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ST6Gal-I sialyltransferase promotes tumor necrosis factor (TNF)-mediated cancer cell survival via sialylation of the TNF receptor 1 (TNFR1) death receptor

The Journal of biological chemistry, 2018-02, Vol.293 (5), p.1610-1622

2018

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Regulation of inflammatory signaling by the ST6Gal-I sialyltransferase

PloS one, 2020-11, Vol.15 (11), p.e0241850-e0241850

2020

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The Tumor-Associated Glycosyltransferase ST6Gal-I Regulates Stem Cell Transcription Factors and Confers a Cancer Stem Cell Phenotype

Cancer research (Chicago, Ill.), 2016-07, Vol.76 (13), p.3978-3988

2016

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Therapeutic efficacy of suppressing the Jak/STAT pathway in multiple models of experimental autoimmune encephalomyelitis

The Journal of immunology (1950), 2014-01, Vol.192 (1), p.59-72

2014

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Signal transducer and activator of transcription-3/suppressor of cytokine signaling-3 (STAT3/SOCS3) axis in myeloid cells regulates neuroinflammation

Proceedings of the National Academy of Sciences - PNAS, 2012-03, Vol.109 (13), p.5004-5009

2012

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Sialylation of EGFR by the ST6Gal-I sialyltransferase promotes EGFR activation and resistance to gefitinib-mediated cell death

Journal of ovarian research, 2018-02, Vol.11 (1), p.12-11, Article 12

2018

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Preferential Recruitment of Neutrophils into the Cerebellum and Brainstem Contributes to the Atypical Experimental Autoimmune Encephalomyelitis Phenotype

The Journal of immunology (1950), 2015-08, Vol.195 (3), p.841-852

2015

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AMP-activated protein kinase restricts IFN-γ signaling

The Journal of immunology (1950), 2013-01, Vol.190 (1), p.372-380

2013

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Correction: SOCS3 Deficiency Promotes M1 Macrophage Polarization and Inflammation

The Journal of immunology (1950), 2016-07, Vol.197 (1), p.387-389

2016

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CS10-4. Function of SOCS3 on Macrophage Polarization and Inflammation

Cytokine (Philadelphia, Pa.), 2011-10, Vol.56 (1), p.58-58

2011

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Abstract 2334: Sialylation of the TNFR1 death receptor promotes cancer cell survival

Cancer research (Chicago, Ill.), 2018-07, Vol.78 (13_Supplement), p.2334-2334

2018

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AMP-activated protein kinase (AMPK) Restricts Interferon Gamma Signaling

The Journal of immunology (1950), 2012-11, Vol.190 (1), p.372-380

2012

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ST6Gal-I Sialyltransferase Regulates the Activity of Surface Receptors to Control Cell Signaling in Cancer and Immune Cells

Holdbrooks, Andrew T

2018

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Abstract 3327: The tumor associated sialyltransferase ST6Gal-I promotes a cancer stem cell phenotype and upregulates stem-related transcription factors

Cancer research (Chicago, Ill.), 2016-07, Vol.76 (14_Supplement), p.3327-3327

2016

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Therapeutic Efficacy of Suppressing the JAK/STAT Pathway in Multiple Models of EAE1

The Journal of immunology (1950), 2013-12, Vol.192 (1), p.59-72

2013

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Abstract 2504: Sialylation of EGFR by the glycosyltransferase ST6Gal-I results in receptor activation and resistance to gefitinib mediated apoptosis

Cancer research (Chicago, Ill.), 2018-07, Vol.78 (13_Supplement), p.2504-2504

2018

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Abstract 3566: Glycosylation of the TNFR1 death receptor controls cell fate

Cancer research (Chicago, Ill.), 2016-07, Vol.76 (14_Supplement), p.3566-3566

2016

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Targeting the JAK/STAT pathway in the treatment of Parkinson’s disease (THER6P.847)

The Journal of immunology (1950), 2014-05, Vol.192 (1_Supplement), p.201-201.3

2014

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Critical function of myeloid SOCS3 is in limiting the neuroinflammation of EAE (147.21)

The Journal of immunology (1950), 2011-04, Vol.186 (1_Supplement), p.147-147.21

2011

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ST6Gal-I sialyltransferase promotes tumor necrosis factor (TNF)-mediated cancer cell survival via sialylation of the TNF receptor 1 (TNFR1) death receptor

Regulation of inflammatory signaling by the ST6Gal-I sialyltransferase

The Tumor-Associated Glycosyltransferase ST6Gal-I Regulates Stem Cell Transcription Factors and Confers a Cancer Stem Cell Phenotype

Therapeutic efficacy of suppressing the Jak/STAT pathway in multiple models of experimental autoimmune encephalomyelitis

Signal transducer and activator of transcription-3/suppressor of cytokine signaling-3 (STAT3/SOCS3) axis in myeloid cells regulates neuroinflammation

Sialylation of EGFR by the ST6Gal-I sialyltransferase promotes EGFR activation and resistance to gefitinib-mediated cell death

Preferential Recruitment of Neutrophils into the Cerebellum and Brainstem Contributes to the Atypical Experimental Autoimmune Encephalomyelitis Phenotype

AMP-activated protein kinase restricts IFN-γ signaling

Correction: SOCS3 Deficiency Promotes M1 Macrophage Polarization and Inflammation

CS10-4. Function of SOCS3 on Macrophage Polarization and Inflammation

Abstract 2334: Sialylation of the TNFR1 death receptor promotes cancer cell survival

AMP-activated protein kinase (AMPK) Restricts Interferon Gamma Signaling

ST6Gal-I Sialyltransferase Regulates the Activity of Surface Receptors to Control Cell Signaling in Cancer and Immune Cells

Abstract 3327: The tumor associated sialyltransferase ST6Gal-I promotes a cancer stem cell phenotype and upregulates stem-related transcription factors

Therapeutic Efficacy of Suppressing the JAK/STAT Pathway in Multiple Models of EAE1

Abstract 2504: Sialylation of EGFR by the glycosyltransferase ST6Gal-I results in receptor activation and resistance to gefitinib mediated apoptosis

Abstract 3566: Glycosylation of the TNFR1 death receptor controls cell fate

Targeting the JAK/STAT pathway in the treatment of Parkinson’s disease (THER6P.847)

Critical function of myeloid SOCS3 is in limiting the neuroinflammation of EAE (147.21)

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