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The Angiogenic Growth Factors HGF and VEGF in Serum and Plasma from Neuroblastoma Patients

Anticancer research, 2009-08, Vol.29 (8), p.3311-3319

2009

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Differentiation in neuroblastoma: diffusion-limited hypoxia induces neuro-endocrine secretory protein 55 and other markers of a chromaffin phenotype

PloS one, 2010-09, Vol.5 (9), p.e12825

2010

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Emergence of new ALK mutations at relapse of neuroblastoma

Journal of clinical oncology, 2014-09, Vol.32 (25), p.2727-2734

2014

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Classification, incidence and survival analyses of children with CNS tumours diagnosed in Sweden 1984-2005

Acta Paediatrica, 2009-10, Vol.98 (10), p.1620-1627

2009

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Inhibition of Angiogenesis Induces Chromaffin Differentiation and Apoptosis in Neuroblastoma

The American journal of pathology, 1999-02, Vol.154 (2), p.395-403

1999

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Dynamic and selective HERV RNA expression in neuroblastoma cells subjected to variation in oxygen tension and demethylation

APMIS : acta pathologica, microbiologica et immunologica Scandinavica, 2016-01, Vol.124 (1-2), p.140-149

2016

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Age dependence of tumor genetics in unfavorable neuroblastoma: arrayCGH profiles of 34 consecutive cases, using a Swedish 25-year neuroblastoma cohort for validation

BMC cancer, 2013-05, Vol.13 (1), p.231-231, Article 231

2013

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Weekly vinblastine is a therapeutic option in recurrent/refractory pediatric low-grade gliomas

Neuro-oncology (Charlottesville, Va.), 2012, Vol.14 (suppl 1), p.i70

2012

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Emergence of new ALK mutations at relapse of neuroblastoma

Journal of clinical oncology, 2014-05, Vol.32 (15_suppl), p.11006-11006

2014

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Revisions of the international criteria for neuroblastoma diagnosis, staging, and response to treatment

Journal of clinical oncology, 1993-08, Vol.11 (8), p.1466-1477

1993

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Nuclear receptor binding protein 2 is induced during neural progenitor differentiation and affects cell survival

Molecular and cellular neuroscience, 2008-09, Vol.39 (1), p.32-39

2008

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DIFFERENCES IN BIOLOGICAL FEATURES AND SURVIVAL IMPROVEMENT BETWEEN GENETIC SUBSETS OF HIGH-RISK NEUROBLASTOMA INDICATE THE NEED OF ADAPTED TREATMENT

PEDIATRIC BLOOD & CANCER, 2010, Vol.55 (5), p.897

2010

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AGE-DEPENDENT GENOTYPES IN HIGH-RISK NEUROBLASTOMA: MYCN AMPLIFICATION IS A FAST TRACK TO AGGRESSIVE DISEASE WHEREAS SEGMENTAL DELETION OF 11Q IMPLIES A MORE COMPLEX, MULTI-STEP TUMOR EVOLUTION

PEDIATRIC BLOOD & CANCER, 2010, Vol.55 (5), p.896

2010

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A developmental model of neuroblastoma: differentiating stroma-poor tumors' progress along an extra-adrenal chromaffin lineage

Laboratory investigation, 1996-11, Vol.75 (5), p.659

1996

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IGF2 is parentally imprinted during human embryogenesis and in the Beckwith-Wiedemann syndrome

Nature genetics, 1993-05, Vol.4 (1), p.94-97

1993

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Deletion of chromosome 1p loci and microsatellite instability in neuroblastomas analyzed with short-tandem repeat polymorphisms

Cancer research (Chicago, Ill.), 1995-12, Vol.55 (23), p.5681-5686

1995

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Overlapping patterns of IGF2 and H19 expression during human development: biallelic IGF2 expression correlates with a lack of H19 expression

Development (Cambridge), 1994-02, Vol.120 (2), p.361-368

1994

Volltextzugriff (PDF)

The cell type-specific IGF2 expression during early human development correlates to the pattern of overgrowth and neoplasia in the Beckwith- Wiedemann syndrome

The American journal of pathology, 1994-10, Vol.145 (4), p.802-817

1994

Volltextzugriff (PDF)

Evidence for hypoxia‐induced neuronal‐to‐chromaffin metaplasia in neuroblastoma

The FASEB journal, 2003-04, Vol.17 (6), p.598-609

2003

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Developmental gene expression of sympathetic nervous system tumors reflects their histogenesis

Laboratory investigation, 1998-01, Vol.78 (1), p.29

1998

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Inactivation of H19, an imprinted and putative tumor repressor gene, is a preneoplastic event during Wilms' tumorigenesis

Cancer research (Chicago, Ill.), 1997-10, Vol.57 (20), p.4469-4473

1997

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Developmental aspects of neuroblastoma and of the childhood cancer predisposing Beckwith-Wiedemann syndrome

Hedborg, Fredrik Johan Olov

1994

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Mosaic allelic insulin-like growth factor 2 expression patterns reveal a link between Wilms' tumorigenesis and epigenetic heterogeneity

Cancer research (Chicago, Ill.), 1999-08, Vol.59 (16), p.3889-3892

1999

Volltextzugriff (PDF)

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The Risk-Associated Long Noncoding RNA NBAT-1 Controls Neuroblastoma Progression by Regulating Cell Proliferation and Neuronal Differentiation

The Angiogenic Growth Factors HGF and VEGF in Serum and Plasma from Neuroblastoma Patients

Differentiation in neuroblastoma: diffusion-limited hypoxia induces neuro-endocrine secretory protein 55 and other markers of a chromaffin phenotype

Emergence of new ALK mutations at relapse of neuroblastoma

Classification, incidence and survival analyses of children with CNS tumours diagnosed in Sweden 1984-2005

Inhibition of Angiogenesis Induces Chromaffin Differentiation and Apoptosis in Neuroblastoma

Dynamic and selective HERV RNA expression in neuroblastoma cells subjected to variation in oxygen tension and demethylation

Age dependence of tumor genetics in unfavorable neuroblastoma: arrayCGH profiles of 34 consecutive cases, using a Swedish 25-year neuroblastoma cohort for validation

Weekly vinblastine is a therapeutic option in recurrent/refractory pediatric low-grade gliomas

Emergence of new ALK mutations at relapse of neuroblastoma

The Risk-Associated Long Noncoding RNA NBAT-1Controls Neuroblastoma Progression by RegulatingCell Proliferation and Neuronal Differentiation

Revisions of the international criteria for neuroblastoma diagnosis, staging, and response to treatment

Nuclear receptor binding protein 2 is induced during neural progenitor differentiation and affects cell survival

DIFFERENCES IN BIOLOGICAL FEATURES AND SURVIVAL IMPROVEMENT BETWEEN GENETIC SUBSETS OF HIGH-RISK NEUROBLASTOMA INDICATE THE NEED OF ADAPTED TREATMENT

AGE-DEPENDENT GENOTYPES IN HIGH-RISK NEUROBLASTOMA: MYCN AMPLIFICATION IS A FAST TRACK TO AGGRESSIVE DISEASE WHEREAS SEGMENTAL DELETION OF 11Q IMPLIES A MORE COMPLEX, MULTI-STEP TUMOR EVOLUTION

A developmental model of neuroblastoma: differentiating stroma-poor tumors' progress along an extra-adrenal chromaffin lineage

IGF2 is parentally imprinted during human embryogenesis and in the Beckwith-Wiedemann syndrome

Deletion of chromosome 1p loci and microsatellite instability in neuroblastomas analyzed with short-tandem repeat polymorphisms

Overlapping patterns of IGF2 and H19 expression during human development: biallelic IGF2 expression correlates with a lack of H19 expression

The cell type-specific IGF2 expression during early human development correlates to the pattern of overgrowth and neoplasia in the Beckwith- Wiedemann syndrome

Evidence for hypoxia‐induced neuronal‐to‐chromaffin metaplasia in neuroblastoma

Developmental gene expression of sympathetic nervous system tumors reflects their histogenesis

Inactivation of H19, an imprinted and putative tumor repressor gene, is a preneoplastic event during Wilms' tumorigenesis

Developmental aspects of neuroblastoma and of the childhood cancer predisposing Beckwith-Wiedemann syndrome

Mosaic allelic insulin-like growth factor 2 expression patterns reveal a link between Wilms' tumorigenesis and epigenetic heterogeneity

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