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1,25-dihydroxyvitamin D3 reverses experimental autoimmune encephalomyelitis by inhibiting chemokine synthesis and monocyte trafficking

Journal of neuroscience research, 2007-08, Vol.85 (11), p.2480-2490

2007

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Vitamin D3 alters microglia immune activation by an IL-10 dependent SOCS3 mechanism

Journal of neuroimmunology, 2016-03, Vol.292, p.126-136

2016

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1,25-dihydroxycholecalciferol inhibits the progression of arthritis in murine models of human arthritis

The Journal of nutrition, 1998-01, Vol.128 (1), p.68-72

1998

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Vitamin D3 Confers Protection from Autoimmune Encephalomyelitis Only in Female Mice

The Journal of immunology (1950), 2005-09, Vol.175 (6), p.4119-4126

2005

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Vitamin D3-mediated resistance to a multiple sclerosis model disease depends on myeloid cell 1,25-dihydroxyvitamin D3 synthesis and correlates with increased CD4+ T cell CTLA-4 expression

Journal of neuroimmunology, 2020-01, Vol.338, p.577105-577105, Article 577105

2020

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Vitamin D 3 -mediated resistance to a multiple sclerosis model disease depends on myeloid cell 1,25-dihydroxyvitamin D 3 synthesis and correlates with increased CD4 + T cell CTLA-4 expression

Journal of neuroimmunology, 2020-01, Vol.338, p.577105

2020

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1,25-Dihydroxyvitamin D₃ acts directly on the T lymphocyte vitamin D receptor to inhibit experimental autoimmune encephalomyelitis

European journal of immunology, 2011-03, Vol.41 (3), p.822-832

2011

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Vitamin D and estrogen synergy in Vdr -expressing CD4+ T cells is essential to induce Helios+ FoxP3+ T cells and prevent autoimmune demyelinating disease

Journal of neuroimmunology, 2015-09, Vol.286, p.48-58

2015

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Estrogen Controls Vitamin D3-Mediated Resistance to Experimental Autoimmune Encephalomyelitis by Controlling Vitamin D3 Metabolism and Receptor Expression

The Journal of immunology (1950), 2009-09, Vol.183 (6), p.3672-3681

2009

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Systemic autoimmunity in BAFF-R-mutant A/WySnJ strain mice

European journal of immunology, 2008-02, Vol.38 (2), p.587-598

2008

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One calcitriol dose transiently increases Helios+ FoxP3+ T cells and ameliorates autoimmune demyelinating disease

Journal of neuroimmunology, 2013-10, Vol.263 (1), p.64-74

2013

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1,25-Dihydroxyvitamin D3 increases the methionine cycle, CD4+ T cell DNA methylation and Helios+Foxp3+ T regulatory cells to reverse autoimmune neurodegenerative disease

Journal of neuroimmunology, 2018-11, Vol.324, p.100-114

2018

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Vitamin D: a natural inhibitor of multiple sclerosis

Proceedings of the Nutrition Society, 2000-11, Vol.59 (4), p.531

Hayes, C E

2000

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A unifying multiple sclerosis etiology linking virus infection, sunlight, and vitamin D, through viral interleukin-10

Medical hypotheses, 2008, Vol.71 (1), p.85-90

2008

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Enforced bcl-xL Gene Expression Restored Splenic B Lymphocyte Development in BAFF-R Mutant Mice

The Journal of immunology (1950), 2003-05, Vol.170 (9), p.4593

2003

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The Ifng gene is essential for Vdr gene expression and vitamin D₃-mediated reduction of the pathogenic T cell burden in the central nervous system in experimental autoimmune encephalomyelitis, a multiple sclerosis model

The Journal of immunology (1950), 2012-09, Vol.189 (6), p.3188-3197

2012

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Retinoic Acid Enhances the T Helper 2 Cell Development That Is Essential for Robust Antibody Responses through Its Action on Antigen-Presenting Cells

The Journal of nutrition, 2002-12, Vol.132 (12), p.3736-3739

2002

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Gene expression analysis suggests that 1,25-dihydroxyvitamin D3 reverses experimental autoimmune encephalomyelitis by stimulating inflammatory cell apoptosis

Physiological genomics, 2004-07, Vol.18 (2), p.141-151

2004

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1,25-dihydroxyvitamin D3 treatment decreases macrophage accumulation in the CNS of mice with experimental autoimmune encephalomyelitis

Journal of neuroimmunology, 2000-03, Vol.103 (2), p.171-179

2000

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Altered BAFF-receptor signaling and additional modifier loci contribute to systemic autoimmunity in A/WySnJ mice

European journal of immunology, 2009-02, Vol.39 (2), p.589-599

2009

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Murine BAFF-receptor residues 168–175 are essential for optimal CD21/35 expression but dispensable for B cell survival

Molecular immunology, 2009-12, Vol.47 (2-3), p.590-599

2009

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1,25‐Dihydroxyvitamin D 3 acts directly on the T lymphocyte vitamin D receptor to inhibit experimental autoimmune encephalomyelitis

European journal of immunology, 2011-03, Vol.41 (3), p.822-832

2011

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Regulation of Constitutive p50/c-Rel Activity via Proteasome Inhibitor-Resistant IκBα Degradation in B Cells

Molecular and Cellular Biology, 2004-06, Vol.24 (11), p.4895-4908

2004

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Bcmd Decreases the Life Span of B-2 But Not B-1 Cells in A/WySnJ Mice

The Journal of immunology (1950), 1998-04, Vol.160 (8), p.3743-3747

1998

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Vitamin A deficiency results in a priming environment conducive for Th1 cell development

European journal of immunology, 1995-06, Vol.25 (6), p.1673-1679

1995

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1,25-dihydroxyvitamin D3 reverses experimental autoimmune encephalomyelitis by inhibiting chemokine synthesis and monocyte trafficking

Vitamin D3 alters microglia immune activation by an IL-10 dependent SOCS3 mechanism

1,25-dihydroxycholecalciferol inhibits the progression of arthritis in murine models of human arthritis

Vitamin D3 Confers Protection from Autoimmune Encephalomyelitis Only in Female Mice

Vitamin D3-mediated resistance to a multiple sclerosis model disease depends on myeloid cell 1,25-dihydroxyvitamin D3 synthesis and correlates with increased CD4+ T cell CTLA-4 expression

Vitamin D 3 -mediated resistance to a multiple sclerosis model disease depends on myeloid cell 1,25-dihydroxyvitamin D 3 synthesis and correlates with increased CD4 + T cell CTLA-4 expression

1,25-Dihydroxyvitamin D₃ acts directly on the T lymphocyte vitamin D receptor to inhibit experimental autoimmune encephalomyelitis

Vitamin D and estrogen synergy in Vdr -expressing CD4+ T cells is essential to induce Helios+ FoxP3+ T cells and prevent autoimmune demyelinating disease

Estrogen Controls Vitamin D3-Mediated Resistance to Experimental Autoimmune Encephalomyelitis by Controlling Vitamin D3 Metabolism and Receptor Expression

Systemic autoimmunity in BAFF-R-mutant A/WySnJ strain mice

One calcitriol dose transiently increases Helios+ FoxP3+ T cells and ameliorates autoimmune demyelinating disease

1,25-Dihydroxyvitamin D3 increases the methionine cycle, CD4+ T cell DNA methylation and Helios+Foxp3+ T regulatory cells to reverse autoimmune neurodegenerative disease

Vitamin D: a natural inhibitor of multiple sclerosis

A unifying multiple sclerosis etiology linking virus infection, sunlight, and vitamin D, through viral interleukin-10

Enforced bcl-xL Gene Expression Restored Splenic B Lymphocyte Development in BAFF-R Mutant Mice

The Ifng gene is essential for Vdr gene expression and vitamin D₃-mediated reduction of the pathogenic T cell burden in the central nervous system in experimental autoimmune encephalomyelitis, a multiple sclerosis model

Retinoic Acid Enhances the T Helper 2 Cell Development That Is Essential for Robust Antibody Responses through Its Action on Antigen-Presenting Cells

Gene expression analysis suggests that 1,25-dihydroxyvitamin D3 reverses experimental autoimmune encephalomyelitis by stimulating inflammatory cell apoptosis

1,25-dihydroxyvitamin D3 treatment decreases macrophage accumulation in the CNS of mice with experimental autoimmune encephalomyelitis

Altered BAFF-receptor signaling and additional modifier loci contribute to systemic autoimmunity in A/WySnJ mice

Murine BAFF-receptor residues 168–175 are essential for optimal CD21/35 expression but dispensable for B cell survival

1,25‐Dihydroxyvitamin D 3 acts directly on the T lymphocyte vitamin D receptor to inhibit experimental autoimmune encephalomyelitis

Regulation of Constitutive p50/c-Rel Activity via Proteasome Inhibitor-Resistant IκBα Degradation in B Cells

Bcmd Decreases the Life Span of B-2 But Not B-1 Cells in A/WySnJ Mice

Vitamin A deficiency results in a priming environment conducive for Th1 cell development

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