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The bromodomain protein BRD4 regulates the KEAP1/NRF2-dependent oxidative stress response

Cell death & disease, 2014-04, Vol.5 (4), p.e1195-e1195

2014

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The role of oxygen metabolism for the pathological phenotype of Fanconi anemia

Human genetics, 1997-06, Vol.99 (6), p.710-719

1997

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Suboptimal Action of NF-κB in Fanconi Anemia Cells Results from Low Levels of Thioredoxin

Biological chemistry, 2003-11, Vol.384 (10-11), p.1501-1507

2003

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The cellular control enzyme polyADP ribosyl transferase is eliminated in cultured Fanconi anemia fibroblasts at confluency

Biological chemistry, 2003-01, Vol.384 (1), p.169-174

2003

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Characterization of recombinant human nicotinamide mononucleotide adenylyl transferase (NMNAT), a nuclear enzyme essential for NAD synthesis

FEBS letters, 2001-03, Vol.492 (1), p.95-100

2001

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Human Nuclear NAD+ADP-Ribosyltransferase: Localization of the Gene on Chromosome 1q41-q42 and Expression of an Active Human Enzyme in Escherichia coli

Proceedings of the National Academy of Sciences - PNAS, 1989-05, Vol.86 (10), p.3514-3518

1989

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Aging and chromosomal instability

Reviews of physiology, biochemistry and pharmacology, 1999-01, Vol.139, p.141

1999

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Physical and functional interaction of the Werner syndrome protein with poly-ADP ribosyl transferase

FEBS letters, 2003-11, Vol.554 (1-2), p.55-58

2003

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Nuclear deformation characterizes Werner syndrome cells

Cell biology international, 2005-12, Vol.29 (12), p.1032-1037

2005

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Accelerated telomere shortening in Fanconi anemia fibroblasts – a longitudinal study

FEBS letters, 2001-09, Vol.506 (1), p.22-26

2001

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Fanconi anaemia proteins: Major roles in cell protection against oxidative damage

BioEssays, 2003-06, Vol.25 (6), p.589-595

2003

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Overexpression of thioredoxin in Fanconi anemia fibroblasts prevents the cytotoxic and DNA damaging effect of mitomycin C and diepoxybutane

FEBS letters, 1998-01, Vol.422 (1), p.99-102

1998

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Overexpressed thioredoxin compensates Fanconi anemia related chromosomal instability

Oncogene, 2002-04, Vol.21 (15), p.2406-2412

2002

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DNA repair dependent NAD+ metabolism is impaired in cells from patients with Fanconi's anemia

The EMBO journal, 1983-01, Vol.2 (3), p.303-307

1983

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Overexpressed thioredoxin compensates Fanconi anemia related chromosomal instability

Oncogene, 2002-04, Vol.21 (15), p.2406-2412

2002

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Thioredoxin, a regulator of gene expression

Oncogene, 2004-03, Vol.23 (12), p.2146-2152

2004

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Regulation of the human poly(ADP-ribosyl) transferase promoter via alternative DNA racket structures

Biochimie, 1995, Vol.77 (6), p.480-485

1995

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Fluorescence in situ mapping of the human nuclear NAD+ ADP-ribosyltransferase gene (ADPRT) and two secondary sites to human chromosomal bands 1q42, 13q34, and 14q24

Cytogenetic and genome research, 1992, Vol.61 (3), p.172-174

1992

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Duchenne muscular dystrophy : lack of differences in the expression of endogenous carbohydrate- and heparin-binding proteins (lectins) in cultured fibroblasts

European journal of cell biology, 1993-10, Vol.62 (1), p.173-181

1993

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Lipmann Symposium. Energy, Regulation and Biosynthesis in Molecular Biology

2020

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Ultrastructure of Fanconi anemia fibroblasts

Journal of cell science, 1989-08, Vol.93 (4), p.651-665

1989

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Involvement of the Fanconi anemia protein FA-C in repair processes of oxidative DNA damages

FEBS letters, 1998-11, Vol.440 (1), p.103-106

1998

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Chromosomal Instability of Fanconi Anemia Cells Is Not the Consequence of a Defective Repair Activity of the Ribosomal Protein S3

Biochemical and biophysical research communications, 1999-10, Vol.264 (2), p.518-524

1999

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DNA repair in human cells: in Cockayne Syndrome cells rejoining of DNA strands is impaired

European journal of cell biology, 1986-08, Vol.41 (2), p.352-355

1986

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Radiation sensitivity of Down's syndrome fibroblasts might be due to overexpressed Cu/Zn-superoxide dismutase (EC 1.15.1.1)

European journal of cell biology, 1989-02, Vol.48 (1), p.79-87

1989

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The bromodomain protein BRD4 regulates the KEAP1/NRF2-dependent oxidative stress response

The role of oxygen metabolism for the pathological phenotype of Fanconi anemia

Suboptimal Action of NF-κB in Fanconi Anemia Cells Results from Low Levels of Thioredoxin

The cellular control enzyme polyADP ribosyl transferase is eliminated in cultured Fanconi anemia fibroblasts at confluency

Characterization of recombinant human nicotinamide mononucleotide adenylyl transferase (NMNAT), a nuclear enzyme essential for NAD synthesis

Human Nuclear NAD+ADP-Ribosyltransferase: Localization of the Gene on Chromosome 1q41-q42 and Expression of an Active Human Enzyme in Escherichia coli

Aging and chromosomal instability

Physical and functional interaction of the Werner syndrome protein with poly-ADP ribosyl transferase

Nuclear deformation characterizes Werner syndrome cells

Accelerated telomere shortening in Fanconi anemia fibroblasts – a longitudinal study

Fanconi anaemia proteins: Major roles in cell protection against oxidative damage

Overexpression of thioredoxin in Fanconi anemia fibroblasts prevents the cytotoxic and DNA damaging effect of mitomycin C and diepoxybutane

Overexpressed thioredoxin compensates Fanconi anemia related chromosomal instability

DNA repair dependent NAD+ metabolism is impaired in cells from patients with Fanconi's anemia

Overexpressed thioredoxin compensates Fanconi anemia related chromosomal instability

Thioredoxin, a regulator of gene expression

Regulation of the human poly(ADP-ribosyl) transferase promoter via alternative DNA racket structures

Fluorescence in situ mapping of the human nuclear NAD+ ADP-ribosyltransferase gene (ADPRT) and two secondary sites to human chromosomal bands 1q42, 13q34, and 14q24

Duchenne muscular dystrophy : lack of differences in the expression of endogenous carbohydrate- and heparin-binding proteins (lectins) in cultured fibroblasts

Lipmann Symposium. Energy, Regulation and Biosynthesis in Molecular Biology

Ultrastructure of Fanconi anemia fibroblasts

Involvement of the Fanconi anemia protein FA-C in repair processes of oxidative DNA damages

Chromosomal Instability of Fanconi Anemia Cells Is Not the Consequence of a Defective Repair Activity of the Ribosomal Protein S3

DNA repair in human cells: in Cockayne Syndrome cells rejoining of DNA strands is impaired

Radiation sensitivity of Down's syndrome fibroblasts might be due to overexpressed Cu/Zn-superoxide dismutase (EC 1.15.1.1)

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