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AP-1 imprints a reversible transcriptional programme of senescent cells

Nature cell biology, 2020-07, Vol.22 (7), p.842-855

2020

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The replicometer is broken: telomeres activate cellular senescence in response to genotoxic stresses

Aging cell, 2014-10, Vol.13 (5), p.780-786

2014

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Cellular Senescence in Aging Primates

Science (American Association for the Advancement of Science), 2006-03, Vol.311 (5765), p.1257-1257

2006

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Telomeres and replicative cellular aging of the human placenta and chorioamniotic membranes

Scientific reports, 2021-03, Vol.11 (1), p.5115-5115, Article 5115

2021

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Accumulation of senescent cells in mitotic tissue of aging primates

Mechanisms of ageing and development, 2007-01, Vol.128 (1), p.36-44

2007

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Tracking and fixed ranking of leukocyte telomere length across the adult life course

Aging cell, 2013-08, Vol.12 (4), p.615-621

2013

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Shorter telomere length in Europeans than in Africans due to polygenetic adaptation

Human molecular genetics, 2016-06, Vol.25 (11), p.2324-2330

2016

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BRCA2 acts as a RAD51 loader to facilitate telomere replication and capping

Nature structural & molecular biology, 2010-12, Vol.17 (12), p.1461-1469

2010

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Genome-wide association identifies OBFC1 as a locus involved in human leukocyte telomere biology

Proceedings of the National Academy of Sciences - PNAS, 2010-05, Vol.107 (20), p.9293-9298

2010

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Author Correction: AP-1 imprints a reversible transcriptional programme of senescent cells

Nature cell biology, 2020-10, Vol.22 (10), p.1286-1288

2020

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Stn1 is critical for telomere maintenance and long‐term viability of somatic human cells

Aging cell, 2015-06, Vol.14 (3), p.372-381

2015

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Telomere dysfunction promotes transdifferentiation of human fibroblasts into myofibroblasts

Aging cell, 2018-12, Vol.17 (6), p.e12838-n/a

2018

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Senescence is an endogenous trigger for microRNA-directed transcriptional gene silencing in human cells

Nature cell biology, 2012-03, Vol.14 (3), p.266-275

2012

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Expression of (NES-)hTERT in cancer cells delays cell cycle progression and increases sensitivity to genotoxic stress

PloS one, 2010-05, Vol.5 (5), p.e10812-e10812

2010

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Cellular senescence, p21, and the path to fibrosis

The EMBO journal, 2024-09

2024

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Derepression of hTERT gene expression promotes escape from oncogene-induced cellular senescence

Proceedings of the National Academy of Sciences - PNAS, 2016-08, Vol.113 (34), p.E5024-E5033

2016

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Expression of NES-hTERT in Cancer Cells Delays Cell Cycle Progression and Increases Sensitivity to Genotoxic Stress

PloS one, 2010-05, Vol.5 (5), p.e10812

2010

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Telomerase gene therapy: a novel approach to combat aging

EMBO molecular medicine, 2012-08, Vol.4 (8), p.685-687

2012

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Senescence‐associated β‐galactosidase reveals the abundance of senescent CD8+ T cells in aging humans

Aging cell, 2021-05, Vol.20 (5), p.e13344-n/a

2021

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Telomeric DNA damage is irreparable and causes persistent DNA-damage-response activation

Nature cell biology, 2012-04, Vol.14 (4), p.355-365

2012

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Irreparable telomeric DNA damage and persistent DDR signalling as a shared causative mechanism of cellular senescence and ageing

Current opinion in genetics & development, 2014-06, Vol.26, p.89-95

2014

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New intranasal and injectable gene therapy for healthy life extension

Proceedings of the National Academy of Sciences - PNAS, 2022-05, Vol.119 (20), p.e2121499119

2022

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Senescence-associated β-galactosidase activity and other markers of senescence are present in human peripheral blood CD8+ cells during healthy aging

The Journal of immunology (1950), 2021-05, Vol.206 (1_Supplement), p.98-98.31

2021

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Oncogene-induced telomere dysfunction enforces cellular senescence in human cancer precursor lesions

The EMBO journal, 2012-06, Vol.31 (13), p.2839-2851

2012

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Senescence-associated β-galactosidase activity and other markers of senescence are present in human peripheral blood mononuclear cells during healthy aging

The Journal of immunology (1950), 2020-05, Vol.204 (1_Supplement), p.154-154.15

2020

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AP-1 imprints a reversible transcriptional programme of senescent cells

The replicometer is broken: telomeres activate cellular senescence in response to genotoxic stresses

Cellular Senescence in Aging Primates

Telomeres and replicative cellular aging of the human placenta and chorioamniotic membranes

Accumulation of senescent cells in mitotic tissue of aging primates

Tracking and fixed ranking of leukocyte telomere length across the adult life course

Shorter telomere length in Europeans than in Africans due to polygenetic adaptation

BRCA2 acts as a RAD51 loader to facilitate telomere replication and capping

Genome-wide association identifies OBFC1 as a locus involved in human leukocyte telomere biology

Author Correction: AP-1 imprints a reversible transcriptional programme of senescent cells

Stn1 is critical for telomere maintenance and long‐term viability of somatic human cells

Telomere dysfunction promotes transdifferentiation of human fibroblasts into myofibroblasts

Senescence is an endogenous trigger for microRNA-directed transcriptional gene silencing in human cells

Expression of (NES-)hTERT in cancer cells delays cell cycle progression and increases sensitivity to genotoxic stress

Cellular senescence, p21, and the path to fibrosis

Derepression of hTERT gene expression promotes escape from oncogene-induced cellular senescence

Expression of NES-hTERT in Cancer Cells Delays Cell Cycle Progression and Increases Sensitivity to Genotoxic Stress

Telomerase gene therapy: a novel approach to combat aging

Senescence‐associated β‐galactosidase reveals the abundance of senescent CD8+ T cells in aging humans

Telomeric DNA damage is irreparable and causes persistent DNA-damage-response activation

Irreparable telomeric DNA damage and persistent DDR signalling as a shared causative mechanism of cellular senescence and ageing

New intranasal and injectable gene therapy for healthy life extension

Senescence-associated β-galactosidase activity and other markers of senescence are present in human peripheral blood CD8+ cells during healthy aging

Oncogene-induced telomere dysfunction enforces cellular senescence in human cancer precursor lesions

Senescence-associated β-galactosidase activity and other markers of senescence are present in human peripheral blood mononuclear cells during healthy aging

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