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Evaluation of the NAD+ biosynthetic pathway in ALS patients and effect of modulating NAD+ levels in hSOD1-linked ALS mouse models

Experimental neurology, 2020-05, Vol.327, p.113219-113219, Article 113219

2020

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FABP7 upregulation induces a neurotoxic phenotype in astrocytes

Glia, 2020-12, Vol.68 (12), p.2693-2704

2020

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Nicotinamide Adenine Dinucleotide Metabolism and Neurodegeneration

Antioxidants & redox signaling, 2018-06, Vol.28 (18), p.1652

2018

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Enhanced SIRT6 activity abrogates the neurotoxic phenotype of astrocytes expressing ALS‐linked mutant SOD1

The FASEB journal, 2019-06, Vol.33 (6), p.7084-7091

2019

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Role and Therapeutic Potential of Astrocytes in Amyotrophic Lateral Sclerosis

Current pharmaceutical design, 2017-01, Vol.23 (33), p.5010

2017

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NR1D1 downregulation in astrocytes induces a phenotype that is detrimental to cocultured motor neurons

The FASEB journal, 2022-04, Vol.36 (4), p.e22262-n/a

2022

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Enhancing NAD+ Salvage Pathway Reverts the Toxicity of Primary Astrocytes Expressing Amyotrophic Lateral Sclerosis-linked Mutant Superoxide Dismutase 1 (SOD1)

The Journal of biological chemistry, 2016-05, Vol.291 (20), p.10836-10846

2016

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Altered expression of clock and clock‐controlled genes in a hSOD1‐linked amyotrophic lateral sclerosis mouse model

The FASEB journal, 2021-02, Vol.35 (2), p.e21343-n/a

2021

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When aging switches on Alzheimer’s

Aging (Albany, NY.), 2021-05, Vol.13 (10), p.13376-13377

2021

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Changes in Protein Expression and Lysine Acetylation Induced by Decreased Glutathione Levels in Astrocytes

Molecular & cellular proteomics, 2016-02, Vol.15 (2), p.493-505

2016

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Restoration of Kv7 Channel-Mediated Inhibition Reduces Cued-Reinstatement of Cocaine Seeking

The Journal of neuroscience, 2018-04, Vol.38 (17), p.4212-4229

2018

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GTP energy dependence of endocytosis and autophagy in the aging brain and Alzheimer’s disease

GeroScience, 2023-04, Vol.45 (2), p.757-780

2023

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Decreased glutathione levels cause overt motor neuron degeneration in hSOD1WT over-expressing mice

Experimental neurology, 2018-04, Vol.302, p.129-135

2018

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Nitration and Glycation Turn Mature NGF into a Toxic Factor for Motor Neurons: A Role for p75 NTR and RAGE Signaling in ALS

Antioxidants & redox signaling, 2018-06, Vol.28 (18), p.1587

2018

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Regulatory Issues and Challenges to Artificial Intelligence Adoption

The Radiologic clinics of North America, 2021-11, Vol.59 (6), p.1075-1083

2021

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Dopamine terminals from the ventral tegmental area gate intrinsic inhibition in the prefrontal cortex

Physiological reports, 2017-03, Vol.5 (6), p.np-n/a

2017

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FABP7 up-regulation induces a neurotoxic phenotype in astrocytes

Glia, 2020-07, Vol.68 (12), p.2693-2704

2020

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Decreased glutathione levels cause overt motor neuron degeneration in hSOD1WT overexpressing mice

Experimental neurology, 2018-01, Vol.302, p.129-135

2018

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Decreased glutathione levels cause overt motor neuron degeneration in hSOD1 WT over-expressing mice

Experimental neurology, 2018-04, Vol.302, p.129

2018

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Mountain pine beetle impacts on tree resources and growth release in lodgepole pine dominated subalpine forest

Brayden, Benjamin Harlan

2013

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ProQuest

Formation and Properties of Some Uretediones

Freyermuth, Harlan Benjamin

1942

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ProQuest

The New Era of Informed Consent: Getting to a Reasonable-Patient Standard Through Shared Decision Making

JAMA : the journal of the American Medical Association, 2016-05, Vol.315 (19), p.2063

2016

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Micro-scale urban surface temperatures are related to land-cover features and residential heat related health impacts in Phoenix, AZ USA

Landscape ecology, 2016-05, Vol.31 (4), p.745-760

2016

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Prime Time for Shared Decision Making

JAMA : the journal of the American Medical Association, 2017-04, Vol.317 (13), p.1309

2017

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Evaluation of the NAD+ biosynthetic pathway in ALS patients and effect of modulating NAD+ levels in hSOD1-linked ALS mouse models

FABP7 upregulation induces a neurotoxic phenotype in astrocytes

Nicotinamide Adenine Dinucleotide Metabolism and Neurodegeneration

Enhanced SIRT6 activity abrogates the neurotoxic phenotype of astrocytes expressing ALS‐linked mutant SOD1

Role and Therapeutic Potential of Astrocytes in Amyotrophic Lateral Sclerosis

NR1D1 downregulation in astrocytes induces a phenotype that is detrimental to cocultured motor neurons

Enhancing NAD+ Salvage Pathway Reverts the Toxicity of Primary Astrocytes Expressing Amyotrophic Lateral Sclerosis-linked Mutant Superoxide Dismutase 1 (SOD1)

Altered expression of clock and clock‐controlled genes in a hSOD1‐linked amyotrophic lateral sclerosis mouse model

When aging switches on Alzheimer’s

Changes in Protein Expression and Lysine Acetylation Induced by Decreased Glutathione Levels in Astrocytes

Restoration of Kv7 Channel-Mediated Inhibition Reduces Cued-Reinstatement of Cocaine Seeking

GTP energy dependence of endocytosis and autophagy in the aging brain and Alzheimer’s disease

Decreased glutathione levels cause overt motor neuron degeneration in hSOD1WT over-expressing mice

Nitration and Glycation Turn Mature NGF into a Toxic Factor for Motor Neurons: A Role for p75 NTR and RAGE Signaling in ALS

Regulatory Issues and Challenges to Artificial Intelligence Adoption

Dopamine terminals from the ventral tegmental area gate intrinsic inhibition in the prefrontal cortex

FABP7 up-regulation induces a neurotoxic phenotype in astrocytes

Decreased glutathione levels cause overt motor neuron degeneration in hSOD1WT overexpressing mice

Decreased glutathione levels cause overt motor neuron degeneration in hSOD1 WT over-expressing mice

Mountain pine beetle impacts on tree resources and growth release in lodgepole pine dominated subalpine forest

Formation and Properties of Some Uretediones

The New Era of Informed Consent: Getting to a Reasonable-Patient Standard Through Shared Decision Making

Micro-scale urban surface temperatures are related to land-cover features and residential heat related health impacts in Phoenix, AZ USA

Prime Time for Shared Decision Making

Study protocol for the Innovative Support for Patients with SARS-COV-2 Infections Registry (INSPIRE): A longitudinal study of the medium and long-term sequelae of SARS-CoV-2 infection

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