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Inhibition of mutagenic translesion synthesis: A possible strategy for improving chemotherapy?

PLoS genetics, 2017-08, Vol.13 (8), p.e1006842

2017

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A senescence secretory switch mediated by PI3K/AKT/mTOR activation controls chemoprotective endothelial secretory responses

Genes & development, 2016-08, Vol.30 (16), p.1811-1821

2016

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A subset of platinum-containing chemotherapeutic agents kills cells by inducing ribosome biogenesis stress

Nature medicine, 2017-04, Vol.23 (4), p.461-471

2017

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The primary mechanism of cytotoxicity of the chemotherapeutic agent CX-5461 is topoisomerase II poisoning

Proceedings of the National Academy of Sciences - PNAS, 2020-02, Vol.117 (8), p.4053-4060

2020

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Microenvironmental IL-6 inhibits anti-cancer immune responses generated by cytotoxic chemotherapy

Nature communications, 2021-10, Vol.12 (1), p.6218-13, Article 6218

2021

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Coordinated Splicing of Regulatory Detained Introns within Oncogenic Transcripts Creates an Exploitable Vulnerability in Malignant Glioma

Cancer cell, 2017-10, Vol.32 (4), p.411-426.e11

2017

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Intratumor heterogeneity alters most effective drugs in designed combinations

Proceedings of the National Academy of Sciences - PNAS, 2014-07, Vol.111 (29), p.10773-10778

2014

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Sensitizing Protective Tumor Microenvironments to Antibody-Mediated Therapy

Cell, 2014-01, Vol.156 (3), p.590-602

2014

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Rev7 loss alters cisplatin response and increases drug efficacy in chemotherapy-resistant lung cancer

Proceedings of the National Academy of Sciences - PNAS, 2020-11, Vol.117 (46), p.28922-28924

2020

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Exploiting Temporal Collateral Sensitivity in Tumor Clonal Evolution

Cell, 2016-03, Vol.165 (1), p.234-246

2016

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Error-prone translesion synthesis mediates acquired chemoresistance

Proceedings of the National Academy of Sciences - PNAS, 2010-11, Vol.107 (48), p.20792-20797

2010

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Versatile in vivo regulation of tumor phenotypes by dCas9-mediated transcriptional perturbation

Proceedings of the National Academy of Sciences - PNAS, 2016-07, Vol.113 (27), p.E3892-E3900

2016

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Defining principles of combination drug mechanisms of action

Proceedings of the National Academy of Sciences - PNAS, 2013-01, Vol.110 (2), p.E170-E179

2013

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Stage-specific sensitivity to p53 restoration during lung cancer progression

Nature (London), 2010-11, Vol.468 (7323), p.572-575

2010

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Nanoparticle conjugates of a highly potent toxin enhance safety and circumvent platinum resistance in ovarian cancer

Nature communications, 2017-12, Vol.8 (1), p.2166-2166, Article 2166

2017

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Integrated regulatory models for inference of subtype‐specific susceptibilities in glioblastoma

Molecular systems biology, 2020-09, Vol.16 (9), p.e9506-n/a

2020

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The creatine kinase pathway is a metabolic vulnerability in EVI1-positive acute myeloid leukemia

Nature medicine, 2017-03, Vol.23 (3), p.301-313

2017

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REV1 inhibitor JH-RE-06 enhances tumor cell response to chemotherapy by triggering senescence hallmarks

Proceedings of the National Academy of Sciences - PNAS, 2020-11, Vol.117 (46), p.28918-28921

2020

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A Pleiotropic RNA-Binding Protein Controls Distinct Cell Cycle Checkpoints to Drive Resistance of p53-Defective Tumors to Chemotherapy

Cancer cell, 2015-11, Vol.28 (5), p.623-637

2015

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Simultaneous screening of overexpressed genes in breast cancer for oncogenic drivers and tumor dependencies

Scientific reports, 2024-06, Vol.14 (1), p.13227-13

2024

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SYK Is a Critical Regulator of FLT3 in Acute Myeloid Leukemia

Cancer cell, 2014-02, Vol.25 (2), p.226-242

2014

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Collateral responses to classical cytotoxic chemotherapies are heterogeneous and sensitivities are sparse

Scientific reports, 2022-03, Vol.12 (1), p.5453-5453, Article 5453

2022

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Topoisomerase levels determine chemotherapy response in vitro and in vivo

Proceedings of the National Academy of Sciences - PNAS, 2008-07, Vol.105 (26), p.9053-9058

2008

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Addressing genetic tumor heterogeneity through computationally predictive combination therapy

Cancer discovery, 2014-02, Vol.4 (2), p.166-174

2014

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Inhibition of mutagenic translesion synthesis: A possible strategy for improving chemotherapy?

A senescence secretory switch mediated by PI3K/AKT/mTOR activation controls chemoprotective endothelial secretory responses

A subset of platinum-containing chemotherapeutic agents kills cells by inducing ribosome biogenesis stress

The primary mechanism of cytotoxicity of the chemotherapeutic agent CX-5461 is topoisomerase II poisoning

Microenvironmental IL-6 inhibits anti-cancer immune responses generated by cytotoxic chemotherapy

Coordinated Splicing of Regulatory Detained Introns within Oncogenic Transcripts Creates an Exploitable Vulnerability in Malignant Glioma

Intratumor heterogeneity alters most effective drugs in designed combinations

Sensitizing Protective Tumor Microenvironments to Antibody-Mediated Therapy

Rev7 loss alters cisplatin response and increases drug efficacy in chemotherapy-resistant lung cancer

Exploiting Temporal Collateral Sensitivity in Tumor Clonal Evolution

Error-prone translesion synthesis mediates acquired chemoresistance

Versatile in vivo regulation of tumor phenotypes by dCas9-mediated transcriptional perturbation

Defining principles of combination drug mechanisms of action

Stage-specific sensitivity to p53 restoration during lung cancer progression

Nanoparticle conjugates of a highly potent toxin enhance safety and circumvent platinum resistance in ovarian cancer

Integrated regulatory models for inference of subtype‐specific susceptibilities in glioblastoma

The creatine kinase pathway is a metabolic vulnerability in EVI1-positive acute myeloid leukemia

REV1 inhibitor JH-RE-06 enhances tumor cell response to chemotherapy by triggering senescence hallmarks

A Pleiotropic RNA-Binding Protein Controls Distinct Cell Cycle Checkpoints to Drive Resistance of p53-Defective Tumors to Chemotherapy

Simultaneous screening of overexpressed genes in breast cancer for oncogenic drivers and tumor dependencies

SYK Is a Critical Regulator of FLT3 in Acute Myeloid Leukemia

Collateral responses to classical cytotoxic chemotherapies are heterogeneous and sensitivities are sparse

Topoisomerase levels determine chemotherapy response in vitro and in vivo

Addressing genetic tumor heterogeneity through computationally predictive combination therapy

Exploiting an Asp-Glu "switch" in glycogen synthase kinase 3 to design paralog-selective inhibitors for use in acute myeloid leukemia

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