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Age- and gender-related changes in contractile properties of non-atrophied EDL muscle

PloS one, 2010-08, Vol.5 (8), p.e12345-e12345

2010

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Six weeks of N-acetylcysteine antioxidant in drinking water decreases pathological fiber branching in MDX mouse dystrophic fast-twitch skeletal muscle

Frontiers in physiology, 2023-02, Vol.14, p.1109587-1109587

2023

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Lifespan Analysis of Dystrophic mdx Fast-Twitch Muscle Morphology and Its Impact on Contractile Function

Frontiers in physiology, 2021-12, Vol.12, p.771499-771499

2021

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Altered Ca2+ kinetics associated with α-actinin-3 deficiency may explain positive selection for ACTN3 null allele in human evolution

PLoS genetics, 2015-01, Vol.11 (2), p.e1004862-e1004862

2015

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Minocycline Treatment Reduces Mass and Force Output From Fast-Twitch Mouse Muscles and Inhibits Myosin Production in C2C12 Myotubes

Frontiers in physiology, 2021-07, Vol.12, p.696039-696039

2021

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Absence of the Z-disc protein α-actinin-3 impairs the mechanical stability of Actn3KO mouse fast-twitch muscle fibres without altering their contractile properties or twitch kinetics

Skeletal muscle, 2022-06, Vol.12 (1), p.1-14, Article 14

2022

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Incubating isolated mouse EDL muscles with creatine improves force production and twitch kinetics in fatigue due to reduction in ionic strength

PloS one, 2011-08, Vol.6 (8), p.e22742-e22742

2011

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Assessment of Cell Viability in Electrically Excitable Muscle Cells Through Intact Twitch Stimulation

Methods in molecular biology (Clifton, N.J.), 2023, Vol.2644, p.177

2023

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Antioxidant therapy in a mouse model of Duchenne muscular dystrophy: some promising results but with a weighty caveat

The Journal of physiology, 2017-12, Vol.595 (23), p.7015-7015

Head, Stewart I.

2017

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RhoA/ROCK signaling and pleiotropic α1A-adrenergic receptor regulation of cardiac contractility

PloS one, 2014-06, Vol.9 (6), p.e99024-e99024

2014

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Branched fibres in old dystrophic mdx muscle are associated with mechanical weakening of the sarcolemma, abnormal Ca2+ transients and a breakdown of Ca2+ homeostasis during fatigue

Experimental physiology, 2010-05, Vol.95 (5), p.641-656

Head, Stewart I.

2010

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Loss of α-actinin-3 confers protection from eccentric contraction damage in fast-twitch EDL muscles from aged mdx dystrophic mice by reducing pathological fibre branching

Human molecular genetics, 2022-05, Vol.31 (9), p.1417-1429

2022

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Properties of extensor digitorum longus muscle and skinned fibers from adult and aged male and female Actn3 knockout mice

Muscle & nerve, 2011-01, Vol.43 (1), p.37-48

2011

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Calcium and strontium contractile activation properties of single skinned skeletal muscle fibres from elderly women 66–90 years of age

Journal of muscle research and cell motility, 2022-12, Vol.43 (4), p.173-183

2022

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Membrane Injury and Repair in the Muscular Dystrophies

The Neuroscientist, 2015-12, Vol.21 (6), p.653-668

2015

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Dystrophin-negative slow-twitch soleus muscles are not susceptible to eccentric contraction induced injury over the lifespan of the mdx mouse

American Journal of Physiology: Cell Physiology, 2021-10, Vol.321 (4), p.C704-C720

2021

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Sarcoplasmic reticulum calcium handling in unbranched, immediately post‐necrotic fast‐twitch mdx fibres is similar to wild‐type littermates

Experimental physiology, 2022-06, Vol.107 (6), p.601-614

2022

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Gadolinium reduces short-term stretch-induced muscle damage in isolated mdx mouse muscle fibres

The Journal of physiology, 2003-10, Vol.552 (2), p.449-458

2003

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Branched fibers from old fast-twitch dystrophic muscles are the sites of terminal damage in muscular dystrophy

American Journal of Physiology: Cell Physiology, 2018-06, Vol.314 (6), p.C662-C674

2018

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Single muscle fibre biomechanics and biomechatronics – The challenges, the pitfalls and the future

The international journal of biochemistry & cell biology, 2019-09, Vol.114, p.105563-105563, Article 105563

2019

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Analysis of the ACTN3 heterozygous genotype suggests that α-actinin-3 controls sarcomeric composition and muscle function in a dose-dependent fashion

Human molecular genetics, 2016-03, Vol.25 (5), p.866-877

2016

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Evidence for ACTN3 as a genetic modifier of Duchenne muscular dystrophy

Nature communications, 2017-01, Vol.8 (1), p.14143-14143, Article 14143

2017

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The Effect of ACTN3 Gene Doping on Skeletal Muscle Performance

American journal of human genetics, 2018-05, Vol.102 (5), p.845-857

2018

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An Actn3 knockout mouse provides mechanistic insights into the association between α-actinin-3 deficiency and human athletic performance

Human molecular genetics, 2008-04, Vol.17 (8), p.1076-1086

2008

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Old men still have the skeletal muscle contractile function to get up and go even after they have had their leg in a cast

The Journal of physiology, 2011-10, Vol.589 (19), p.4639-4639

Head, Stewart I.

2011

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Age- and gender-related changes in contractile properties of non-atrophied EDL muscle

Six weeks of N-acetylcysteine antioxidant in drinking water decreases pathological fiber branching in MDX mouse dystrophic fast-twitch skeletal muscle

Lifespan Analysis of Dystrophic mdx Fast-Twitch Muscle Morphology and Its Impact on Contractile Function

Altered Ca2+ kinetics associated with α-actinin-3 deficiency may explain positive selection for ACTN3 null allele in human evolution

Minocycline Treatment Reduces Mass and Force Output From Fast-Twitch Mouse Muscles and Inhibits Myosin Production in C2C12 Myotubes

Absence of the Z-disc protein α-actinin-3 impairs the mechanical stability of Actn3KO mouse fast-twitch muscle fibres without altering their contractile properties or twitch kinetics

Incubating isolated mouse EDL muscles with creatine improves force production and twitch kinetics in fatigue due to reduction in ionic strength

Assessment of Cell Viability in Electrically Excitable Muscle Cells Through Intact Twitch Stimulation

Antioxidant therapy in a mouse model of Duchenne muscular dystrophy: some promising results but with a weighty caveat

RhoA/ROCK signaling and pleiotropic α1A-adrenergic receptor regulation of cardiac contractility

Branched fibres in old dystrophic mdx muscle are associated with mechanical weakening of the sarcolemma, abnormal Ca2+ transients and a breakdown of Ca2+ homeostasis during fatigue

Loss of α-actinin-3 confers protection from eccentric contraction damage in fast-twitch EDL muscles from aged mdx dystrophic mice by reducing pathological fibre branching

Properties of extensor digitorum longus muscle and skinned fibers from adult and aged male and female Actn3 knockout mice

Calcium and strontium contractile activation properties of single skinned skeletal muscle fibres from elderly women 66–90 years of age

Membrane Injury and Repair in the Muscular Dystrophies

Dystrophin-negative slow-twitch soleus muscles are not susceptible to eccentric contraction induced injury over the lifespan of the mdx mouse

Sarcoplasmic reticulum calcium handling in unbranched, immediately post‐necrotic fast‐twitch mdx fibres is similar to wild‐type littermates

Gadolinium reduces short-term stretch-induced muscle damage in isolated mdx mouse muscle fibres

Branched fibers from old fast-twitch dystrophic muscles are the sites of terminal damage in muscular dystrophy

Single muscle fibre biomechanics and biomechatronics – The challenges, the pitfalls and the future

Analysis of the ACTN3 heterozygous genotype suggests that α-actinin-3 controls sarcomeric composition and muscle function in a dose-dependent fashion

Evidence for ACTN3 as a genetic modifier of Duchenne muscular dystrophy

The Effect of ACTN3 Gene Doping on Skeletal Muscle Performance

An Actn3 knockout mouse provides mechanistic insights into the association between α-actinin-3 deficiency and human athletic performance

Old men still have the skeletal muscle contractile function to get up and go even after they have had their leg in a cast

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