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β-adrenergic regulation of late Na+ current during cardiac action potential is mediated by both PKA and CaMKII

Journal of molecular and cellular cardiology, 2018-10, Vol.123, p.168-179

2018

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Metabolic Maturation Media Improve Physiological Function of Human iPSC-Derived Cardiomyocytes

Cell reports (Cambridge), 2020-07, Vol.32 (3), p.107925-107925, Article 107925

2020

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Cardiomyocyte Na+ and Ca2+ mishandling drives vicious cycle involving CaMKII, ROS, and ryanodine receptors

Basic research in cardiology, 2021-12, Vol.116 (1), p.58-58, Article 58

2021

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CaMKII Serine 280 O-GlcNAcylation Links Diabetic Hyperglycemia to Proarrhythmia

Circulation research, 2021-06, Vol.129 (1), p.98-113

2021

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Hyperglycemia regulates cardiac K+ channels via O-GlcNAc-CaMKII and NOX2-ROS-PKC pathways

Basic research in cardiology, 2020-12, Vol.115 (6), p.71-71, Article 71

2020

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Complex electrophysiological remodeling in postinfarction ischemic heart failure

Proceedings of the National Academy of Sciences - PNAS, 2018-03, Vol.115 (13), p.E3036-E3044

2018

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Dynamics of the late Na+ current during cardiac action potential and its contribution to afterdepolarizations

Journal of molecular and cellular cardiology, 2013-11, Vol.64, p.59-68

2013

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Diabetes and Excess Aldosterone Promote Heart Failure With Preserved Ejection Fraction

Journal of the American Heart Association, 2022-12, Vol.11 (23), p.e027164-e027164

2022

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Protein Kinase D1 Regulates Cardiac Hypertrophy, Potassium Channel Remodeling, and Arrhythmias in Heart Failure

Journal of the American Heart Association, 2022-10, Vol.11 (19), p.e027573-e027573

2022

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Electrophysiological Determination of Submembrane Na+ Concentration in Cardiac Myocytes

Biophysical journal, 2016-09, Vol.111 (6), p.1304-1315

2016

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Emergence of Mechano-Sensitive Contraction Autoregulation in Cardiomyocytes

Life (Basel, Switzerland), 2021-05, Vol.11 (6), p.503

2021

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The Janus face of adenosine: antiarrhythmic and proarrhythmic actions

Current pharmaceutical design, 2015-01, Vol.21 (8), p.965-976

2015

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CaMKII signaling in heart diseases: Emerging role in diabetic cardiomyopathy

Journal of molecular and cellular cardiology, 2019-02, Vol.127, p.246-259

2019

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Distinct microdomain of sodium channel complexes with intracellular fibroblast growth factors in cardiomyocytes

Biophysical journal, 2024-02, Vol.123 (3), p.386a-387a

2024

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Chemistry, physiology, and pharmacology of β-adrenergic mechanisms in the heart. Why are β-blocker antiarrhythmics superior?

Current pharmaceutical design, 2015-01, Vol.21 (8), p.1030-1041

2015

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Selectivity Problems with Drugs Acting on Cardiac Na + and Ca 2+ Channels

Current medicinal chemistry, 2013-05, Vol.20 (20), p.2552-2571

2013

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Class IV antiarrhythmic agents: new compounds using an old strategy

Current pharmaceutical design, 2015-01, Vol.21 (8), p.977-1010

2015

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Intracellular fibroblast growth factors regulate the arrhythmogenic late sodium current in heart failure

Biophysical journal, 2024-02, Vol.123 (3), p.191a-191a

2024

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Quantitative investigation of the mechanisms of ventricular myocyte dysfunction in a murine model of heart failure with preserved ejection fraction

Biophysical journal, 2024-02, Vol.123 (3), p.457a-457a

2024

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New cardiac targets for empagliflozin: O -GlcNAcylation, CaMKII, and calcium handling

American journal of physiology. Heart and circulatory physiology, 2023-03, Vol.324 (3), p.H338-H340

2023

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Two-hit mechanism of cardiac arrhythmias in diabetic hyperglycaemia: reduced repolarization reserve, neurohormonal stimulation, and heart failure exacerbate susceptibility

Cardiovascular research, 2021-12, Vol.117 (14), p.2781-2793

2021

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Tetrodotoxin blockade on canine cardiac L-type Ca²⁺ channels depends on pH and redox potential

Marine drugs, 2013-06, Vol.11 (6), p.2140-2153

2013

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Determining sex-driven excitation-contraction coupling mechanisms in young populations

Biophysical journal, 2024-02, Vol.123 (3), p.383a-383a

2024

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Empagliflozin Reverses Late Na + Current Enhancement and Cardiomyocyte Proarrhythmia in a Translational Murine Model of Heart Failure With Preserved Ejection Fraction

Circulation (New York, N.Y.), 2022-03, Vol.145 (13), p.1029-1031

2022

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A de novo designed peptide selectivity inhibits arrhythmogenic late Na+ current

Biophysical journal, 2024-02, Vol.123 (3), p.12a-12a

2024

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β-adrenergic regulation of late Na+ current during cardiac action potential is mediated by both PKA and CaMKII

Metabolic Maturation Media Improve Physiological Function of Human iPSC-Derived Cardiomyocytes

Cardiomyocyte Na+ and Ca2+ mishandling drives vicious cycle involving CaMKII, ROS, and ryanodine receptors

CaMKII Serine 280 O-GlcNAcylation Links Diabetic Hyperglycemia to Proarrhythmia

Hyperglycemia regulates cardiac K+ channels via O-GlcNAc-CaMKII and NOX2-ROS-PKC pathways

Complex electrophysiological remodeling in postinfarction ischemic heart failure

Dynamics of the late Na+ current during cardiac action potential and its contribution to afterdepolarizations

Diabetes and Excess Aldosterone Promote Heart Failure With Preserved Ejection Fraction

Protein Kinase D1 Regulates Cardiac Hypertrophy, Potassium Channel Remodeling, and Arrhythmias in Heart Failure

Electrophysiological Determination of Submembrane Na+ Concentration in Cardiac Myocytes

Emergence of Mechano-Sensitive Contraction Autoregulation in Cardiomyocytes

The Janus face of adenosine: antiarrhythmic and proarrhythmic actions

CaMKII signaling in heart diseases: Emerging role in diabetic cardiomyopathy

Distinct microdomain of sodium channel complexes with intracellular fibroblast growth factors in cardiomyocytes

Chemistry, physiology, and pharmacology of β-adrenergic mechanisms in the heart. Why are β-blocker antiarrhythmics superior?

Selectivity Problems with Drugs Acting on Cardiac Na + and Ca 2+ Channels

Class IV antiarrhythmic agents: new compounds using an old strategy

Intracellular fibroblast growth factors regulate the arrhythmogenic late sodium current in heart failure

Quantitative investigation of the mechanisms of ventricular myocyte dysfunction in a murine model of heart failure with preserved ejection fraction

New cardiac targets for empagliflozin: O -GlcNAcylation, CaMKII, and calcium handling

Two-hit mechanism of cardiac arrhythmias in diabetic hyperglycaemia: reduced repolarization reserve, neurohormonal stimulation, and heart failure exacerbate susceptibility

Tetrodotoxin blockade on canine cardiac L-type Ca²⁺ channels depends on pH and redox potential

Determining sex-driven excitation-contraction coupling mechanisms in young populations

Empagliflozin Reverses Late Na + Current Enhancement and Cardiomyocyte Proarrhythmia in a Translational Murine Model of Heart Failure With Preserved Ejection Fraction

A de novo designed peptide selectivity inhibits arrhythmogenic late Na+ current

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