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CAR T cells redirected to cell surface GRP78 display robust anti-acute myeloid leukemia activity and do not target hematopoietic progenitor cells

Nature communications, 2022-01, Vol.13 (1), p.587-14, Article 587

2022

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Mechanisms of apoptosis by the tumor suppressor Par-4

Journal of cellular physiology, 2012-12, Vol.227 (12), p.3715-3721

2012

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Improving the anti-acute myeloid leukemia activity of CD123-specific engager T cells by MyD88 and CD40 costimulation

Haematologica (Roma), 2023-04, Vol.108 (4), p.1039-1052

2023

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Chloroquine-Inducible Par-4 Secretion Is Essential for Tumor Cell Apoptosis and Inhibition of Metastasis

Cell reports (Cambridge), 2017-01, Vol.18 (2), p.508-519

2017

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A Naturally Generated Decoy of the Prostate Apoptosis Response-4 Protein Overcomes Therapy Resistance in Tumors

Cancer research (Chicago, Ill.), 2017-08, Vol.77 (15), p.4039-4050

2017

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Stromal-induced epithelial-mesenchymal transition induces targetable drug resistance in acute lymphoblastic leukemia

Cell reports (Cambridge), 2023-07, Vol.42 (7), p.112804-112804, Article 112804

2023

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The Tumor Suppressor Par-4 Regulates Adipogenesis by Transcriptional Repression of PPARγ

Cells (Basel, Switzerland), 2024-09, Vol.13 (17), p.1495

2024

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Novel Mechanism of Apoptosis Resistance in Cancer Mediated by Extracellular PAR-4

Cancer research (Chicago, Ill.), 2013-01, Vol.73 (2), p.1011-1019

2013

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Protocol for live-cell imaging of immune synapse formation and activation of CAR T cells against cancer cells

STAR protocols, 2024-12, Vol.5 (4), p.103422, Article 103422

2024

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Tumor Suppressor Par-4 Regulates Complement Factor C3 and Obesity

Frontiers in oncology, 2022-03, Vol.12, p.860446

2022

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Paracrine Apoptotic Effect of p53 Mediated by Tumor Suppressor Par-4

Cell reports (Cambridge), 2014-01, Vol.6 (2), p.271-277

2014

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A facile radiosynthesis of [18F]FBA-Pep42, a cyclic peptide targeting glucose-regulated protein 78 (GRP78)

Nuclear medicine and biology, 2021-05, Vol.96-97, p.S72

2021

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SP-064 - A facile radiosynthesis of [18F]FBA-Pep42, a cyclic peptide targeting glucose-regulated protein 78 (GRP78)

Nuclear medicine and biology, 2021-05, Vol.96-97, p.S72-S72

2021

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DIPG-34. IMMUNE SYNAPSE OPTIMIZED GRP78-SPECIFIC CAR T CELLS ELICIT AN IMPROVED ANTITUMOR RESPONSE AGAINST DIPGS

Neuro-oncology (Charlottesville, Va.), 2023-06, Vol.25 (Supplement_1), p.i20-i20

2023

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GRP78 Is Expressed on the Cell Surface of Pediatric AML Blasts and Can be Targeted with GRP78-CAR T Cells without Toxicity to Hematopoietic Progenitor Cells

Blood, 2020-11, Vol.136 (Supplement 1), p.12-12

2020

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EXTH-40. DIPGS SUPPRESS THE EFFICACY OF T CELL THERAPIES THROUGH DYSFUNCTIONAL IMMUNE SYNAPSE FORMATION

Neuro-oncology (Charlottesville, Va.), 2022-11, Vol.24 (Supplement_7), p.vii218-vii218

2022

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GRP78 Is a Targetable Receptor on Cancer and Stromal Cells

EBioMedicine, 2018-07, Vol.33, p.2-3

2018

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Arylquins target vimentin to trigger Par-4 secretion for tumor cell apoptosis

Nature chemical biology, 2014-11, Vol.10 (11), p.924-926

2014

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Development of a novel prostate apoptosis response-4 (Par-4) protein entity with an extended duration of action for therapeutic treatment of cancer

Protein engineering, design and selection, 2019-12, Vol.32 (3), p.159-166

2019

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Multi-Omic Based Antigen Discovery for the Immunotherapy of Pediatric Acute T Cell Lymphoblastic Leukemia

Blood, 2020-11, Vol.136 (Supplement 1), p.17-18

2020

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Par(−4)oxysm in Breast Cancer

Cancer cell, 2013-07, Vol.24 (1), p.3-5

2013

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GRP78-CAR T cell effector function against solid and brain tumors is controlled by GRP78 expression on T cells

Cell reports. Medicine, 2023-11, Vol.4 (11), p.101297, Article 101297

2023

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Abstract A52: Paracrine apoptotic effect of the tumor suppressor p53 is mediated by secreted Par-4

Cancer research (Chicago, Ill.), 2015-01, Vol.75 (1_Supplement), p.A52-A52

2015

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Par-4 prevents breast cancer recurrence

Breast cancer research : BCR, 2013-01, Vol.15 (5), p.314-314, Article 314

2013

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Cancer-selective apoptosis by tumor suppressor par-4

Advances in experimental medicine and biology, 2014, Vol.818, p.155

2014

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CAR T cells redirected to cell surface GRP78 display robust anti-acute myeloid leukemia activity and do not target hematopoietic progenitor cells

Mechanisms of apoptosis by the tumor suppressor Par-4

Improving the anti-acute myeloid leukemia activity of CD123-specific engager T cells by MyD88 and CD40 costimulation

Chloroquine-Inducible Par-4 Secretion Is Essential for Tumor Cell Apoptosis and Inhibition of Metastasis

A Naturally Generated Decoy of the Prostate Apoptosis Response-4 Protein Overcomes Therapy Resistance in Tumors

Stromal-induced epithelial-mesenchymal transition induces targetable drug resistance in acute lymphoblastic leukemia

The Tumor Suppressor Par-4 Regulates Adipogenesis by Transcriptional Repression of PPARγ

Novel Mechanism of Apoptosis Resistance in Cancer Mediated by Extracellular PAR-4

Protocol for live-cell imaging of immune synapse formation and activation of CAR T cells against cancer cells

Tumor Suppressor Par-4 Regulates Complement Factor C3 and Obesity

Paracrine Apoptotic Effect of p53 Mediated by Tumor Suppressor Par-4

A facile radiosynthesis of [18F]FBA-Pep42, a cyclic peptide targeting glucose-regulated protein 78 (GRP78)

SP-064 - A facile radiosynthesis of [18F]FBA-Pep42, a cyclic peptide targeting glucose-regulated protein 78 (GRP78)

DIPG-34. IMMUNE SYNAPSE OPTIMIZED GRP78-SPECIFIC CAR T CELLS ELICIT AN IMPROVED ANTITUMOR RESPONSE AGAINST DIPGS

GRP78 Is Expressed on the Cell Surface of Pediatric AML Blasts and Can be Targeted with GRP78-CAR T Cells without Toxicity to Hematopoietic Progenitor Cells

EXTH-40. DIPGS SUPPRESS THE EFFICACY OF T CELL THERAPIES THROUGH DYSFUNCTIONAL IMMUNE SYNAPSE FORMATION

GRP78 Is a Targetable Receptor on Cancer and Stromal Cells

Arylquins target vimentin to trigger Par-4 secretion for tumor cell apoptosis

Development of a novel prostate apoptosis response-4 (Par-4) protein entity with an extended duration of action for therapeutic treatment of cancer

Multi-Omic Based Antigen Discovery for the Immunotherapy of Pediatric Acute T Cell Lymphoblastic Leukemia

Par(−4)oxysm in Breast Cancer

GRP78-CAR T cell effector function against solid and brain tumors is controlled by GRP78 expression on T cells

Abstract A52: Paracrine apoptotic effect of the tumor suppressor p53 is mediated by secreted Par-4

Par-4 prevents breast cancer recurrence

Cancer-selective apoptosis by tumor suppressor par-4

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