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Primary cilia on muscle stem cells are critical to maintain regenerative capacity and are lost during aging

Nature communications, 2022-03, Vol.13 (1), p.1439-1439, Article 1439

2022

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Metabolic profiling shows pre‐existing mitochondrial dysfunction contributes to muscle loss in a model of ICU‐acquired weakness

Journal of cachexia, sarcopenia and muscle, 2020-10, Vol.11 (5), p.1321-1335

2020

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Human and mouse skeletal muscle stem cells: convergent and divergent mechanisms of myogenesis

PloS one, 2014-02, Vol.9 (2), p.e90398-e90398

2014

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Fast skeletal muscle troponin activator tirasemtiv increases muscle function and performance in the B6SJL-SOD1G93A ALS mouse model

PloS one, 2014-05, Vol.9 (5), p.e96921-e96921

2014

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Ablation of Ventricular Myosin Regulatory Light Chain Phosphorylation in Mice Causes Cardiac Dysfunction in Situ and Affects Neighboring Myofilament Protein Phosphorylation

The Journal of biological chemistry, 2009-02, Vol.284 (8), p.5097-5106

2009

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H2O2 alters rat cardiac sarcomere function and protein phosphorylation through redox signaling

American journal of physiology. Heart and circulatory physiology, 2010-09, Vol.299 (3), p.H723-H730

2010

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HIF prolyl hydroxylase inhibition protects skeletal muscle from eccentric contraction-induced injury

Skeletal muscle, 2018-11, Vol.8 (1), p.35-35, Article 35

2018

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Prostaglandin D2 signaling is not involved in the recovery of rat hind limb tendons from injury

Physiological reports, 2019-11, Vol.7 (22), p.e14289-n/a

2019

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Lack of evidence for GDF11 as a rejuvenator of aged skeletal muscle satellite cells

Aging cell, 2016-06, Vol.15 (3), p.582-584

2016

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Fast skeletal muscle troponin activator CK‐2066260 increases fatigue resistance by reducing the energetic cost of muscle contraction

The Journal of physiology, 2019-09, Vol.597 (17), p.4615-4625

2019

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Correction to: HIF prolyl hydroxylase inhibition protects skeletal muscle from eccentric contraction induced injury

Skeletal muscle, 2018-12, Vol.8 (1), p.38-38, Article 38

2018

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Activation of fast skeletal muscle troponin as a potential therapeutic approach for treating neuromuscular diseases

Nature medicine, 2012-03, Vol.18 (3), p.452-455

2012

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The Ca2+ sensitizer CK‐2066260 increases myofibrillar Ca2+ sensitivity and submaximal force selectively in fast skeletal muscle

The Journal of physiology, 2017-03, Vol.595 (5), p.1657-1670

2017

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Mechano-growth factor peptide, the COOH terminus of unprocessed insulin-like growth factor 1, has no apparent effect on myoblasts or primary muscle stem cells

American journal of physiology: endocrinology and metabolism, 2014-01, Vol.306 (2), p.E150-E156

2014

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A Dominant Role of Cardiac Molecular Motors in the Intrinsic Regulation of Ventricular Ejection and Relaxation

Physiology (Bethesda, Md.), 2007-04, Vol.22 (2), p.73-80

2007

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Desensitization of myofilaments to Ca2+ as a therapeutic target for hypertrophic cardiomyopathy with mutations in thin filament proteins

Circulation. Cardiovascular genetics, 2014-04, Vol.7 (2), p.132

2014

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Discovery of Tirasemtiv, the First Direct Fast Skeletal Muscle Troponin Activator

ACS medicinal chemistry letters, 2018-04, Vol.9 (4), p.354-358

2018

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The Ca 2+ sensitizer CK-2066260 increases myofibrillar Ca 2+ sensitivity and submaximal force selectively in fast skeletal muscle

The Journal of physiology, 2017-03, Vol.595 (5), p.1657-1670

2017

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Tyrosine Phosphorylation Modifies Protein Kinase C δ-dependent Phosphorylation of Cardiac Troponin I

The Journal of biological chemistry, 2008-08, Vol.283 (33), p.22680-22689

2008

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Phosphorylation of Cardiac Troponin I at Protein Kinase C Site Threonine 144 Depresses Cooperative Activation of Thin Filaments

The Journal of biological chemistry, 2010-04, Vol.285 (16), p.11810-11817

2010

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Prostaglandin D 2 signaling is not involved in the recovery of rat hind limb tendons from injury

Physiological reports, 2019-11, Vol.7 (22), p.e14289

2019

Volltextzugriff (PDF)

Protein kinase C depresses cardiac myocyte power output and attenuates myofilament responses induced by protein kinase A

Journal of muscle research and cell motility, 2012-12, Vol.33 (6), p.439-448

2012

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Inorganic phosphate speeds loaded shortening in rat skinned cardiac myocytes

American Journal of Physiology: Cell Physiology, 2004-08, Vol.287 (2), p.C500-C507

2004

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Functional effects of a tropomyosin mutation linked to FHC contribute to maladaptation during acidosis

Journal of molecular and cellular cardiology, 2011-03, Vol.50 (3), p.442-450

2011

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Porcine cardiac myocyte power output is increased after chronic exercise training

Journal of applied physiology (1985), 2006-07, Vol.101 (1), p.40-46

2006

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Primary cilia on muscle stem cells are critical to maintain regenerative capacity and are lost during aging

Metabolic profiling shows pre‐existing mitochondrial dysfunction contributes to muscle loss in a model of ICU‐acquired weakness

Human and mouse skeletal muscle stem cells: convergent and divergent mechanisms of myogenesis

Fast skeletal muscle troponin activator tirasemtiv increases muscle function and performance in the B6SJL-SOD1G93A ALS mouse model

Ablation of Ventricular Myosin Regulatory Light Chain Phosphorylation in Mice Causes Cardiac Dysfunction in Situ and Affects Neighboring Myofilament Protein Phosphorylation

H2O2 alters rat cardiac sarcomere function and protein phosphorylation through redox signaling

HIF prolyl hydroxylase inhibition protects skeletal muscle from eccentric contraction-induced injury

Prostaglandin D2 signaling is not involved in the recovery of rat hind limb tendons from injury

Lack of evidence for GDF11 as a rejuvenator of aged skeletal muscle satellite cells

Fast skeletal muscle troponin activator CK‐2066260 increases fatigue resistance by reducing the energetic cost of muscle contraction

Correction to: HIF prolyl hydroxylase inhibition protects skeletal muscle from eccentric contraction induced injury

Activation of fast skeletal muscle troponin as a potential therapeutic approach for treating neuromuscular diseases

The Ca2+ sensitizer CK‐2066260 increases myofibrillar Ca2+ sensitivity and submaximal force selectively in fast skeletal muscle

Mechano-growth factor peptide, the COOH terminus of unprocessed insulin-like growth factor 1, has no apparent effect on myoblasts or primary muscle stem cells

A Dominant Role of Cardiac Molecular Motors in the Intrinsic Regulation of Ventricular Ejection and Relaxation

Desensitization of myofilaments to Ca2+ as a therapeutic target for hypertrophic cardiomyopathy with mutations in thin filament proteins

Discovery of Tirasemtiv, the First Direct Fast Skeletal Muscle Troponin Activator

The Ca 2+ sensitizer CK-2066260 increases myofibrillar Ca 2+ sensitivity and submaximal force selectively in fast skeletal muscle

Tyrosine Phosphorylation Modifies Protein Kinase C δ-dependent Phosphorylation of Cardiac Troponin I

Phosphorylation of Cardiac Troponin I at Protein Kinase C Site Threonine 144 Depresses Cooperative Activation of Thin Filaments

Prostaglandin D 2 signaling is not involved in the recovery of rat hind limb tendons from injury

Protein kinase C depresses cardiac myocyte power output and attenuates myofilament responses induced by protein kinase A

Inorganic phosphate speeds loaded shortening in rat skinned cardiac myocytes

Functional effects of a tropomyosin mutation linked to FHC contribute to maladaptation during acidosis

Porcine cardiac myocyte power output is increased after chronic exercise training

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