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Transcriptional Control by the SMADs

Cold Spring Harbor perspectives in biology, 2016-10, Vol.8 (10), p.a022079

Hill, Caroline S

2016

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A new class of small molecule inhibitor of BMP signaling

PloS one, 2013-04, Vol.8 (4), p.e62721

2013

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TGFβ-SMAD signal transduction: molecular specificity and functional flexibility

Nature reviews. Molecular cell biology, 2007-12, Vol.8 (12), p.970-982

2007

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Spatial and temporal control of NODAL signaling

Current opinion in cell biology, 2018-04, Vol.51, p.50-57

Hill, Caroline S

2018

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Beyond TGFβ: roles of other TGFβ superfamily members in cancer

Nature reviews. Cancer, 2013-05, Vol.13 (5), p.328

2013

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Nucleocytoplasmic shuttling of Smad proteins

Cell research, 2009-01, Vol.19 (1), p.36-46

Hill, Caroline S

2009

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TGF-β signaling to chromatin: How Smads regulate transcription during self-renewal and differentiation

Seminars in cell & developmental biology, 2014-08, Vol.32, p.107-118

2014

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Framing of Abortion and Church-State Relations in Russian Orthodox Online Portals

Religions (Basel, Switzerland ), 2021-12, Vol.12 (12), p.1084

Hill, Caroline

2021

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Establishment and interpretation of NODAL and BMP signaling gradients in early vertebrate development

Current topics in developmental biology, 2022, Vol.149, p.311-340

Hill, Caroline S

2022

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Mesenchymal Cancer Cell-Stroma Crosstalk Promotes Niche Activation, Epithelial Reversion, and Metastatic Colonization

Cell reports (Cambridge), 2015-12, Vol.13 (11), p.2456-2469

2015

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Localized and reversible TGFβ signalling switches breast cancer cells from cohesive to single cell motility

Nature cell biology, 2009-11, Vol.11 (11), p.1287-1296

2009

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TGF-β Superfamily Signaling in Embryonic Development and Homeostasis

Developmental cell, 2009-03, Vol.16 (3), p.329-343

2009

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How the Smads regulate transcription

The international journal of biochemistry & cell biology, 2008, Vol.40 (3), p.383-408

2008

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Recruitment of TIF1γ to Chromatin via Its PHD Finger-Bromodomain Activates Its Ubiquitin Ligase and Transcriptional Repressor Activities

Molecular cell, 2011-07, Vol.43 (1), p.85-96

2011

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A Temporal Window for Signal Activation Dictates the Dimensions of a Nodal Signaling Domain

Developmental cell, 2015-10, Vol.35 (2), p.175-185

2015

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Context-dependent TGFβ family signalling in cell fate regulation

Nature reviews. Molecular cell biology, 2023-12, Vol.24 (12), p.876

2023

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Nodal signaling establishes a competency window for stochastic cell fate switching

Developmental cell, 2022-12, Vol.57 (23), p.2604-2622.e5

2022

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Older adults' lived experiences of physical rehabilitation for acquired brain injury and their perceptions of well-being: A qualitative phenomenological study

Journal of clinical nursing, 2024-03, Vol.33 (3), p.1134-1149

2024

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The ventral to dorsal BMP activity gradient in the early zebrafish embryo is determined by graded expression of BMP ligands

Developmental biology, 2013-06, Vol.378 (2), p.170-182

2013

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A systematic review of the qualitative literature on older individuals’ experiences of care and well‐being during physical rehabilitation for acquired brain injury

Journal of advanced nursing, 2022-02, Vol.78 (2), p.377-394

2022

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Transforming Growth Factor β-Induced Smad1/5 Phosphorylation in Epithelial Cells Is Mediated by Novel Receptor Complexes and Is Essential for Anchorage-Independent Growth

Molecular and Cellular Biology, 2008-11, Vol.28 (22), p.6889-6902

2008

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Alterations in components of the TGF-β superfamily signaling pathways in human cancer

Cytokine & growth factor reviews, 2006-02, Vol.17 (1), p.41-58

2006

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Smad4 Dependency Defines Two Classes of Transforming Growth Factor β (TGF-β) Target Genes and Distinguishes TGF-β-Induced Epithelial-Mesenchymal Transition from Its Antiproliferative and Migratory Responses

Molecular and Cellular Biology, 2005-09, Vol.25 (18), p.8108-8125

2005

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TGF-β uses a novel mode of receptor activation to phosphorylate SMAD1/5 and induce epithelial-to-mesenchymal transition

eLife, 2018-01, Vol.7

2018

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Hierarchical patterning modes orchestrate hair follicle morphogenesis

PLoS biology, 2017-07, Vol.15 (7), p.e2002117-e2002117

2017

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Transcriptional Control by the SMADs

A new class of small molecule inhibitor of BMP signaling

TGFβ-SMAD signal transduction: molecular specificity and functional flexibility

Spatial and temporal control of NODAL signaling

Beyond TGFβ: roles of other TGFβ superfamily members in cancer

Nucleocytoplasmic shuttling of Smad proteins

TGF-β signaling to chromatin: How Smads regulate transcription during self-renewal and differentiation

Framing of Abortion and Church-State Relations in Russian Orthodox Online Portals

Establishment and interpretation of NODAL and BMP signaling gradients in early vertebrate development

Mesenchymal Cancer Cell-Stroma Crosstalk Promotes Niche Activation, Epithelial Reversion, and Metastatic Colonization

Localized and reversible TGFβ signalling switches breast cancer cells from cohesive to single cell motility

TGF-β Superfamily Signaling in Embryonic Development and Homeostasis

How the Smads regulate transcription

Recruitment of TIF1γ to Chromatin via Its PHD Finger-Bromodomain Activates Its Ubiquitin Ligase and Transcriptional Repressor Activities

A Temporal Window for Signal Activation Dictates the Dimensions of a Nodal Signaling Domain

Context-dependent TGFβ family signalling in cell fate regulation

Nodal signaling establishes a competency window for stochastic cell fate switching

Older adults' lived experiences of physical rehabilitation for acquired brain injury and their perceptions of well-being: A qualitative phenomenological study

The ventral to dorsal BMP activity gradient in the early zebrafish embryo is determined by graded expression of BMP ligands

A systematic review of the qualitative literature on older individuals’ experiences of care and well‐being during physical rehabilitation for acquired brain injury

Transforming Growth Factor β-Induced Smad1/5 Phosphorylation in Epithelial Cells Is Mediated by Novel Receptor Complexes and Is Essential for Anchorage-Independent Growth

Alterations in components of the TGF-β superfamily signaling pathways in human cancer

Smad4 Dependency Defines Two Classes of Transforming Growth Factor β (TGF-β) Target Genes and Distinguishes TGF-β-Induced Epithelial-Mesenchymal Transition from Its Antiproliferative and Migratory Responses

TGF-β uses a novel mode of receptor activation to phosphorylate SMAD1/5 and induce epithelial-to-mesenchymal transition

Hierarchical patterning modes orchestrate hair follicle morphogenesis

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