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Glucokinase activity in diabetes: too much of a good thing?

Trends in endocrinology and metabolism, 2023-02, Vol.34 (2), p.119-130

2023

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Diabetes causes marked inhibition of mitochondrial metabolism in pancreatic β-cells

Nature communications, 2019-06, Vol.10 (1), p.2474-2474, Article 2474

2019

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Beta Cell Hubs Dictate Pancreatic Islet Responses to Glucose

Cell metabolism, 2016-09, Vol.24 (3), p.389-401

2016

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Altered glycolysis triggers impaired mitochondrial metabolism and mTORC1 activation in diabetic β-cells

Nature communications, 2022-11, Vol.13 (1), p.6754-6754, Article 6754

2022

Volltextzugriff (PDF)

Editorial: Ion channels and transporters in diabetes and metabolic diseases

Frontiers in pharmacology, 2022-08, Vol.13, p.975812-975812

2022

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Binding of sulphonylureas to plasma proteins - A KATP channel perspective

PloS one, 2018-05, Vol.13 (5), p.e0197634

2018

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The pore-forming subunit MCU of the mitochondrial Ca2+ uniporter is required for normal glucose-stimulated insulin secretion in vitro and in vivo in mice

Diabetologia, 2020-07, Vol.63 (7), p.1368-1381

2020

Volltextzugriff (PDF)

Chronic d-serine supplementation impairs insulin secretion

Molecular metabolism (Germany), 2018-10, Vol.16, p.191-202

2018

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Phenotype of a transient neonatal diabetes point mutation (SUR1-R1183W) in mice

Wellcome open research, 2020, Vol.5, p.15-15

2020

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Dysregulation of Glucagon Secretion by Hyperglycemia-Induced Sodium-Dependent Reduction of ATP Production

Cell metabolism, 2019-02, Vol.29 (2), p.430-442.e4

2019

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Binding of sulphonylureas to plasma proteins - A K.sub.ATP channel perspective

PloS one, 2018-05, Vol.13 (5), p.e0197634

2018

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Local and regional control of calcium dynamics in the pancreatic islet

Diabetes, obesity & metabolism, 2017-09, Vol.19 (S1), p.30-41

2017

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The KCNJ11-E23K Gene Variant Hastens Diabetes Progression by Impairing Glucose-Induced Insulin Secretion

Diabetes (New York, N.Y.), 2021-05, Vol.70 (5), p.1145-1156

2021

Volltextzugriff (PDF)

Disallowance of Acot7 in [Beta]-Cells Is Required for Normal Glucose Tolerance and Insulin Secretion

Diabetes (New York, N.Y.), 2016-05, Vol.65 (5), p.1268

2016

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Disallowance of Acot7 in β-Cells Is Required for Normal Glucose Tolerance and Insulin Secretion

Diabetes (New York, N.Y.), 2016-05, Vol.65 (5), p.1268-1282

2016

Volltextzugriff (PDF)

343-LB: The Type 2 Diabetes-Associated Lipid Binding Protein STARD10 Controls Insulin Secretory Granule Biogenesis

Diabetes (New York, N.Y.), 2019-06, Vol.68 (Supplement_1)

2019

Volltextzugriff (PDF)

Chronic exposure to KATP channel openers results in attenuated glucose sensing in hypothalamic GT1-7 neurons

Neuropharmacology, 2016-12, Vol.111, p.212-222

2016

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Chronic exposure to K ATP channel openers results in attenuated glucose sensing in hypothalamic GT1-7 neurons

Neuropharmacology, 2016-12, Vol.111, p.212

2016

Volltextzugriff (PDF)

Phenotype of a transient neonatal diabetes point mutation (SUR1-R1183W) in mice

Wellcome open research, 2020, Vol.5, p.15

2020

Volltextzugriff (PDF)

The type 2 diabetes gene product STARD10 is a phosphoinositide-binding protein that controls insulin secretory granule biogenesis

Molecular metabolism (Germany), 2020-10, Vol.40, p.101015-101015, Article 101015

2020

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Continuous hypothalamic K(ATP) activation blunts glucose counter-regulation in vivo in rats and suppresses K(ATP) conductance in vitro

Diabetologia, 2013-09, Vol.56 (9), p.2088

2013

Volltextzugriff (PDF)

Continuous hypothalamic K^sub ATP^ activation blunts glucose counter-regulation in vivo in rats and suppresses K^sub ATP^ conductance in vitro

Diabetologia, 2013-09, Vol.56 (9), p.2088

2013

Volltextzugriff (PDF)

Investigation of katp channel function in response to metabolic and pharmacological manipulation, in the hypothalamic gt1-7 cell line

Haythorne, Elizabeth

2014

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Continuous hypothalamic KATP activation blunts glucose counter-regulation in vivo in rats and suppresses KATP conductance in vitro

Diabetologia, 2013-09, Vol.56 (9), p.2088-2092

2013

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The pore-forming subunit MCU of the mitochondrial Ca 2+ uniporter is required for normal glucose-stimulated insulin secretion in vitro and in vivo in mice

Diabetologia, 2020-07, Vol.63 (7), p.1368

2020

Volltextzugriff (PDF)

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Glucokinase activity in diabetes: too much of a good thing?

Diabetes causes marked inhibition of mitochondrial metabolism in pancreatic β-cells

Beta Cell Hubs Dictate Pancreatic Islet Responses to Glucose

Altered glycolysis triggers impaired mitochondrial metabolism and mTORC1 activation in diabetic β-cells

Editorial: Ion channels and transporters in diabetes and metabolic diseases

Binding of sulphonylureas to plasma proteins - A KATP channel perspective

The pore-forming subunit MCU of the mitochondrial Ca2+ uniporter is required for normal glucose-stimulated insulin secretion in vitro and in vivo in mice

Chronic d-serine supplementation impairs insulin secretion

Phenotype of a transient neonatal diabetes point mutation (SUR1-R1183W) in mice

Dysregulation of Glucagon Secretion by Hyperglycemia-Induced Sodium-Dependent Reduction of ATP Production

Binding of sulphonylureas to plasma proteins - A K.sub.ATP channel perspective

Local and regional control of calcium dynamics in the pancreatic islet

The KCNJ11-E23K Gene Variant Hastens Diabetes Progression by Impairing Glucose-Induced Insulin Secretion

Disallowance of Acot7 in [Beta]-Cells Is Required for Normal Glucose Tolerance and Insulin Secretion

Disallowance of Acot7 in β-Cells Is Required for Normal Glucose Tolerance and Insulin Secretion

343-LB: The Type 2 Diabetes-Associated Lipid Binding Protein STARD10 Controls Insulin Secretory Granule Biogenesis

Chronic exposure to KATP channel openers results in attenuated glucose sensing in hypothalamic GT1-7 neurons

Chronic exposure to K ATP channel openers results in attenuated glucose sensing in hypothalamic GT1-7 neurons

Phenotype of a transient neonatal diabetes point mutation (SUR1-R1183W) in mice

The type 2 diabetes gene product STARD10 is a phosphoinositide-binding protein that controls insulin secretory granule biogenesis

Continuous hypothalamic K(ATP) activation blunts glucose counter-regulation in vivo in rats and suppresses K(ATP) conductance in vitro

Continuous hypothalamic K^sub ATP^ activation blunts glucose counter-regulation in vivo in rats and suppresses K^sub ATP^ conductance in vitro

Investigation of katp channel function in response to metabolic and pharmacological manipulation, in the hypothalamic gt1-7 cell line

Continuous hypothalamic KATP activation blunts glucose counter-regulation in vivo in rats and suppresses KATP conductance in vitro

The pore-forming subunit MCU of the mitochondrial Ca 2+ uniporter is required for normal glucose-stimulated insulin secretion in vitro and in vivo in mice

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