UB Paderborn / Katalog / Suche / in CDI

1 – 25 von 93

Sortiert nach

Alzheimer's disease tau is a prominent pathology in LRRK2 Parkinson's disease

Acta neuropathologica communications, 2019-11, Vol.7 (1), p.183-16, Article 183

2019

Volltextzugriff (PDF)

Network models to enhance the translational impact of cross-species studies

Nature reviews. Neuroscience, 2023-09, Vol.24 (9), p.575

2023

Volltextzugriff (PDF)

Spatial transcriptomics reveals molecular dysfunction associated with cortical Lewy pathology

Nature communications, 2024-03, Vol.15 (1), p.2642-2642, Article 2642

2024

Volltextzugriff (PDF)

The roles of connectivity and neuronal phenotype in determining the pattern of α-synuclein pathology in Parkinson's disease

Neurobiology of disease, 2022-06, Vol.168, p.105687-105687, Article 105687

2022

Volltextzugriff (PDF)

Characterization of novel conformation-selective α-synuclein antibodies as potential immunotherapeutic agents for Parkinson's disease

Neurobiology of disease, 2020-03, Vol.136, p.104712-104712, Article 104712

2020

Volltextzugriff (PDF)

Correction to: Alzheimer’s disease tau is a prominent pathology in LRRK2 Parkinson’s disease

Acta neuropathologica communications, 2024-06, Vol.12 (1), p.110-4, Article 110

2024

Volltextzugriff (PDF)

Unbiased Proteomics of Early Lewy Body Formation Model Implicates Active Microtubule Affinity-Regulating Kinases (MARKs) in Synucleinopathies

The Journal of neuroscience, 2017-06, Vol.37 (24), p.5870-5884

2017

Volltextzugriff (PDF)

Synaptic location is a determinant of the detrimental effects of α-synuclein pathology to glutamatergic transmission in the basolateral amygdala

eLife, 2022-07, Vol.11

2022

Volltextzugriff (PDF)

α-Synuclein pathology in Parkinson’s disease and related α-synucleinopathies

Neuroscience letters, 2019-09, Vol.709, p.134316-134316, Article 134316

2019

Volltextzugriff (PDF)

LRRK2 activity does not dramatically alter α-synuclein pathology in primary neurons

Acta neuropathologica communications, 2018-05, Vol.6 (1), p.45-45, Article 45

2018

Volltextzugriff (PDF)

LRRK2 inhibition does not impart protection from α-synuclein pathology and neuron death in non-transgenic mice

Acta neuropathologica communications, 2019-02, Vol.7 (1), p.28-28, Article 28

2019

Volltextzugriff (PDF)

A novel automated morphological analysis of Iba1+ microglia using a deep learning assisted model

Frontiers in cellular neuroscience, 2022-09, Vol.16, p.944875-944875

2022

Volltextzugriff (PDF)

A topographical atlas of α-synuclein dosage and cell type-specific expression in adult mouse brain and peripheral organs

NPJ Parkinson's Disease, 2024-03, Vol.10 (1), p.65-65, Article 65

2024

Volltextzugriff (PDF)

Spread of α-synuclein pathology through the brain connectome is modulated by selective vulnerability and predicted by network analysis

Nature neuroscience, 2019-08, Vol.22 (8), p.1248

2019

Volltextzugriff (PDF)

LRRK2 kinase inhibition reverses G2019S mutation-dependent effects on tau pathology progression

Translational neurodegeneration, 2024-03, Vol.13 (1), p.13-13, Article 13

2024

Volltextzugriff (PDF)

Glucocerebrosidase activity and lipid levels are related to protein pathologies in Parkinson's disease

NPJ Parkinson's Disease, 2023-05, Vol.9 (1), p.74-14, Article 74

2023

Volltextzugriff (PDF)

Glucocerebrosidase Activity Modulates Neuronal Susceptibility to Pathological α-Synuclein Insult

Neuron (Cambridge, Mass.), 2020-03, Vol.105 (5), p.822-836.e7

2020

Volltextzugriff (PDF)

Glucose sensitivity of mouse olfactory bulb neurons is conveyed by a voltage‐gated potassium channel

The Journal of physiology, 2013-05, Vol.591 (10), p.2541-2561

2013

Volltextzugriff (PDF)

Urinary tract infections trigger synucleinopathy via the innate immune response

Acta neuropathologica, 2023-05, Vol.145 (5), p.541-559

2023

Volltextzugriff (PDF)

Neuronal ceroid lipofuscinosis with DNAJC5/CSPα mutation has PPT1 pathology and exhibit aberrant protein palmitoylation

Acta neuropathologica, 2016-04, Vol.131 (4), p.621-637

2016

Volltextzugriff (PDF)

LRRK2 inhibition does not impart protection from [alpha]-synuclein pathology and neuron death in non-transgenic mice

Acta neuropathologica communications, 2019-02, Vol.7 (1)

2019

Volltextzugriff (PDF)

Computational modeling of tau pathology spread reveals patterns of regional vulnerability and the impact of a genetic risk factor

Science advances, 2021-06, Vol.7 (24)

2021

Volltextzugriff (PDF)

LRRK2 Kinase Activity Does Not Alter Cell-Autonomous Tau Pathology Development in Primary Neurons

Journal of Parkinson's disease, 2021-01, Vol.11 (3), p.1187

2021

Volltextzugriff (PDF)

Synaptic location is a determinant of the detrimental effects of [alpha]-synuclein pathology to glutamatergic transmission in the basolateral amygdala

eLife, 2022-07, Vol.11

2022

Volltextzugriff (PDF)

LRRK2 activity does not dramatically alter [alpha]-synuclein pathology in primary neurons

Acta neuropathologica communications, 2018-05, Vol.6 (1)

2018

Volltextzugriff (PDF)

1 – 25 von 93

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(86)
Mit Volltextzugriff
(82)

Publikationsform

Artikel
(93)

Sprache

Englisch
(93)
Japanisch
(12)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 90 Tagen
(2)

Menü

Weitere Dienste

Einstellungen

Alzheimer's disease tau is a prominent pathology in LRRK2 Parkinson's disease

Network models to enhance the translational impact of cross-species studies

Spatial transcriptomics reveals molecular dysfunction associated with cortical Lewy pathology

The roles of connectivity and neuronal phenotype in determining the pattern of α-synuclein pathology in Parkinson's disease

Characterization of novel conformation-selective α-synuclein antibodies as potential immunotherapeutic agents for Parkinson's disease

Correction to: Alzheimer’s disease tau is a prominent pathology in LRRK2 Parkinson’s disease

Unbiased Proteomics of Early Lewy Body Formation Model Implicates Active Microtubule Affinity-Regulating Kinases (MARKs) in Synucleinopathies

Synaptic location is a determinant of the detrimental effects of α-synuclein pathology to glutamatergic transmission in the basolateral amygdala

α-Synuclein pathology in Parkinson’s disease and related α-synucleinopathies

LRRK2 activity does not dramatically alter α-synuclein pathology in primary neurons

LRRK2 inhibition does not impart protection from α-synuclein pathology and neuron death in non-transgenic mice

A novel automated morphological analysis of Iba1+ microglia using a deep learning assisted model

A topographical atlas of α-synuclein dosage and cell type-specific expression in adult mouse brain and peripheral organs

Spread of α-synuclein pathology through the brain connectome is modulated by selective vulnerability and predicted by network analysis

LRRK2 kinase inhibition reverses G2019S mutation-dependent effects on tau pathology progression

Glucocerebrosidase activity and lipid levels are related to protein pathologies in Parkinson's disease

Glucocerebrosidase Activity Modulates Neuronal Susceptibility to Pathological α-Synuclein Insult

Glucose sensitivity of mouse olfactory bulb neurons is conveyed by a voltage‐gated potassium channel

Urinary tract infections trigger synucleinopathy via the innate immune response

Neuronal ceroid lipofuscinosis with DNAJC5/CSPα mutation has PPT1 pathology and exhibit aberrant protein palmitoylation

LRRK2 inhibition does not impart protection from [alpha]-synuclein pathology and neuron death in non-transgenic mice

Computational modeling of tau pathology spread reveals patterns of regional vulnerability and the impact of a genetic risk factor

LRRK2 Kinase Activity Does Not Alter Cell-Autonomous Tau Pathology Development in Primary Neurons

Synaptic location is a determinant of the detrimental effects of [alpha]-synuclein pathology to glutamatergic transmission in the basolateral amygdala

LRRK2 activity does not dramatically alter [alpha]-synuclein pathology in primary neurons

Suchergebnisse filtern