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Loss of MTX2 causes mandibuloacral dysplasia and links mitochondrial dysfunction to altered nuclear morphology

Nature communications, 2020-09, Vol.11 (1), p.4589-4589, Article 4589

2020

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MG132‐induced progerin clearance is mediated by autophagy activation and splicing regulation

EMBO molecular medicine, 2017-09, Vol.9 (9), p.1294-1313

2017

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Vulnerability of progeroid smooth muscle cells to biomechanical forces is mediated by MMP13

Nature communications, 2020-08, Vol.11 (1), p.4110-4110, Article 4110

2020

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Inhibition of poly(ADP-Ribosyl)ation reduced vascular smooth muscle cells loss and improves aortic disease in a mouse model of human accelerated aging syndrome

Cell death & disease, 2024-10, Vol.15 (10), p.723-14, Article 723

2024

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Low lamin A expression in lung adenocarcinoma cells from pleural effusions is a pejorative factor associated with high number of metastatic sites and poor Performance status

PloS one, 2017-08, Vol.12 (8), p.e0183136-e0183136

2017

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CD146 and its soluble form regulate monocyte transendothelial migration

Arteriosclerosis, thrombosis, and vascular biology, 2009-05, Vol.29 (5), p.746-753

2009

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Soluble CD146 displays angiogenic properties and promotes neovascularization in experimental hind-limb ischemia

Blood, 2010-05, Vol.115 (18), p.3843-3851

2010

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MG132 Induces Progerin Clearance and Improves Disease Phenotypes in HGPS-like Patients' Cells

Cells (Basel, Switzerland), 2022-02, Vol.11 (4), p.610

2022

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CD146 Short Isoform Increases the Proangiogenic Potential of Endothelial Progenitor Cells In Vitro and In Vivo

Circulation research, 2010-07, Vol.107 (1), p.66-75

2010

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Efficient progerin clearance through autophagy induction and SRSF-1 downregulation in Hutchinson-Gilford Progeria Syndrome

Orphanet journal of rare diseases, 2015-11, Vol.10 (S2), p.O9-O9, Article O9

Harhouri, Karim

2015

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Antisense-Based Progerin Downregulation in HGPS-Like Patients' Cells

Cells (Basel, Switzerland), 2016-07, Vol.5 (3), p.31

2016

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Level of Adenosine Diphosphate Receptor P2Y12 Blockade During Percutaneous Coronary Intervention Predicts the Extent of Endothelial Injury, Assessed by Circulating Endothelial Cell Measurement

Journal of the American College of Cardiology, 2010-09, Vol.56 (13), p.1024-1031

2010

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An overview of treatment strategies for Hutchinson-Gilford Progeria syndrome

Nucleus (Austin, Tex.), 2018-01, Vol.9 (1), p.265-276

2018

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High-sensitivity flow cytometry provides access to standardized measurement of small-size microparticles--brief report

Arteriosclerosis, thrombosis, and vascular biology, 2012-04, Vol.32 (4), p.1054-1058

2012

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Nuclear matrix, nuclear envelope and premature aging syndromes in a translational research perspective

Seminars in cell & developmental biology, 2014-05, Vol.29, p.125-147

2014

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VULNERABILITY OF PROGEROID SMOOTH MUSCLE CELLS TO ARTERIAL SHEAR STRESS IS MEDIATED BY MMP13

Nature communications, 2022, Vol.28 (1)

2022

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MicroRNAs in hereditary and sporadic premature aging syndromes and other laminopathies

Aging cell, 2018-08, Vol.17 (4), p.e12766-n/a

2018

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Soluble CD146 boosts therapeutic effect of endothelial progenitors through proteolytic processing of short CD146 isoform

Cardiovascular research, 2016-08, Vol.111 (3), p.240-251

2016

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Soluble Melanoma Cell Adhesion Molecule (sMCAM/sCD146) Promotes Angiogenic Effects on Endothelial Progenitor Cells through Angiomotin

The Journal of biological chemistry, 2013-03, Vol.288 (13), p.8991-9000

2013

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MG 132‐induced progerin clearance is mediated by autophagy activation and splicing regulation

EMBO molecular medicine, 2017-09, Vol.9 (9), p.1294-1313

2017

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C0412 soluble CD146: A new angiogenic factor involved in physiopathology

Thrombosis research, 2012-10, Vol.130, p.S108-S108

2012

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Soluble CD146 boosts therapeutic effect of endothelial progenitors through proteolytic processing of short CD146 isoform

Cardiovascular research, 2016-07, Vol.111 (3), p.240-251

2016

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WITHDRAWN: Nuclear matrix, nuclear envelope and premature aging syndromes in a translational research perspective

Seminars in cell & developmental biology, 2014-03

2014

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Soluble CD146: a new angiogenic molecule involved in physiopathology

Annales de pathologie, 2010-11, Vol.30 (5 Suppl 1), p.37-39

2010

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Loss of MTX2 causes mandibuloacral dysplasia and links mitochondrial dysfunction to altered nuclear morphology

MG132‐induced progerin clearance is mediated by autophagy activation and splicing regulation

Vulnerability of progeroid smooth muscle cells to biomechanical forces is mediated by MMP13

Inhibition of poly(ADP-Ribosyl)ation reduced vascular smooth muscle cells loss and improves aortic disease in a mouse model of human accelerated aging syndrome

Low lamin A expression in lung adenocarcinoma cells from pleural effusions is a pejorative factor associated with high number of metastatic sites and poor Performance status

CD146 and its soluble form regulate monocyte transendothelial migration

Author Correction: Loss of MTX2 causes mandibuloacral dysplasia and links mitochondrial dysfunction to altered nuclear morphology

Soluble CD146 displays angiogenic properties and promotes neovascularization in experimental hind-limb ischemia

MG132 Induces Progerin Clearance and Improves Disease Phenotypes in HGPS-like Patients' Cells

CD146 Short Isoform Increases the Proangiogenic Potential of Endothelial Progenitor Cells In Vitro and In Vivo

Efficient progerin clearance through autophagy induction and SRSF-1 downregulation in Hutchinson-Gilford Progeria Syndrome

Antisense-Based Progerin Downregulation in HGPS-Like Patients' Cells

Level of Adenosine Diphosphate Receptor P2Y12 Blockade During Percutaneous Coronary Intervention Predicts the Extent of Endothelial Injury, Assessed by Circulating Endothelial Cell Measurement

An overview of treatment strategies for Hutchinson-Gilford Progeria syndrome

High-sensitivity flow cytometry provides access to standardized measurement of small-size microparticles--brief report

Nuclear matrix, nuclear envelope and premature aging syndromes in a translational research perspective

VULNERABILITY OF PROGEROID SMOOTH MUSCLE CELLS TO ARTERIAL SHEAR STRESS IS MEDIATED BY MMP13

MicroRNAs in hereditary and sporadic premature aging syndromes and other laminopathies

Soluble CD146 boosts therapeutic effect of endothelial progenitors through proteolytic processing of short CD146 isoform

Soluble Melanoma Cell Adhesion Molecule (sMCAM/sCD146) Promotes Angiogenic Effects on Endothelial Progenitor Cells through Angiomotin

MG 132‐induced progerin clearance is mediated by autophagy activation and splicing regulation

C0412 soluble CD146: A new angiogenic factor involved in physiopathology

Soluble CD146 boosts therapeutic effect of endothelial progenitors through proteolytic processing of short CD146 isoform

WITHDRAWN: Nuclear matrix, nuclear envelope and premature aging syndromes in a translational research perspective

Soluble CD146: a new angiogenic molecule involved in physiopathology

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