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Chronic granulomatous disease

Current opinion in immunology, 2003-10, Vol.15 (5), p.578-584

2003

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Purification and characterization of Rac 2. A cytosolic GTP-binding protein that regulates human neutrophil NADPH oxidase

The Journal of biological chemistry, 1992-11, Vol.267 (33), p.23575-23582

1992

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Investigation of neutrophil signal transduction using a specific inhibitor of phosphatidylinositol 3-kinase

The Journal of immunology (1950), 1995-03, Vol.154 (5), p.2413

1995

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Neutrophil nicotinamide adenine dinucleotide phosphate oxidase assembly : translocation of p47-phox and p67-phox requires interaction between p47-phox cytochrome b558

The Journal of clinical investigation, 1991, Vol.87 (1), p.352-356

1991

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Regulation of NADPH oxidase activity by Rac GTPase activating protein(s)

Molecular biology of the cell, 1993-11, Vol.4 (11), p.1217-1223

1993

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Autosomal recessive chronic granulomatous disease caused by novel mutations in NCF-2, the gene encoding the p67-phox component of phagocyte NADPH oxidase

Human genetics, 1999-11, Vol.105 (5), p.460-467

1999

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Regulation of Phagocyte Oxygen Radical Production by the GTP-Binding Protein Rac 2

Science (American Association for the Advancement of Science), 1991-12, Vol.254 (5037), p.1512-1515

1991

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1,2-dioctanoyl-sn-glycerol can stimulate neutrophils by different mechanisms. Evidence for a pathway that does not involve phosphorylation of the 47-kDa protein

The Journal of biological chemistry, 1989-12, Vol.264 (34), p.20676-20682

1989

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Molecular analysis of 9 new families with chronic granulomatous disease caused by mutations in CYBA, the gene encoding p22(phox)

Blood, 2000-08, Vol.96 (3), p.1106-1112

2000

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Absolute Requirement for GTP in Activation of Human Neutrophil NADPH Oxidase in a Cell-Free System: Role of ATP in Regenerating GTP

Proceedings of the National Academy of Sciences - PNAS, 1992-03, Vol.89 (6), p.2494-2498

1992

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Autosomal recessive chronic granulomatous disease caused by defects in NCF-1, the gene encoding the phagocyte p47-phox: mutations not arising in the NCF-1 pseudogenes

Blood, 2001, Vol.97 (1), p.305-311

2001

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Okadaic acid produces changes in phosphorylation and translocation of proteins and in intracellular calcium in human neutrophils : relationship with the activation of the NADPH oxidase by different stimuli

Biochemical journal, 1992-09, Vol.286 (3), p.687-692

1992

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Stimulated neutrophils from patients with autosomal recessive chronic granulomatous disease fail to phosphorylate a Mr-44,000 protein

Nature (London), 1985-08, Vol.316 (6028), p.547-549

1985

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Neutrophil nicotinamide adenine dinucleotide phosphate oxidase assembly. Translocation of p47-phox and p67-phox requires interaction between p47-phox and cytochrome b558

The Journal of clinical investigation, 1991-01, Vol.87 (1), p.352-356

1991

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Protein Phosphorylation Associated with Synergistic Stimulation of Neutrophils

The Journal of biological chemistry, 1989-09, Vol.264 (25), p.14935-14939

1989

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Hematologically Important Mutations: X-Linked Chronic Granulomatous Disease (Second Update)

Blood cells, molecules, & diseases, 2001, Vol.27 (1), p.16-26

2001

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Further evidence for the involvement of a phosphoprotein in the respiratory burst oxidase of human neutrophils

Biochemical journal, 1986-11, Vol.239 (3), p.723-731

1986

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Continuous phosphorylation of both the 47 and the 49 kDa proteins occurs during superoxide production by neutrophils

Biochimica et biophysica acta, 1990-05, Vol.1052 (2), p.299-305

1990

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Localization of the 47 kDa phosphoprotein involved in the respiratory-burst NADPH oxidase of phagocytic cells

Biochemical journal, 1989-05, Vol.260 (1), p.243-248

1989

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Hematologically Important Mutations: The Autosomal Recessive Forms of Chronic Granulomatous Disease (First Update)

Blood cells, molecules, & diseases, 2000-10, Vol.26 (5), p.561-565

2000

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Activation of human neutrophil NADPH oxidase by phosphatidic acid or diacylglycerol in a cell-free system. Activity of diacylglycerol is dependent on its conversion to phosphatidic acid

The Journal of biological chemistry, 1999-08, Vol.274 (32), p.22243

1999

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Paradoxical Effects of Retinal in Neutrophil Stimulation

The Journal of biological chemistry, 1989-09, Vol.264 (25), p.14947-14953

1989

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Protein Phosphorylation in Neutrophils From Patients With p67-phox-Deficient Chronic Granulomatous Disease

Blood, 1996-05, Vol.87 (10), p.4404-4410

1996

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Naphthalenesulphonamides block neutrophil superoxide production by intact cells and in a cell-free system: is myosin light chain kinase responsible for these effects?

Biochemical journal, 1995-10, Vol.311 ( Pt 1) (1), p.81-87

1995

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Cofilin undergoes rapid dephosphorylation in stimulated neutrophils and translocates to ruffled membranes enriched in products of the NADPH oxidase complex. Evidence for a novel cycle of phosphorylation and dephosphorylation

Histochemistry and cell biology, 1997-09, Vol.108 (3), p.221-233

1997

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Chronic granulomatous disease

Purification and characterization of Rac 2. A cytosolic GTP-binding protein that regulates human neutrophil NADPH oxidase

Investigation of neutrophil signal transduction using a specific inhibitor of phosphatidylinositol 3-kinase

Neutrophil nicotinamide adenine dinucleotide phosphate oxidase assembly : translocation of p47-phox and p67-phox requires interaction between p47-phox cytochrome b558

Regulation of NADPH oxidase activity by Rac GTPase activating protein(s)

Autosomal recessive chronic granulomatous disease caused by novel mutations in NCF-2, the gene encoding the p67-phox component of phagocyte NADPH oxidase

Regulation of Phagocyte Oxygen Radical Production by the GTP-Binding Protein Rac 2

1,2-dioctanoyl-sn-glycerol can stimulate neutrophils by different mechanisms. Evidence for a pathway that does not involve phosphorylation of the 47-kDa protein

Molecular analysis of 9 new families with chronic granulomatous disease caused by mutations in CYBA, the gene encoding p22(phox)

Absolute Requirement for GTP in Activation of Human Neutrophil NADPH Oxidase in a Cell-Free System: Role of ATP in Regenerating GTP

Autosomal recessive chronic granulomatous disease caused by defects in NCF-1, the gene encoding the phagocyte p47-phox: mutations not arising in the NCF-1 pseudogenes

Okadaic acid produces changes in phosphorylation and translocation of proteins and in intracellular calcium in human neutrophils : relationship with the activation of the NADPH oxidase by different stimuli

Stimulated neutrophils from patients with autosomal recessive chronic granulomatous disease fail to phosphorylate a Mr-44,000 protein

Neutrophil nicotinamide adenine dinucleotide phosphate oxidase assembly. Translocation of p47-phox and p67-phox requires interaction between p47-phox and cytochrome b558

Protein Phosphorylation Associated with Synergistic Stimulation of Neutrophils

Hematologically Important Mutations: X-Linked Chronic Granulomatous Disease (Second Update)

Further evidence for the involvement of a phosphoprotein in the respiratory burst oxidase of human neutrophils

Continuous phosphorylation of both the 47 and the 49 kDa proteins occurs during superoxide production by neutrophils

Localization of the 47 kDa phosphoprotein involved in the respiratory-burst NADPH oxidase of phagocytic cells

Hematologically Important Mutations: The Autosomal Recessive Forms of Chronic Granulomatous Disease (First Update)

Activation of human neutrophil NADPH oxidase by phosphatidic acid or diacylglycerol in a cell-free system. Activity of diacylglycerol is dependent on its conversion to phosphatidic acid

Paradoxical Effects of Retinal in Neutrophil Stimulation

Protein Phosphorylation in Neutrophils From Patients With p67-phox-Deficient Chronic Granulomatous Disease

Naphthalenesulphonamides block neutrophil superoxide production by intact cells and in a cell-free system: is myosin light chain kinase responsible for these effects?

Cofilin undergoes rapid dephosphorylation in stimulated neutrophils and translocates to ruffled membranes enriched in products of the NADPH oxidase complex. Evidence for a novel cycle of phosphorylation and dephosphorylation

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