

Nrf2 impacts cellular bioenergetics by controlling substrate availability for mitochondrial respiration

Rare Individual Amyloid‑β Oligomers Act on Astrocytes to Initiate Neuronal Damage

Cellular mechanisms and physiological consequences of redox-dependent signalling

Assessment of cardiac function in mice lacking the mitochondrial calcium uniporter

The PINK1/Parkin-mediated mitophagy is compromised by PD-associated mutations

Brain-penetrating neurotrophic factor mimetics: HER-096 as a disease-modifying therapy for Parkinson’s disease

The spatiotemporal regulation of the Keap1-Nrf2 pathway and its importance in cellular bioenergetics

Broad AOX expression in a genetically tractable mouse model does not disturb normal physiology

EMRE is essential for mitochondrial calcium uniporter activity in a mouse model

Nrf2 regulates ROS production by mitochondria and NADPH oxidase

Cyclophilin D-mediated regulation of the permeability transition pore is altered in mice lacking the mitochondrial calcium uniporter

Acetylcholine and antibodies against the acetylcholine receptor protect neurons and astrocytes against beta-amyloid toxicity

Unresolved questions from the analysis of mice lacking MCU expression

Signalling properties of inorganic polyphosphate in the mammalian brain

HER-096 is a CDNF-derived brain-penetrating peptidomimetic that protects dopaminergic neurons in a mouse synucleinopathy model of Parkinson’s disease

Glucocorticoids reduce intracellular calcium concentration and protects neurons against glutamate toxicity

Modulation of mitochondrial function and morphology by interaction of Omi/HtrA2 with the mitochondrial fusion factor OPA1

Abstract 513: Emre Regulates the Mitochondrial Calcium Uniporter in vivo

Role of Polyphosphate in Mitochondria: From Modification of Energy Metabolism to Induction of the Cell Death

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