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Developmental origins of health and disease: current knowledge and potential mechanisms

Nutrition reviews, 2017-12, Vol.75 (12), p.951-970

2017

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Developmental origins of metabolic diseases

Physiological reviews, 2021-07, Vol.101 (3), p.739-795

2021

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Metabolic Consequences of Gestational Cannabinoid Exposure

International journal of molecular sciences, 2021-09, Vol.22 (17), p.9528

2021

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The Role of Cellular Stress in Intrauterine Growth Restriction and Postnatal Dysmetabolism

International journal of molecular sciences, 2021-06, Vol.22 (13), p.6986

2021

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Δ9-tetrahydrocannabinol exposure during rat pregnancy leads to symmetrical fetal growth restriction and labyrinth-specific vascular defects in the placenta

Scientific reports, 2020-01, Vol.10 (1), p.544, Article 544

2020

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Prenatal Cannabinoid Exposure: Emerging Evidence of Physiological and Neuropsychiatric Abnormalities

Frontiers in psychiatry, 2021-01, Vol.11, p.624275-624275

2021

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Δ9-Tetrahydrocannabinol leads to endoplasmic reticulum stress and mitochondrial dysfunction in human BeWo trophoblasts

Reproductive toxicology (Elmsford, N.Y.), 2019-08, Vol.87, p.21-31

2019

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In Utero Exposure to Δ9-Tetrahydrocannabinol Leads to Postnatal Catch-Up Growth and Dysmetabolism in the Adult Rat Liver

International journal of molecular sciences, 2021-07, Vol.22 (14), p.7502

2021

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Prenatal tetrahydrocannabinol and cannabidiol exposure produce sex-specific pathophysiological phenotypes in the adolescent prefrontal cortex and hippocampus

Neurobiology of disease, 2024-09, Vol.199, p.106588, Article 106588

2024

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Nicotine Directly Induces Endoplasmic Reticulum Stress Response in Rat Placental Trophoblast Giant Cells

Toxicological sciences, 2016-05, Vol.151 (1), p.23-34

2016

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Progesterone Receptor Inhibits Proliferation of Human Breast Cancer Cells via Induction of MAPK Phosphatase 1 (MKP-1/DUSP1)

The Journal of biological chemistry, 2011-12, Vol.286 (50), p.43091-43102

2011

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Inflammatory Status Influences Aromatase and Steroid Receptor Expression in Endometriosis

Endocrinology (Philadelphia), 2008-03, Vol.149 (3), p.1190-1204

2008

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Maternal protein restriction during perinatal life affects lung mechanics and the surfactant system during early postnatal life in female rats

PloS one, 2019-04, Vol.14 (4), p.e0215611-e0215611

2019

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G protein-coupled KISS1 receptor is overexpressed in triple negative breast cancer and promotes drug resistance

Scientific reports, 2017-04, Vol.7 (1), p.46525-46525, Article 46525

2017

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Prenatal Exposure to Delta-9-tetrahydrocannabinol (THC) Alters the Expression of miR-122-5p and Its Target Igf1r in the Adult Rat Ovary

International journal of molecular sciences, 2022-07, Vol.23 (14), p.8000

2022

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Exposure to the RXR Agonist SR11237 in Early Life Causes Disturbed Skeletal Morphogenesis in a Rat Model

International journal of molecular sciences, 2019-10, Vol.20 (20), p.5198

2019

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Higher Hepatic miR-29 Expression in Undernourished Male Rats During the Postnatal Period Targets the Long-Term Repression of IGF-1

Endocrinology (Philadelphia), 2015-09, Vol.156 (9), p.3069-3076

2015

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Maternal nicotine exposure leads to impaired disulfide bond formation and augmented endoplasmic reticulum stress in the rat placenta

PloS one, 2015-03, Vol.10 (3), p.e0122295-e0122295

2015

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Maternal protein restriction leads to enhanced hepatic gluconeogenic gene expression in adult male rat offspring due to impaired expression of the liver X receptor

Journal of endocrinology, 2013-07, Vol.218 (1), p.85-97

2013

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Maternal nicotine exposure leads to decreased cardiac protein disulfide isomerase and impaired mitochondrial function in male rat offspring

Journal of applied toxicology, 2017-12, Vol.37 (12), p.1517-1526

2017

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Fetal and neonatal exposure to nicotine leads to augmented hepatic and circulating triglycerides in adult male offspring due to increased expression of fatty acid synthase

Toxicology and applied pharmacology, 2014-02, Vol.275 (1), p.1-11

2014

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Exposure to Δ9-tetrahydrocannabinol during rat pregnancy leads to impaired cardiac dysfunction in postnatal life

Pediatric research, 2021-09, Vol.90 (3), p.532-539

2021

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Progesterone Receptor Inhibits Aromatase and Inflammatory Response Pathways in Breast Cancer Cells via Ligand-Dependent and Ligand-Independent Mechanisms

Molecular endocrinology (Baltimore, Md.), 2008-08, Vol.22 (8), p.1812-1824

2008

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Gonadotropin releasing hormone antagonists suppress aromatase and anti-Müllerian hormone expression in human granulosa cells

Fertility and sterility, 2010-10, Vol.94 (5), p.1832-1839

2010

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Maternal Protein Restriction Elevates Cholesterol in Adult Rat Offspring Due to Repressive Changes in Histone Modifications at the Cholesterol 7α-Hydroxylase Promoter

Molecular endocrinology (Baltimore, Md.), 2011-05, Vol.25 (5), p.785-798

2011

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Developmental origins of health and disease: current knowledge and potential mechanisms

Developmental origins of metabolic diseases

Metabolic Consequences of Gestational Cannabinoid Exposure

The Role of Cellular Stress in Intrauterine Growth Restriction and Postnatal Dysmetabolism

Δ9-tetrahydrocannabinol exposure during rat pregnancy leads to symmetrical fetal growth restriction and labyrinth-specific vascular defects in the placenta

Prenatal Cannabinoid Exposure: Emerging Evidence of Physiological and Neuropsychiatric Abnormalities

Δ9-Tetrahydrocannabinol leads to endoplasmic reticulum stress and mitochondrial dysfunction in human BeWo trophoblasts

In Utero Exposure to Δ9-Tetrahydrocannabinol Leads to Postnatal Catch-Up Growth and Dysmetabolism in the Adult Rat Liver

Prenatal tetrahydrocannabinol and cannabidiol exposure produce sex-specific pathophysiological phenotypes in the adolescent prefrontal cortex and hippocampus

Nicotine Directly Induces Endoplasmic Reticulum Stress Response in Rat Placental Trophoblast Giant Cells

Progesterone Receptor Inhibits Proliferation of Human Breast Cancer Cells via Induction of MAPK Phosphatase 1 (MKP-1/DUSP1)

Inflammatory Status Influences Aromatase and Steroid Receptor Expression in Endometriosis

Maternal protein restriction during perinatal life affects lung mechanics and the surfactant system during early postnatal life in female rats

G protein-coupled KISS1 receptor is overexpressed in triple negative breast cancer and promotes drug resistance

Prenatal Exposure to Delta-9-tetrahydrocannabinol (THC) Alters the Expression of miR-122-5p and Its Target Igf1r in the Adult Rat Ovary

Exposure to the RXR Agonist SR11237 in Early Life Causes Disturbed Skeletal Morphogenesis in a Rat Model

Higher Hepatic miR-29 Expression in Undernourished Male Rats During the Postnatal Period Targets the Long-Term Repression of IGF-1

Maternal nicotine exposure leads to impaired disulfide bond formation and augmented endoplasmic reticulum stress in the rat placenta

Maternal protein restriction leads to enhanced hepatic gluconeogenic gene expression in adult male rat offspring due to impaired expression of the liver X receptor

Maternal nicotine exposure leads to decreased cardiac protein disulfide isomerase and impaired mitochondrial function in male rat offspring

Fetal and neonatal exposure to nicotine leads to augmented hepatic and circulating triglycerides in adult male offspring due to increased expression of fatty acid synthase

Exposure to Δ9-tetrahydrocannabinol during rat pregnancy leads to impaired cardiac dysfunction in postnatal life

Progesterone Receptor Inhibits Aromatase and Inflammatory Response Pathways in Breast Cancer Cells via Ligand-Dependent and Ligand-Independent Mechanisms

Gonadotropin releasing hormone antagonists suppress aromatase and anti-Müllerian hormone expression in human granulosa cells

Maternal Protein Restriction Elevates Cholesterol in Adult Rat Offspring Due to Repressive Changes in Histone Modifications at the Cholesterol 7α-Hydroxylase Promoter

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