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Cellular senescence and its effector programs

Genes & development, 2014-01, Vol.28 (2), p.99-114

2014

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Somatic mutations and clonal dynamics in healthy and cirrhotic human liver

Nature (London), 2019-10, Vol.574 (7779), p.538-542

2019

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NOTCH1 mediates a switch between two distinct secretomes during senescence

Nature cell biology, 2016-09, Vol.18 (9), p.979-992

2016

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Lipid Remodeling in Hepatocyte Proliferation and Hepatocellular Carcinoma

Hepatology (Baltimore, Md.), 2021-03, Vol.73 (3), p.1028-1044

2021

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Mouse models of hepatocyte biology – Known unknowns

Journal of hepatology, 2023-05, Vol.78 (5), p.898-900

Hoare, Matthew

2023

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Robustness of radiomic features in CT images with different slice thickness, comparing liver tumour and muscle

Scientific reports, 2021-04, Vol.11 (1), p.8262-8262, Article 8262

2021

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NOTCH-mediated non-cell autonomous regulation of chromatin structure during senescence

Nature communications, 2018-05, Vol.9 (1), p.1840-15, Article 1840

2018

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Hepatocyte expression of the senescence marker p21 is linked to fibrosis and an adverse liver-related outcome in alcohol-related liver disease

PloS one, 2013-09, Vol.8 (9), p.e72904

2013

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Transmitting senescence to the cell neighbourhood

Nature cell biology, 2013-08, Vol.15 (8), p.887-889

2013

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Acetyl-CoA metabolism drives epigenome change and contributes to carcinogenesis risk in fatty liver disease

Genome medicine, 2022-06, Vol.14 (1), p.67-16, Article 67

2022

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Novel immunotherapeutics against LGR5 to target multiple cancer types

EMBO molecular medicine, 2024-08, Vol.16 (9), p.2233-2261

2024

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The clinical value of the hepatic venous pressure gradient in patients undergoing hepatic resection for hepatocellular carcinoma with or without liver cirrhosis

Open medicine (Warsaw, Poland), 2024-01, Vol.19 (1), p.20230851-20230851

2024

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Correction: Acetyl-CoA metabolism drives epigenome change and contributes to carcinogenesis risk in fatty liver disease

Genome medicine, 2023-05, Vol.15 (1), p.38-38, Article 38

2023

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Persistence of Hepatitis C Virus Traces after Spontaneous Resolution of Hepatitis C

PloS one, 2015-10, Vol.10 (10), p.e0140312-e0140312

2015

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Replication-timing boundaries facilitate cell-type and species-specific regulation of a rearranged human chromosome in mouse

Human molecular genetics, 2012-10, Vol.21 (19), p.4162-4170

2012

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The senescent secretome drives PLVAP expression in cultured human hepatic endothelial cells to promote monocyte transmigration

iScience, 2023-10, Vol.26 (10), p.107966-107966, Article 107966

2023

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Notch and Senescence

Advances in experimental medicine and biology, 2018, Vol.1066, p.299-318

2018

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Plasma membrane proteomics identifies Notch1 as a potential regulator of ras-induced senescence

The Lancet (British edition), 2013-02, Vol.381, p.S49-S49

2013

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NOTCH and the 2 SASPs of senescence

Cell cycle (Georgetown, Tex.), 2017-02, Vol.16 (3), p.239-240

2017

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Ageing, telomeres, senescence, and liver injury

Journal of hepatology, 2010-11, Vol.53 (5), p.950-961

2010

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Histological changes in HCV antibody–positive, HCV RNA–negative subjects suggest persistent virus infection

Hepatology (Baltimore, Md.), 2008-12, Vol.48 (6), p.1737-1745

2008

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The Power Behind the Throne: Senescence and the Hallmarks of Cancer

Annual review of cancer biology, 2018-03, Vol.2 (1), p.175-194

2018

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Plasmalemma vesicle-associated protein expression is driven by senescent cell-endothelial crosstalk and shapes the immune landscape in chronic liver disease

Journal of hepatology, 2022-07, Vol.77, p.S195

2022

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A minority of somatically mutated genes in pre‐existing fatty liver disease have prognostic importance in the development of NAFLD

Liver international, 2022-08, Vol.42 (8), p.1823-1835

2022

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Spatial lipidomics reveal dysregulated sphingolipid metabolism in liver fibrosis

Journal of hepatology, 2023-06, Vol.78, p.S323-S324

2023

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Cellular senescence and its effector programs

Somatic mutations and clonal dynamics in healthy and cirrhotic human liver

NOTCH1 mediates a switch between two distinct secretomes during senescence

Lipid Remodeling in Hepatocyte Proliferation and Hepatocellular Carcinoma

Mouse models of hepatocyte biology – Known unknowns

Robustness of radiomic features in CT images with different slice thickness, comparing liver tumour and muscle

NOTCH-mediated non-cell autonomous regulation of chromatin structure during senescence

Hepatocyte expression of the senescence marker p21 is linked to fibrosis and an adverse liver-related outcome in alcohol-related liver disease

Transmitting senescence to the cell neighbourhood

Acetyl-CoA metabolism drives epigenome change and contributes to carcinogenesis risk in fatty liver disease

Novel immunotherapeutics against LGR5 to target multiple cancer types

The clinical value of the hepatic venous pressure gradient in patients undergoing hepatic resection for hepatocellular carcinoma with or without liver cirrhosis

Correction: Acetyl-CoA metabolism drives epigenome change and contributes to carcinogenesis risk in fatty liver disease

Persistence of Hepatitis C Virus Traces after Spontaneous Resolution of Hepatitis C

Replication-timing boundaries facilitate cell-type and species-specific regulation of a rearranged human chromosome in mouse

The senescent secretome drives PLVAP expression in cultured human hepatic endothelial cells to promote monocyte transmigration

Notch and Senescence

Plasma membrane proteomics identifies Notch1 as a potential regulator of ras-induced senescence

NOTCH and the 2 SASPs of senescence

Ageing, telomeres, senescence, and liver injury

Histological changes in HCV antibody–positive, HCV RNA–negative subjects suggest persistent virus infection

The Power Behind the Throne: Senescence and the Hallmarks of Cancer

Plasmalemma vesicle-associated protein expression is driven by senescent cell-endothelial crosstalk and shapes the immune landscape in chronic liver disease

A minority of somatically mutated genes in pre‐existing fatty liver disease have prognostic importance in the development of NAFLD

Spatial lipidomics reveal dysregulated sphingolipid metabolism in liver fibrosis

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