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Histone deacetylase inhibitors induce the degradation of the t(8;21) fusion oncoprotein

Oncogene, 2007-01, Vol.26 (1), p.91-101

2007

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Role of the transcription factor AML-1 in acute leukemia and hematopoietic differentiation

Gene, 2000-03, Vol.245 (2), p.223-235

2000

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ETO, a Target of t(8;21) in Acute Leukemia, Makes Distinct Contacts with Multiple Histone Deacetylases and Binds mSin3A through Its Oligomerization Domain

Molecular and Cellular Biology, 2001-10, Vol.21 (19), p.6470-6483

2001

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Identification of AML-1 and the (8;21) translocation protein (AML-1/ETO) as sequence-specific DNA-binding proteins: the runt homology domain is required for DNA binding and protein-protein interactions

Molecular and Cellular Biology, 1993-10, Vol.13 (10), p.6336-6345

1993

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RUNX1 associates with histone deacetylases and SUV39H1 to repress transcription

Oncogene, 2006-09, Vol.25 (42), p.5777-5786

2006

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CCAAT enhancer-binding protein (C/EBP) and AML1 (CBF alpha2) synergistically activate the macrophage colony-stimulating factor receptor promoter

Molecular and Cellular Biology, 1996-03, Vol.16 (3), p.1231-1240

1996

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The t(8;21) fusion protein interferes with AML-1B-dependent transcriptional activation

Molecular and Cellular Biology, 1995-04, Vol.15 (4), p.1974-1982

1995

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TEL contacts multiple co-repressors and specifically associates with histone deacetylase-3

Oncogene, 2001-06, Vol.20 (28), p.3716-3725

2001

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Regions of the retinoblastoma gene product required for its interaction with the E2F transcription factor are necessary for E2 promoter repression and pRb-mediated growth suppression

Molecular and Cellular Biology, 1993-06, Vol.13 (6), p.3384-3391

HIEBERT, S. W

1993

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Direct binding of cyclin D to the retinoblastoma gene product (pRb) and pRb phosphorylation by the cyclin D-dependent kinase CDK4

Genes & development, 1993-03, Vol.7 (3), p.331-342

1993

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Fusion of the TEL Gene on 12p13 to the AML1 Gene on 21q22 in Acute Lymphoblastic Leukemia

Proceedings of the National Academy of Sciences - PNAS, 1995-05, Vol.92 (11), p.4917-4921

1995

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E1A-Dependent Trans-Activation of the Human MYC Promoter is Mediated by the E2F Factor

Proceedings of the National Academy of Sciences - PNAS, 1989-05, Vol.86 (10), p.3594-3598

1989

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The t(12;21) translocation converts AML-1B from an activator to a repressor of transcription

Molecular and Cellular Biology, 1996-04, Vol.16 (4), p.1349-1355

1996

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The interaction of RB with E2F coincides with an inhibition of the transcriptional activity of E2F

Genes & development, 1992-02, Vol.6 (2), p.177-185

1992

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Myeloid translocation genes differentially regulate colorectal cancer programs

Oncogene, 2016-12, Vol.35 (49), p.6341-6349

2016

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TEL, a Putative Tumor Suppressor, Modulates Cell Growth and Cell Morphology of Ras-Transformed Cells While Repressing the Transcription of stromelysin-1

Molecular and Cellular Biology, 2000-08, Vol.20 (16), p.5828-5839

2000

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Role of RUNX family members in transcriptional repression and gene silencing

Oncogene, 2004-05, Vol.23 (24), p.4220-4224

2004

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The assembly competence domain is essential for inv(16)-associated acute myeloid leukemia

Leukemia, 2017-10, Vol.31 (10), p.2267-2271

2017

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An AML-1 Consensus Sequence Binds an Osteoblast-Specific Complex and Transcriptionally Activates the Osteocalcin Gene

Proceedings of the National Academy of Sciences - PNAS, 1996-05, Vol.93 (10), p.4968-4973

1996

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Translocation Products in Acute Myeloid Leukemia Activate the Wnt Signaling Pathway in Hematopoietic Cells

Molecular and Cellular Biology, 2004-04, Vol.24 (7), p.2890-2904

2004

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E2F-1:DP-1 induces p53 and overrides survival factors to trigger apoptosis

Molecular and Cellular Biology, 1995-12, Vol.15 (12), p.6864-6874

1995

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Cell cycle regulation of the E2F transcription factor involves an interaction with cyclin A

Cell, 1991-06, Vol.65 (7), p.1243-1253

1991

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Synergistic Up-Regulation of the Myeloid-Specific Promoter for the Macrophage Colony-Stimulating Factor Receptor by AML1 and the t(8;21) Fusion Protein May Contribute to Leukemogenesis

Proceedings of the National Academy of Sciences - PNAS, 1996-10, Vol.93 (21), p.11895-11900

1996

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E2F-1 cooperates with topoisomerase II inhibition and DNA damage to selectively augment p53-independent apoptosis

Molecular and Cellular Biology, 1997-03, Vol.17 (3), p.1049-1056

1997

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AML1, the Target of Multiple Chromosomal Translocations in Human Leukemia, Is Essential for Normal Fetal Liver Hematopoiesis

Cell, 1996-01, Vol.84 (2), p.321-330

1996

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Histone deacetylase inhibitors induce the degradation of the t(8;21) fusion oncoprotein

Role of the transcription factor AML-1 in acute leukemia and hematopoietic differentiation

ETO, a Target of t(8;21) in Acute Leukemia, Makes Distinct Contacts with Multiple Histone Deacetylases and Binds mSin3A through Its Oligomerization Domain

Identification of AML-1 and the (8;21) translocation protein (AML-1/ETO) as sequence-specific DNA-binding proteins: the runt homology domain is required for DNA binding and protein-protein interactions

RUNX1 associates with histone deacetylases and SUV39H1 to repress transcription

CCAAT enhancer-binding protein (C/EBP) and AML1 (CBF alpha2) synergistically activate the macrophage colony-stimulating factor receptor promoter

The t(8;21) fusion protein interferes with AML-1B-dependent transcriptional activation

TEL contacts multiple co-repressors and specifically associates with histone deacetylase-3

Regions of the retinoblastoma gene product required for its interaction with the E2F transcription factor are necessary for E2 promoter repression and pRb-mediated growth suppression

Direct binding of cyclin D to the retinoblastoma gene product (pRb) and pRb phosphorylation by the cyclin D-dependent kinase CDK4

Fusion of the TEL Gene on 12p13 to the AML1 Gene on 21q22 in Acute Lymphoblastic Leukemia

E1A-Dependent Trans-Activation of the Human MYC Promoter is Mediated by the E2F Factor

The t(12;21) translocation converts AML-1B from an activator to a repressor of transcription

The interaction of RB with E2F coincides with an inhibition of the transcriptional activity of E2F

Myeloid translocation genes differentially regulate colorectal cancer programs

TEL, a Putative Tumor Suppressor, Modulates Cell Growth and Cell Morphology of Ras-Transformed Cells While Repressing the Transcription of stromelysin-1

Role of RUNX family members in transcriptional repression and gene silencing

The assembly competence domain is essential for inv(16)-associated acute myeloid leukemia

An AML-1 Consensus Sequence Binds an Osteoblast-Specific Complex and Transcriptionally Activates the Osteocalcin Gene

Translocation Products in Acute Myeloid Leukemia Activate the Wnt Signaling Pathway in Hematopoietic Cells

E2F-1:DP-1 induces p53 and overrides survival factors to trigger apoptosis

Cell cycle regulation of the E2F transcription factor involves an interaction with cyclin A

Synergistic Up-Regulation of the Myeloid-Specific Promoter for the Macrophage Colony-Stimulating Factor Receptor by AML1 and the t(8;21) Fusion Protein May Contribute to Leukemogenesis

E2F-1 cooperates with topoisomerase II inhibition and DNA damage to selectively augment p53-independent apoptosis

AML1, the Target of Multiple Chromosomal Translocations in Human Leukemia, Is Essential for Normal Fetal Liver Hematopoiesis

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