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Role of the Immune System in Diabetic Kidney Disease

Current diabetes reports, 2018-04, Vol.18 (4), p.20-10, Article 20

2018

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Myeloid Engraftment in Humanized Mice: Impact of Granulocyte-Colony Stimulating Factor Treatment and Transgenic Mouse Strain

Stem cells and development, 2016-04, Vol.25 (7), p.530

2016

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Adenylate cycalse toxin of Bordetella pertussis inhibits TLR-induced IRF-1 and IRF-8 activation and IL-12 production and enhances IL-10 through MAPK activation in dendritic cells

Journal of leukocyte biology, 2008-07, Vol.84 (1), p.234-243

2008

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Pro-inflammatory Stimulation of Monocytes by ANCA Is Linked to Changes in Cellular Metabolism

Frontiers in medicine, 2020-09, Vol.7, p.553-553

2020

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Diabetic kidney disease and immune modulation

Current opinion in pharmacology, 2013-08, Vol.13 (4), p.602-612

2013

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Urinary Soluble CD163 in Active Renal Vasculitis

Journal of the American Society of Nephrology, 2016-09, Vol.27 (9), p.2906-2916

2016

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Intermediate monocytes in ANCA vasculitis: increased surface expression of ANCA autoantigens and IL-1β secretion in response to anti-MPO antibodies

Scientific reports, 2015-07, Vol.5 (1), p.11888-11888, Article 11888

2015

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IHG-1 increases mitochondrial fusion and bioenergetic function

Diabetes (New York, N.Y.), 2014-12, Vol.63 (12), p.4314-4325

2014

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Alkylating histone deacetylase inhibitors may have therapeutic value in experimental myeloperoxidase-ANCA vasculitis

Kidney international, 2018-11, Vol.94 (5), p.926-936

2018

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Changes in urinary metabolomic profile during relapsing renal vasculitis

Scientific reports, 2016-12, Vol.6 (1), p.38074, Article 38074

2016

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Bcr-Abl regulates osteopontin transcription via Ras, PI-3K, aPKC, Raf-1, and MEK

Journal of leukocyte biology, 2005-07, Vol.78 (1), p.289-300

2005

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IHG-1 Promotes Mitochondrial Biogenesis by Stabilizing PGC-1α

Journal of the American Society of Nephrology, 2011-08, Vol.22 (8), p.1475-1485

2011

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BCR-ABL Regulates Phosphatidylinositol 3-Kinase-p110γ Transcription and Activation and Is Required for Proliferation and Drug Resistance

The Journal of biological chemistry, 2006-02, Vol.281 (5), p.2441-2450

2006

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Insulin receptor substrate 2 and FoxO3a signalling are involved in E‐cadherin expression and transforming growth factor‐β1‐induced repression in kidney epithelial cells

The FEBS journal, 2011-09, Vol.278 (18), p.3370-3380

2011

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IHG-1 must be localised to mitochondria to decrease Smad7 expression and amplify TGF-β1-induced fibrotic responses

Biochimica et biophysica acta, 2013-08, Vol.1833 (8), p.1969-1978

2013

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Deletion of Gremlin1 increases cell proliferation and migration responses in mouse embryonic fibroblasts

Cellular signalling, 2012-04, Vol.24 (4), p.889-898

2012

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Diabetes mellitus and apoptosis: inflammatory cells

Apoptosis (London), 2009-12, Vol.14 (12), p.1435-1450

2009

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Identification of transcriptional targets associated with the expression of p210 Bcr-Abl

European journal of haematology, 2006-05, Vol.76 (5), p.369-383

2006

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Insulin receptor substrate 2 and FoxO3a signalling are involved in E-cadherin expression and transforming growth factor-[beta]1-induced repression in kidney epithelial cells

The FEBS journal, 2011-09, Vol.278 (18), p.3370

2011

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BCR-ABL Regulates Phosphatidylinositol 3-Kinase-p110Î³ Transcription and Activation and Is Required for Proliferation and Drug Resistance

The Journal of biological chemistry, 2006-02, Vol.281 (5), p.2441

2006

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BCR-ABL regulates phosphatidylinositol 3-kinase-p110gamma transcription and activation and is required for proliferation and drug resistance

The Journal of biological chemistry, 2006-02, Vol.281 (5), p.2441-2450

2006

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Intermediate monocytes in ANCA vasculitis: increased surface expression of ANCA autoantigens and IL-1[beta] secretion in response to anti-MPO antibodies

Scientific reports, 2015-07, Vol.5, p.11888

2015

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Role of the Immune System in Diabetic Kidney Disease

Myeloid Engraftment in Humanized Mice: Impact of Granulocyte-Colony Stimulating Factor Treatment and Transgenic Mouse Strain

Adenylate cycalse toxin of Bordetella pertussis inhibits TLR-induced IRF-1 and IRF-8 activation and IL-12 production and enhances IL-10 through MAPK activation in dendritic cells

Pro-inflammatory Stimulation of Monocytes by ANCA Is Linked to Changes in Cellular Metabolism

Diabetic kidney disease and immune modulation

Urinary Soluble CD163 in Active Renal Vasculitis

Intermediate monocytes in ANCA vasculitis: increased surface expression of ANCA autoantigens and IL-1β secretion in response to anti-MPO antibodies

IHG-1 increases mitochondrial fusion and bioenergetic function

Alkylating histone deacetylase inhibitors may have therapeutic value in experimental myeloperoxidase-ANCA vasculitis

Changes in urinary metabolomic profile during relapsing renal vasculitis

Bcr-Abl regulates osteopontin transcription via Ras, PI-3K, aPKC, Raf-1, and MEK

IHG-1 Promotes Mitochondrial Biogenesis by Stabilizing PGC-1α

BCR-ABL Regulates Phosphatidylinositol 3-Kinase-p110γ Transcription and Activation and Is Required for Proliferation and Drug Resistance

Insulin receptor substrate 2 and FoxO3a signalling are involved in E‐cadherin expression and transforming growth factor‐β1‐induced repression in kidney epithelial cells

IHG-1 must be localised to mitochondria to decrease Smad7 expression and amplify TGF-β1-induced fibrotic responses

Deletion of Gremlin1 increases cell proliferation and migration responses in mouse embryonic fibroblasts

Diabetes mellitus and apoptosis: inflammatory cells

Identification of transcriptional targets associated with the expression of p210 Bcr-Abl

Insulin receptor substrate 2 and FoxO3a signalling are involved in E-cadherin expression and transforming growth factor-[beta]1-induced repression in kidney epithelial cells

BCR-ABL Regulates Phosphatidylinositol 3-Kinase-p110Î³ Transcription and Activation and Is Required for Proliferation and Drug Resistance

BCR-ABL regulates phosphatidylinositol 3-kinase-p110gamma transcription and activation and is required for proliferation and drug resistance

Intermediate monocytes in ANCA vasculitis: increased surface expression of ANCA autoantigens and IL-1[beta] secretion in response to anti-MPO antibodies

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