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Proteome-wide systems genetics identifies UFMylation as a regulator of skeletal muscle function

eLife, 2022-12, Vol.11

2022

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Evidence for disrupted copper availability in human spinal cord supports CuII(atsm) as a treatment option for sporadic cases of ALS

Scientific reports, 2024-03, Vol.14 (1), p.5929-5929, Article 5929

2024

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Sex-dependent effects of amyloid precursor-like protein 2 in the SOD1-G37R transgenic mouse model of MND

Cellular and molecular life sciences : CMLS, 2021-10, Vol.78 (19-20), p.6605-6630

2021

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Microglial ferroptotic stress causes non-cell autonomous neuronal death

Molecular neurodegeneration, 2024-02, Vol.19 (1), p.14-14, Article 14

2024

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CuII(atsm) improves the neurological phenotype and survival of SOD1G93A mice and selectively increases enzymatically active SOD1 in the spinal cord

Scientific reports, 2017-02, Vol.7 (1), p.42292-42292, Article 42292

2017

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Superoxide Dismutase 1 in Health and Disease: How a Frontline Antioxidant Becomes Neurotoxic

Angewandte Chemie International Edition, 2021-04, Vol.60 (17), p.9215-9246

2021

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CuII(atsm) significantly decreases microglial reactivity in patients with sporadic amyotrophic lateral sclerosis

Neuropathology and applied neurobiology, 2023-10, Vol.49 (5), p.e12938-n/a

2023

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Fundamental Neurochemistry Review: Copper availability as a potential therapeutic target in progressive supranuclear palsy: Insight from other neurodegenerative diseases

Journal of neurochemistry, 2023-11, Vol.167 (3), p.337-346

2023

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Cu II (atsm) significantly decreases microglial reactivity in patients with sporadic amyotrophic lateral sclerosis

Neuropathology and applied neurobiology, 2023-10, Vol.49 (5), p.e12938

2023

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Metal-deficient SOD1 in amyotrophic lateral sclerosis

Journal of molecular medicine (Berlin, Germany), 2015-05, Vol.93 (5), p.481-487

2015

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The accumulation of enzymatically inactive cuproenzymes is a CNS-specific phenomenon of the SOD1G37R mouse model of ALS and can be restored by overexpressing the human copper transporter hCTR1

Experimental neurology, 2018-09, Vol.307, p.118-128

2018

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Transdermal Application of Soluble CuII(atsm) Increases Brain and Spinal Cord Uptake Compared to Gavage with an Insoluble Suspension

Neuroscience, 2023-01, Vol.509, p.125-131

2023

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Copper-ATSM as a Treatment for ALS: Support from Mutant SOD1 Models and Beyond

Life (Basel, Switzerland), 2020-11, Vol.10 (11), p.271

2020

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Integrated elemental analysis supports targeting copper perturbations as a therapeutic strategy in multiple sclerosis

Neurotherapeutics, 2024-09, Vol.21 (5), p.e00432, Article e00432

2024

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A versatile quantitative microdroplet elemental imaging method optimised for integration in biochemical workflows for low-volume samples

Analytical and bioanalytical chemistry, 2019-01, Vol.411 (3), p.603-616

2019

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Evidence for decreased copper associated with demyelination in the corpus callosum of cuprizone-treated mice

Metallomics, 2024-01, Vol.16 (1)

2024

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An integrated mass spectrometry imaging and digital pathology workflow for objective detection of colorectal tumours by unique atomic signatures

Chemical science (Cambridge), 2021-08, Vol.12 (3), p.1321-1333

2021

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Localized changes to glycogen synthase kinase-3 and collapsin response mediator protein-2 in the Huntington's disease affected brain

Human molecular genetics, 2014-08, Vol.23 (15), p.4051-4063

2014

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Superoxide Dismutase 1 in Health and Disease: HowaFrontline Antioxidant Becomes Neurotoxic

Angewandte Chemie International Edition, 2021-04, Vol.60 (17), p.9215-9246

International ed. in English, 2021

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Superoxide Dismutase 1 in Health and Disease: How a Frontline Antioxidant Becomes Neurotoxic

Angewandte Chemie, 2021-04, Vol.133 (17), p.9299-9330

2021

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Superoxide Dismutase 1 in Health and Disease: HowaFrontline Antioxidant Becomes Neurotoxic

Angewandte Chemie, 2021-04, Vol.133 (17), p.9299-9330

2021

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Transdermal Application of Soluble Cu II (atsm) Increases Brain and Spinal Cord Uptake Compared to Gavage with an Insoluble Suspension

Neuroscience, 2023-01, Vol.509, p.125

2023

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Evidence for disrupted copper availability in human spinal cord supports Cu II (atsm) as a treatment option for sporadic cases of ALS

Scientific reports, 2024-03, Vol.14 (1), p.5929

2024

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The accumulation of enzymatically inactive cuproenzymes is a CNS-specific phenomenon of the SOD1 G37R mouse model of ALS and can be restored by overexpressing the human copper transporter hCTR1

Experimental neurology, 2018-09, Vol.307, p.118

2018

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Proteome-wide systems genetics identifies UFMylation as a regulator of skeletal muscle function

Evidence for disrupted copper availability in human spinal cord supports CuII(atsm) as a treatment option for sporadic cases of ALS

Sex-dependent effects of amyloid precursor-like protein 2 in the SOD1-G37R transgenic mouse model of MND

Microglial ferroptotic stress causes non-cell autonomous neuronal death

CuII(atsm) improves the neurological phenotype and survival of SOD1G93A mice and selectively increases enzymatically active SOD1 in the spinal cord

Superoxide Dismutase 1 in Health and Disease: How a Frontline Antioxidant Becomes Neurotoxic

TDP-43 Triggers Mitochondrial DNA Release via mPTP to Activate cGAS/STING in ALS

CuII(atsm) significantly decreases microglial reactivity in patients with sporadic amyotrophic lateral sclerosis

Fundamental Neurochemistry Review: Copper availability as a potential therapeutic target in progressive supranuclear palsy: Insight from other neurodegenerative diseases

Cu II (atsm) significantly decreases microglial reactivity in patients with sporadic amyotrophic lateral sclerosis

Metal-deficient SOD1 in amyotrophic lateral sclerosis

The accumulation of enzymatically inactive cuproenzymes is a CNS-specific phenomenon of the SOD1G37R mouse model of ALS and can be restored by overexpressing the human copper transporter hCTR1

Transdermal Application of Soluble CuII(atsm) Increases Brain and Spinal Cord Uptake Compared to Gavage with an Insoluble Suspension

Copper-ATSM as a Treatment for ALS: Support from Mutant SOD1 Models and Beyond

Integrated elemental analysis supports targeting copper perturbations as a therapeutic strategy in multiple sclerosis

A versatile quantitative microdroplet elemental imaging method optimised for integration in biochemical workflows for low-volume samples

Evidence for decreased copper associated with demyelination in the corpus callosum of cuprizone-treated mice

An integrated mass spectrometry imaging and digital pathology workflow for objective detection of colorectal tumours by unique atomic signatures

Localized changes to glycogen synthase kinase-3 and collapsin response mediator protein-2 in the Huntington's disease affected brain

Superoxide Dismutase 1 in Health and Disease: HowaFrontline Antioxidant Becomes Neurotoxic

Superoxide Dismutase 1 in Health and Disease: How a Frontline Antioxidant Becomes Neurotoxic

Superoxide Dismutase 1 in Health and Disease: HowaFrontline Antioxidant Becomes Neurotoxic

Transdermal Application of Soluble Cu II (atsm) Increases Brain and Spinal Cord Uptake Compared to Gavage with an Insoluble Suspension

Evidence for disrupted copper availability in human spinal cord supports Cu II (atsm) as a treatment option for sporadic cases of ALS

The accumulation of enzymatically inactive cuproenzymes is a CNS-specific phenomenon of the SOD1 G37R mouse model of ALS and can be restored by overexpressing the human copper transporter hCTR1

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