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Mechanical regulation of cardiac fibroblast profibrotic phenotypes

Molecular biology of the cell, 2017-07, Vol.28 (14), p.1871-1882

2017

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Sweet, yet underappreciated: Proteoglycans and extracellular matrix remodeling in heart disease

Matrix biology, 2019-01, Vol.75-76, p.286-299

2019

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Maintaining resting cardiac fibroblasts in vitro by disrupting mechanotransduction

PloS one, 2020-10, Vol.15 (10), p.e0241390

2020

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Syndecan-4 is a key determinant of collagen cross-linking and passive myocardial stiffness in the pressure-overloaded heart

Cardiovascular research, 2015-05, Vol.106 (2), p.217-226

2015

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Syndecan-4 Protects the Heart From the Profibrotic Effects of Thrombin-Cleaved Osteopontin

Journal of the American Heart Association, 2020-02, Vol.9 (3), p.e013518-e013518

2020

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Cancer-associated fibroblasts: how do they contribute to metastasis?

Clinical & experimental metastasis, 2019-04, Vol.36 (2), p.71-86

2019

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The Heparan Sulfate Proteoglycan Glypican-6 Is Upregulated in the Failing Heart, and Regulates Cardiomyocyte Growth through ERK1/2 Signaling

PloS one, 2016-10, Vol.11 (10), p.e0165079-e0165079

2016

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Fibrotic Signaling in Cardiac Fibroblasts and Vascular Smooth Muscle Cells: The Dual Roles of Fibrosis in HFpEF and CAD

Cells (Basel, Switzerland), 2022-05, Vol.11 (10), p.1657

2022

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The Soft- and Hard-Heartedness of Cardiac Fibroblasts: Mechanotransduction Signaling Pathways in Fibrosis of the Heart

Journal of clinical medicine, 2017-05, Vol.6 (5), p.53

2017

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Syndecans in heart fibrosis

Cell and Tissue Research, 2016-09, Vol.365 (3), p.539-552

2016

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Syndecan-4 signaling via NFAT regulates extracellular matrix production and cardiac myofibroblast differentiation in response to mechanical stress

Journal of molecular and cellular cardiology, 2013-01, Vol.54, p.73-81

2013

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Integrin α11β1 and syndecan-4 dual receptor ablation attenuate cardiac hypertrophy in the pressure overloaded heart

American journal of physiology. Heart and circulatory physiology, 2022-06, Vol.322 (6), p.H1057-H1071

2022

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Array atomic force microscopy for real-time multiparametric analysis

Proceedings of the National Academy of Sciences - PNAS, 2019-03, Vol.116 (13), p.5872-5877

2019

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Lack of collagen VIII reduces fibrosis and promotes early mortality and cardiac dilatation in pressure overload in mice

Cardiovascular research, 2015-04, Vol.106 (1), p.32-42

2015

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Innate immune signaling induces expression and shedding of the heparan sulfate proteoglycan syndecan‐4 in cardiac fibroblasts and myocytes, affecting inflammation in the pressure‐overloaded heart

The FEBS journal, 2013-05, Vol.280 (10), p.2228-2247

2013

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Combining Stiffness and Stretch to Study Cardiac Fibroblast Pro‐Fibrotic Activity

The FASEB journal, 2018-04, Vol.32 (S1), p.896.2-896.2

2018

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Abstract 417: Syndecan-4 Regulates the Effect of Osteopontin on Cardiac Fibroblast Function and Phenotype

Circulation research, 2017-07, Vol.121 (suppl_1)

2017

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Integrin α11β1 and syndecan-4 dual receptor ablation attenuates cardiac hypertrophy in the pressure overloaded heart

American journal of physiology. Heart and circulatory physiology, 2022-05

2022

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Downregulation of L-type Ca2+ channel in rat mesenteric arteries leads to loss of smooth muscle contractile phenotype and inward hypertrophic remodeling

American journal of physiology. Heart and circulatory physiology, 2014-05, Vol.306 (9), p.H1287-H1301

2014

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Downregulation of L-type Ca^sup 2+^ channel in rat mesenteric arteries leads to loss of smooth muscle contractile phenotype and inward hypertrophic remodeling

American journal of physiology. Heart and circulatory physiology, 2014-05, Vol.306 (9), p.H1287

2014

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Downregulation of L-type Ca 2+ channel in rat mesenteric arteries leads to loss of smooth muscle contractile phenotype and inward hypertrophic remodeling

American journal of physiology. Heart and circulatory physiology, 2014-05, Vol.306 (9), p.H1287-H1301

2014

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Mechanical regulation of cardiac fibroblast profibrotic phenotypes

Sweet, yet underappreciated: Proteoglycans and extracellular matrix remodeling in heart disease

Maintaining resting cardiac fibroblasts in vitro by disrupting mechanotransduction

Syndecan-4 is a key determinant of collagen cross-linking and passive myocardial stiffness in the pressure-overloaded heart

Syndecan-4 Protects the Heart From the Profibrotic Effects of Thrombin-Cleaved Osteopontin

Cancer-associated fibroblasts: how do they contribute to metastasis?

The Heparan Sulfate Proteoglycan Glypican-6 Is Upregulated in the Failing Heart, and Regulates Cardiomyocyte Growth through ERK1/2 Signaling

Fibrotic Signaling in Cardiac Fibroblasts and Vascular Smooth Muscle Cells: The Dual Roles of Fibrosis in HFpEF and CAD

The Soft- and Hard-Heartedness of Cardiac Fibroblasts: Mechanotransduction Signaling Pathways in Fibrosis of the Heart

Syndecans in heart fibrosis

Syndecan-4 signaling via NFAT regulates extracellular matrix production and cardiac myofibroblast differentiation in response to mechanical stress

Integrin α11β1 and syndecan-4 dual receptor ablation attenuate cardiac hypertrophy in the pressure overloaded heart

Array atomic force microscopy for real-time multiparametric analysis

Lack of collagen VIII reduces fibrosis and promotes early mortality and cardiac dilatation in pressure overload in mice

Innate immune signaling induces expression and shedding of the heparan sulfate proteoglycan syndecan‐4 in cardiac fibroblasts and myocytes, affecting inflammation in the pressure‐overloaded heart

Combining Stiffness and Stretch to Study Cardiac Fibroblast Pro‐Fibrotic Activity

Abstract 417: Syndecan-4 Regulates the Effect of Osteopontin on Cardiac Fibroblast Function and Phenotype

Integrin α11β1 and syndecan-4 dual receptor ablation attenuates cardiac hypertrophy in the pressure overloaded heart

Downregulation of L-type Ca2+ channel in rat mesenteric arteries leads to loss of smooth muscle contractile phenotype and inward hypertrophic remodeling

Downregulation of L-type Ca^sup 2+^ channel in rat mesenteric arteries leads to loss of smooth muscle contractile phenotype and inward hypertrophic remodeling

Downregulation of L-type Ca 2+ channel in rat mesenteric arteries leads to loss of smooth muscle contractile phenotype and inward hypertrophic remodeling

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