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Using Zebrafish as a Model System for Studying the Transgenerational Effects of Dioxin

Toxicological sciences, 2014-04, Vol.138 (2), p.403-411

2014

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Cardiac myocyte-specific AHR activation phenocopies TCDD-induced toxicity in zebrafish

Toxicological sciences, 2014-09, Vol.141 (1), p.141-154

2014

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Dioxin disrupts cranial cartilage and dermal bone development in zebrafish larvae

Aquatic toxicology, 2015-07, Vol.164, p.52-60

2015

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Dioxin inhibits zebrafish epicardium and proepicardium development

Toxicological sciences, 2013-02, Vol.131 (2), p.558-567

2013

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Heart Malformation Is an Early Response to TCDD in Embryonic Zebrafish

Toxicological sciences, 2005-04, Vol.84 (2), p.368-377

2005

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Aryl Hydrocarbon Receptor-Mediated Down-Regulation of Sox9b Causes Jaw Malformation in Zebrafish Embryos

Molecular pharmacology, 2008-12, Vol.74 (6), p.1544-1553

2008

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sox9b is required in cardiomyocytes for cardiac morphogenesis and function

Scientific reports, 2018-09, Vol.8 (1), p.13906-16, Article 13906

2018

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Developmental toxicity of low generation PAMAM dendrimers in zebrafish

Toxicology and applied pharmacology, 2007-11, Vol.225 (1), p.70-79

2007

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2,3,7,8-Tetrachlorodibenzo-p-dioxin Activation of the Aryl Hydrocarbon Receptor/Aryl Hydrocarbon Receptor Nuclear Translocator Pathway Causes Developmental Toxicity through a CYP1A-Independent Mechanism in Zebrafish

Molecular pharmacology, 2004-09, Vol.66 (3), p.512-521

2004

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Sensitivity to dioxin decreases as zebrafish mature

Toxicological sciences, 2012-06, Vol.127 (2), p.360-370

2012

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Stb3 Binds to Ribosomal RNA Processing Element Motifs That Control Transcriptional Responses to Growth in Saccharomyces cerevisiae

The Journal of biological chemistry, 2007-09, Vol.282 (36), p.26623-26628

2007

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Persistent Adverse Effects on Health and Reproduction Caused by Exposure of Zebrafish to 2,3,7,8-Tetrachlorodibenzo-p-dioxin During Early Development and Gonad Differentiation

Toxicological sciences, 2009-05, Vol.109 (1), p.75-87

2009

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Aryl Hydrocarbon Receptor Activation Produces Heart-Specific Transcriptional and Toxic Responses in Developing Zebrafish

Molecular pharmacology, 2006-08, Vol.70 (2), p.549-561

2006

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Understanding dioxin developmental toxicity using the zebrafish model

Birth defects research. A Clinical and molecular teratology, 2006-01, Vol.76 (1), p.7-18

2006

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Glucose, nitrogen, and phosphate repletion in Saccharomyces cerevisiae: common transcriptional responses to different nutrient signals

G3 : genes - genomes - genetics, 2012-09, Vol.2 (9), p.1003-1017

2012

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Protein Kinase A, TOR, and Glucose Transport Control the Response to Nutrient Repletion in Saccharomyces cerevisiae

Eukaryotic Cell, 2008-02, Vol.7 (2), p.358-367

2008

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A co-culture assay of embryonic zebrafish hearts to assess migration of epicardial cells in vitro

BMC developmental biology, 2015-12, Vol.15 (51), p.50-50, Article 50

2015

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Identification of Zebrafish ARNT1 Homologs: 2,3,7,8-Tetrachlorodibenzo-p-dioxin Toxicity in the Developing Zebrafish Requires ARNT1

Molecular pharmacology, 2006-03, Vol.69 (3), p.776-787

2006

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The Zebrafish (Danio rerio) Aryl Hydrocarbon Receptor Type 1 Is a Novel Vertebrate Receptor

Molecular pharmacology, 2002-08, Vol.62 (2), p.234-249

2002

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A dominant negative zebrafish Ahr2 partially protects developing zebrafish from dioxin toxicity

PloS one, 2011-12, Vol.6 (12), p.e28020

2011

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Disruption of erythropoiesis by dioxin in the zebrafish

Developmental dynamics, 2001-12, Vol.222 (4), p.581-594

2001

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Coordinated Regulation of Growth Genes in Saccharomyces cerevisiae

Cell cycle (Georgetown, Tex.), 2007-05, Vol.6 (10), p.1210-1219

2007

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The Function and Properties of the Azf1 Transcriptional Regulator Change with Growth Conditions in Saccharomyces cerevisiae

Eukaryotic Cell, 2006-02, Vol.5 (2), p.313-320

2006

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Using zebrafish as a model system for studying the transgenerational effects of dioxin

Toxicological sciences, 2014-04, Vol.138 (2), p.403-411

2014

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Zebrafish as a Model Vertebrate for Investigating Chemical Toxicity

Toxicological sciences, 2005-07, Vol.86 (1), p.6-19

2005

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Using Zebrafish as a Model System for Studying the Transgenerational Effects of Dioxin

Cardiac myocyte-specific AHR activation phenocopies TCDD-induced toxicity in zebrafish

Dioxin disrupts cranial cartilage and dermal bone development in zebrafish larvae

Dioxin inhibits zebrafish epicardium and proepicardium development

Heart Malformation Is an Early Response to TCDD in Embryonic Zebrafish

Aryl Hydrocarbon Receptor-Mediated Down-Regulation of Sox9b Causes Jaw Malformation in Zebrafish Embryos

sox9b is required in cardiomyocytes for cardiac morphogenesis and function

Developmental toxicity of low generation PAMAM dendrimers in zebrafish

2,3,7,8-Tetrachlorodibenzo-p-dioxin Activation of the Aryl Hydrocarbon Receptor/Aryl Hydrocarbon Receptor Nuclear Translocator Pathway Causes Developmental Toxicity through a CYP1A-Independent Mechanism in Zebrafish

Sensitivity to dioxin decreases as zebrafish mature

Stb3 Binds to Ribosomal RNA Processing Element Motifs That Control Transcriptional Responses to Growth in Saccharomyces cerevisiae

Persistent Adverse Effects on Health and Reproduction Caused by Exposure of Zebrafish to 2,3,7,8-Tetrachlorodibenzo-p-dioxin During Early Development and Gonad Differentiation

Aryl Hydrocarbon Receptor Activation Produces Heart-Specific Transcriptional and Toxic Responses in Developing Zebrafish

Understanding dioxin developmental toxicity using the zebrafish model

Glucose, nitrogen, and phosphate repletion in Saccharomyces cerevisiae: common transcriptional responses to different nutrient signals

Protein Kinase A, TOR, and Glucose Transport Control the Response to Nutrient Repletion in Saccharomyces cerevisiae

A co-culture assay of embryonic zebrafish hearts to assess migration of epicardial cells in vitro

Identification of Zebrafish ARNT1 Homologs: 2,3,7,8-Tetrachlorodibenzo-p-dioxin Toxicity in the Developing Zebrafish Requires ARNT1

The Zebrafish (Danio rerio) Aryl Hydrocarbon Receptor Type 1 Is a Novel Vertebrate Receptor

A dominant negative zebrafish Ahr2 partially protects developing zebrafish from dioxin toxicity

Disruption of erythropoiesis by dioxin in the zebrafish

Coordinated Regulation of Growth Genes in Saccharomyces cerevisiae

The Function and Properties of the Azf1 Transcriptional Regulator Change with Growth Conditions in Saccharomyces cerevisiae

Using zebrafish as a model system for studying the transgenerational effects of dioxin

Zebrafish as a Model Vertebrate for Investigating Chemical Toxicity

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