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From promises to practical strategies in epigenetic epidemiology

Nature reviews. Genetics, 2013-08, Vol.14 (8), p.585-594

2013

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DNA methylation aging clocks: challenges and recommendations

Genome Biology, 2019-11, Vol.20 (1), p.249-24, Article 249

2019

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Commentary: The seven plagues of epigenetic epidemiology

International journal of epidemiology, 2012-02, Vol.41 (1), p.74-78

2012

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Omixer: multivariate and reproducible sample randomization to proactively counter batch effects in omics studies

Bioinformatics (Oxford, England), 2021-09, Vol.37 (18), p.3051-3052

2021

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DNA methylation signatures link prenatal famine exposure to growth and metabolism

Nature communications, 2014-11, Vol.5 (1), p.5592-5592, Article 5592

2014

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Persistent epigenetic differences associated with prenatal exposure to famine in humans

Proceedings of the National Academy of Sciences - PNAS, 2008-11, Vol.105 (44), p.17046-17049

2008

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DNA methylation differences after exposure to prenatal famine are common and timing- and sex-specific

Human molecular genetics, 2009-11, Vol.18 (21), p.4046-4053

2009

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DNA Methylation Landscapes of Human Fetal Development

PLoS genetics, 2015-10, Vol.11 (10), p.e1005583-e1005583

2015

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Data Resource Profile: Accessible Resource for Integrated Epigenomic Studies (ARIES)

International journal of epidemiology, 2015-08, Vol.44 (4), p.1181-1190

2015

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Age-related DNA methylation changes are tissue-specific with ELOVL2 promoter methylation as exception

Epigenetics & chromatin, 2018-05, Vol.11 (1), p.25-25, Article 25

2018

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Prenatal famine and genetic variation are independently and additively associated with DNA methylation at regulatory loci within IGF2/H19

PloS one, 2012-05, Vol.7 (5), p.e37933

2012

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Effects of fatty acids on T cell function: role in atherosclerosis

Nature reviews cardiology, 2021-12, Vol.18 (12), p.824-837

2021

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Epigenetic variation during the adult lifespan: cross‐sectional and longitudinal data on monozygotic twin pairs

Aging cell, 2012-08, Vol.11 (4), p.694-703

2012

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omicsPrint: detection of data linkage errors in multiple omics studies

Bioinformatics, 2018-06, Vol.34 (12), p.2142-2143

2018

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Controlling bias and inflation in epigenome- and transcriptome-wide association studies using the empirical null distribution

Genome Biology, 2017-01, Vol.18 (1), p.19-19, Article 19

2017

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Early gestation as the critical time-window for changes in the prenatal environment to affect the adult human blood methylome

International journal of epidemiology, 2015-08, Vol.44 (4), p.1211-1223

2015

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Variation, patterns, and temporal stability of DNA methylation: considerations for epigenetic epidemiology

The FASEB journal, 2010-09, Vol.24 (9), p.3135-3144

2010

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Alternative Routes to Induce Naïve Pluripotency in Human Embryonic Stem Cells

Stem cells (Dayton, Ohio), 2015-09, Vol.33 (9), p.2686-2698

2015

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Functional genomics analysis identifies T and NK cell activation as a driver of epigenetic clock progression

Genome Biology, 2022-01, Vol.23 (1), p.24-24, Article 24

2022

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MethylAid: visual and interactive quality control of large Illumina 450k datasets

Bioinformatics, 2014-12, Vol.30 (23), p.3435-3437

2014

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Epigenome-wide change and variation in DNA methylation in childhood: trajectories from birth to late adolescence

Human molecular genetics, 2021-03, Vol.30 (1), p.119-134

2021

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The effect of the Chinese Famine on type 2 diabetes mellitus epidemics

Nature reviews. Endocrinology, 2019-06, Vol.15 (6), p.313-314

2019

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Epigenetic drift in the aging genome: a ten-year follow-up in an elderly twin cohort

International journal of epidemiology, 2016-08, Vol.45 (4), p.1146-1158

2016

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Adults prenatally exposed to the Dutch Famine exhibit a metabolic signature associated with a broad spectrum of common diseases

BMC medicine, 2024-07, Vol.22 (1), p.309-10, Article 309

2024

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Mesenchymal stromal cells as a tool to unravel the developmental origins of disease

Trends in endocrinology and metabolism, 2022-09, Vol.33 (9), p.614-627

2022

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From promises to practical strategies in epigenetic epidemiology

DNA methylation aging clocks: challenges and recommendations

Commentary: The seven plagues of epigenetic epidemiology

Omixer: multivariate and reproducible sample randomization to proactively counter batch effects in omics studies

DNA methylation signatures link prenatal famine exposure to growth and metabolism

Persistent epigenetic differences associated with prenatal exposure to famine in humans

DNA methylation differences after exposure to prenatal famine are common and timing- and sex-specific

DNA Methylation Landscapes of Human Fetal Development

Data Resource Profile: Accessible Resource for Integrated Epigenomic Studies (ARIES)

Age-related DNA methylation changes are tissue-specific with ELOVL2 promoter methylation as exception

Prenatal famine and genetic variation are independently and additively associated with DNA methylation at regulatory loci within IGF2/H19

Effects of fatty acids on T cell function: role in atherosclerosis

Epigenetic variation during the adult lifespan: cross‐sectional and longitudinal data on monozygotic twin pairs

omicsPrint: detection of data linkage errors in multiple omics studies

Controlling bias and inflation in epigenome- and transcriptome-wide association studies using the empirical null distribution

Early gestation as the critical time-window for changes in the prenatal environment to affect the adult human blood methylome

Variation, patterns, and temporal stability of DNA methylation: considerations for epigenetic epidemiology

Alternative Routes to Induce Naïve Pluripotency in Human Embryonic Stem Cells

Functional genomics analysis identifies T and NK cell activation as a driver of epigenetic clock progression

MethylAid: visual and interactive quality control of large Illumina 450k datasets

Epigenome-wide change and variation in DNA methylation in childhood: trajectories from birth to late adolescence

The effect of the Chinese Famine on type 2 diabetes mellitus epidemics

Epigenetic drift in the aging genome: a ten-year follow-up in an elderly twin cohort

Adults prenatally exposed to the Dutch Famine exhibit a metabolic signature associated with a broad spectrum of common diseases

Mesenchymal stromal cells as a tool to unravel the developmental origins of disease

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