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H2O2 Mediates VEGF- and Flow-Induced Dilations of Coronary Arterioles in Early Type 1 Diabetes: Role of Vascular Arginase and PI3K-Linked eNOS Uncoupling

International journal of molecular sciences, 2022-12, Vol.24 (1), p.489

2022

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Role of Arginase in Selective Impairment of Endothelium-Dependent Nitric Oxide Synthase-Mediated Dilation of Retinal Arterioles during Early Diabetes

Investigative ophthalmology & visual science, 2020-05, Vol.61 (5), p.36-36

2020

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Organ-on-chips made of blood: endothelial progenitor cells from blood reconstitute vascular thromboinflammation in vessel-chips

Lab on a chip, 2019-07, Vol.19 (15), p.25-2511

2019

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Activation of Coronary Arteriolar PKCβ2 Impairs Endothelial NO-Mediated Vasodilation: Role of JNK/Rho Kinase Signaling and Xanthine Oxidase Activation

International journal of molecular sciences, 2021-09, Vol.22 (18), p.9763

2021

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Constriction of Retinal Venules to Endothelin-1: Obligatory Roles of ETA Receptors, Extracellular Calcium Entry, and Rho Kinase

Investigative ophthalmology & visual science, 2018-10, Vol.59 (12), p.5167-5175

2018

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Hyperglycemia Enhances Constriction of Retinal Venules via Activation of the Reverse-Mode Sodium-Calcium Exchanger

Diabetes (New York, N.Y.), 2019-08, Vol.68 (8), p.1624-1634

2019

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Acute and Chronic Hyperglycemia Elicit JIP1/JNK-Mediated Endothelial Vasodilator Dysfunction of Retinal Arterioles

Investigative ophthalmology & visual science, 2016-08, Vol.57 (10), p.4333-4340

2016

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Endothelin-1 impairs coronary arteriolar dilation: Role of p38 kinase-mediated superoxide production from NADPH oxidase

Journal of molecular and cellular cardiology, 2015-09, Vol.86, p.75-84

2015

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Human C-reactive protein induces endothelial dysfunction and uncoupling of eNOS in vivo

Atherosclerosis, 2009-09, Vol.206 (1), p.61-68

2009

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Modulation of the Tryptophan Hydroxylase 1/Monoamine Oxidase‐A/5‐Hydroxytryptamine/5‐Hydroxytryptamine Receptor 2A/2B/2C Axis Regulates Biliary Proliferation and Liver Fibrosis During Cholestasis

Hepatology (Baltimore, Md.), 2020-03, Vol.71 (3), p.990-1008

2020

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Chronic hypoxia attenuates VEGF signaling and angiogenic responses by downregulation of KDR in human endothelial cells

American Journal of Physiology: Cell Physiology, 2009-05, Vol.296 (5), p.C1162-C1170

2009

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Histamine-Induced Dilation of Isolated Porcine Retinal Arterioles: Role of Endothelium-Derived Hyperpolarizing Factor

Investigative ophthalmology & visual science, 2016-09, Vol.57 (11), p.4791-4798

2016

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Alterations of Ocular Hemodynamics Impair Ophthalmic Vascular and Neuroretinal Function

The American journal of pathology, 2018-03, Vol.188 (3), p.818-827

2018

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Selective Activation of Lectin-Like Oxidized Low-Density Lipoprotein Receptor-1 Mediates C-Reactive Protein–Evoked Endothelial Vasodilator Dysfunction in Coronary Arterioles

Circulation research, 2014-01, Vol.114 (1), p.92-100

2014

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Resveratrol, a Component of Red Wine, Elicits Dilation of Isolated Porcine Retinal Arterioles: Role of Nitric Oxide and Potassium Channels

Investigative ophthalmology & visual science, 2007-09, Vol.48 (9), p.4232-4239

2007

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Temporal development of retinal arteriolar endothelial dysfunction in porcine type 1 diabetes

Investigative ophthalmology & visual science, 2012-12, Vol.53 (13), p.7943-7949

2012

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Vasomotor regulation of coronary microcirculation by oxidative stress: role of arginase

Frontiers in immunology, 2013-01, Vol.4, p.237-237

2013

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Dilation of Retinal Arterioles in Response to Lactate: Role of Nitric Oxide, Guanylyl Cyclase, and ATP-Sensitive Potassium Channels

Investigative ophthalmology & visual science, 2006-02, Vol.47 (2), p.693-699

2006

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Divergent Roles of Nitric Oxide and Rho Kinase in Vasomotor Regulation of Human Retinal Arterioles

Investigative ophthalmology & visual science, 2010-03, Vol.51 (3), p.1583-1590

2010

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C-Reactive Protein Inhibits Endothelium-Dependent Nitric Oxide-Mediated Dilation of Retinal Arterioles via Enhanced Superoxide Production

Investigative ophthalmology & visual science, 2008-05, Vol.49 (5), p.2053-2060

2008

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Upregulation of Vascular Arginase in Hypertension Decreases Nitric Oxide–Mediated Dilation of Coronary Arterioles

Hypertension (Dallas, Tex. 1979), 2004-12, Vol.44 (6), p.935-943

2004

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Constriction of retinal arterioles to endothelin-1: requisite role of rho kinase independent of protein kinase C and L-type calcium channels

Investigative ophthalmology & visual science, 2012-05, Vol.53 (6), p.2904-2912

2012

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Intravascular Flow Decreases Erythrocyte Consumption of Nitric Oxide

Proceedings of the National Academy of Sciences - PNAS, 1999-07, Vol.96 (15), p.8757-8761

1999

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Role of endothelium in vasomotor responses to endothelin system and protein kinase C activation in porcine retinal arterioles

Investigative ophthalmology & visual science, 2013-11, Vol.54 (12), p.7587

2013

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Enhanced endothelin-1/Rho-kinase signalling and coronary microvascular dysfunction in hypertensive myocardial hypertrophy

Cardiovascular research, 2017-09, Vol.113 (11), p.1329-1337

2017

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H2O2 Mediates VEGF- and Flow-Induced Dilations of Coronary Arterioles in Early Type 1 Diabetes: Role of Vascular Arginase and PI3K-Linked eNOS Uncoupling

Role of Arginase in Selective Impairment of Endothelium-Dependent Nitric Oxide Synthase-Mediated Dilation of Retinal Arterioles during Early Diabetes

Organ-on-chips made of blood: endothelial progenitor cells from blood reconstitute vascular thromboinflammation in vessel-chips

Activation of Coronary Arteriolar PKCβ2 Impairs Endothelial NO-Mediated Vasodilation: Role of JNK/Rho Kinase Signaling and Xanthine Oxidase Activation

Constriction of Retinal Venules to Endothelin-1: Obligatory Roles of ETA Receptors, Extracellular Calcium Entry, and Rho Kinase

Hyperglycemia Enhances Constriction of Retinal Venules via Activation of the Reverse-Mode Sodium-Calcium Exchanger

Acute and Chronic Hyperglycemia Elicit JIP1/JNK-Mediated Endothelial Vasodilator Dysfunction of Retinal Arterioles

Endothelin-1 impairs coronary arteriolar dilation: Role of p38 kinase-mediated superoxide production from NADPH oxidase

Human C-reactive protein induces endothelial dysfunction and uncoupling of eNOS in vivo

Modulation of the Tryptophan Hydroxylase 1/Monoamine Oxidase‐A/5‐Hydroxytryptamine/5‐Hydroxytryptamine Receptor 2A/2B/2C Axis Regulates Biliary Proliferation and Liver Fibrosis During Cholestasis

Chronic hypoxia attenuates VEGF signaling and angiogenic responses by downregulation of KDR in human endothelial cells

Histamine-Induced Dilation of Isolated Porcine Retinal Arterioles: Role of Endothelium-Derived Hyperpolarizing Factor

Alterations of Ocular Hemodynamics Impair Ophthalmic Vascular and Neuroretinal Function

Selective Activation of Lectin-Like Oxidized Low-Density Lipoprotein Receptor-1 Mediates C-Reactive Protein–Evoked Endothelial Vasodilator Dysfunction in Coronary Arterioles

Resveratrol, a Component of Red Wine, Elicits Dilation of Isolated Porcine Retinal Arterioles: Role of Nitric Oxide and Potassium Channels

Temporal development of retinal arteriolar endothelial dysfunction in porcine type 1 diabetes

Vasomotor regulation of coronary microcirculation by oxidative stress: role of arginase

Dilation of Retinal Arterioles in Response to Lactate: Role of Nitric Oxide, Guanylyl Cyclase, and ATP-Sensitive Potassium Channels

Divergent Roles of Nitric Oxide and Rho Kinase in Vasomotor Regulation of Human Retinal Arterioles

C-Reactive Protein Inhibits Endothelium-Dependent Nitric Oxide-Mediated Dilation of Retinal Arterioles via Enhanced Superoxide Production

Upregulation of Vascular Arginase in Hypertension Decreases Nitric Oxide–Mediated Dilation of Coronary Arterioles

Constriction of retinal arterioles to endothelin-1: requisite role of rho kinase independent of protein kinase C and L-type calcium channels

Intravascular Flow Decreases Erythrocyte Consumption of Nitric Oxide

Role of endothelium in vasomotor responses to endothelin system and protein kinase C activation in porcine retinal arterioles

Enhanced endothelin-1/Rho-kinase signalling and coronary microvascular dysfunction in hypertensive myocardial hypertrophy

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