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Molecular genetics of RecQ helicase disorders

Cellular and molecular life sciences : CMLS, 2007-09, Vol.64 (17), p.2306-2322

2007

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Development and evaluation of human AP endonuclease inhibitors in melanoma and glioma cell lines

British journal of cancer, 2011-02, Vol.104 (4), p.653-663

2011

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BLM has early and late functions in homologous recombination repair in mouse embryonic stem cells

Oncogene, 2010-08, Vol.29 (33), p.4705-4714

2010

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Cellular responses to DNA damage

Annual review of pharmacology and toxicology, 2001-01, Vol.41 (1), p.367-401

2001

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The dissolution of double Holliday junctions

Cold Spring Harbor perspectives in biology, 2014-07, Vol.6 (7), p.a016477-a016477

2014

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Rad54 and Mus81 cooperation promotes DNA damage repair and restrains chromosome missegregation

Oncogene, 2016-09, Vol.35 (37), p.4836-4845

2016

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DNA helicase deficiencies associated with cancer predisposition and premature ageing disorders

Human molecular genetics, 2001-04, Vol.10 (7), p.741-746

2001

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Isolation of a small molecule inhibitor of DNA base excision repair

Nucleic acids research, 2005-01, Vol.33 (15), p.4711-4724

2005

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P12.11 RECQL4 promotes immune evasion and limits response to anti-PD-1 therapy and survival in melanoma patients

Journal for immunotherapy of cancer, 2024-03, Vol.12 (Suppl 1), p.A36-A37

2024

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SGS1, a homologue of the Bloom's and Werner's syndrome genes, is required for maintenance of genome stability in Saccharomyces cerevisiae

Genetics (Austin), 1996-11, Vol.144 (3), p.935-945

1996

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The Bloom's syndrome helicase suppresses crossing over during homologous recombination

Nature, 2003-12, Vol.426 (6968), p.870-874

2003

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RecQ helicases: caretakers of the genome

Nature reviews. Cancer, 2003-03, Vol.3 (3), p.169-178

Hickson, Ian D

2003

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Structure and function of type II DNA topoisomerases

Biochemical journal, 1994-11, Vol.303 ( Pt 3) (3), p.681-695

1994

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Redundancy of DNA helicases in p53-mediated apoptosis

Oncogene, 2006-03, Vol.25 (14), p.2119-2123

2006

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RecQ helicases and topoisomerases: components of a conserved complex for the regulation of genetic recombination

Cellular and molecular life sciences : CMLS, 2001-06, Vol.58 (7), p.894-901

2001

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Identification of residues in the human DNA repair enzyme HAP1 (Ref-1) that are essential for redox regulation of Jun DNA binding

Molecular and Cellular Biology, 1993-09, Vol.13 (9), p.5370-5376

1993

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The distribution and expression of the two isoforms of DNA topoisomerase II in normal and neoplastic human tissues

British journal of cancer, 1997-05, Vol.75 (9), p.1340-1346

1997

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Inhibition of the Bloom's and Werner's Syndrome Helicases by G-Quadruplex Interacting Ligands

Biochemistry (Easton), 2001-12, Vol.40 (50), p.15194-15202

2001

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Reconstitution of the base excision repair pathway for 7,8-dihydro-8-oxoguanine with purified human proteins

Nucleic acids research, 2002-05, Vol.30 (10), p.2124-2130

2002

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The distribution and expression of the Bloom's syndrome gene product in normal and neoplastic human cells

British journal of cancer, 2001-07, Vol.85 (2), p.261-265

2001

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Telomere-binding Protein TRF2 Binds to and Stimulates the Werner and Bloom Syndrome Helicases

The Journal of biological chemistry, 2002-10, Vol.277 (43), p.41110-41119

2002

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The Bloom's Syndrome Helicase Interacts Directly with the Human DNA Mismatch Repair Protein hMSH6

Biological chemistry, 2003-08, Vol.384 (8), p.1155-1164

2003

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Expression and subcellular localization of human AP endonuclease 1 (HAP1/Ref-1) protein: a basis for its role in human disease

Histopathology, 1998-12, Vol.33 (6), p.561-569

1998

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The Bloom's and Werner's syndrome proteins are DNA structure-specific helicases

Nucleic acids research, 2001-07, Vol.29 (13), p.2843-2849

2001

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Expression of aminopeptidase-n (CD 13) in normal tissues and malignant neoplasms of epithelial and lymphoid origin

Journal of clinical pathology, 1994-01, Vol.47 (1), p.43-47

1994

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Molecular genetics of RecQ helicase disorders

Development and evaluation of human AP endonuclease inhibitors in melanoma and glioma cell lines

BLM has early and late functions in homologous recombination repair in mouse embryonic stem cells

Cellular responses to DNA damage

The dissolution of double Holliday junctions

Rad54 and Mus81 cooperation promotes DNA damage repair and restrains chromosome missegregation

DNA helicase deficiencies associated with cancer predisposition and premature ageing disorders

Isolation of a small molecule inhibitor of DNA base excision repair

P12.11 RECQL4 promotes immune evasion and limits response to anti-PD-1 therapy and survival in melanoma patients

SGS1, a homologue of the Bloom's and Werner's syndrome genes, is required for maintenance of genome stability in Saccharomyces cerevisiae

The Bloom's syndrome helicase suppresses crossing over during homologous recombination

RecQ helicases: caretakers of the genome

Structure and function of type II DNA topoisomerases

Redundancy of DNA helicases in p53-mediated apoptosis

RecQ helicases and topoisomerases: components of a conserved complex for the regulation of genetic recombination

Identification of residues in the human DNA repair enzyme HAP1 (Ref-1) that are essential for redox regulation of Jun DNA binding

The distribution and expression of the two isoforms of DNA topoisomerase II in normal and neoplastic human tissues

Inhibition of the Bloom's and Werner's Syndrome Helicases by G-Quadruplex Interacting Ligands

Reconstitution of the base excision repair pathway for 7,8-dihydro-8-oxoguanine with purified human proteins

The distribution and expression of the Bloom's syndrome gene product in normal and neoplastic human cells

Telomere-binding Protein TRF2 Binds to and Stimulates the Werner and Bloom Syndrome Helicases

The Bloom's Syndrome Helicase Interacts Directly with the Human DNA Mismatch Repair Protein hMSH6

Expression and subcellular localization of human AP endonuclease 1 (HAP1/Ref-1) protein: a basis for its role in human disease

The Bloom's and Werner's syndrome proteins are DNA structure-specific helicases

Expression of aminopeptidase-n (CD 13) in normal tissues and malignant neoplasms of epithelial and lymphoid origin

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