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Influence of GluN2 subunit identity on NMDA receptor function

Neuropharmacology, 2013-11, Vol.74, p.4-17

2013

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In developing hippocampal neurons, NR2B-containing N -methyl- d -aspartate receptors (NMDARs) can mediate signaling to neuronal survival and synaptic potentiation, as well as neuronal death

Neuroscience, 2009-01, Vol.158 (1), p.334-343

2009

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TCN 201 selectively blocks GluN2A-containing NMDARs in a GluN1 co-agonist dependent but non-competitive manner

Neuropharmacology, 2012-09, Vol.63 (3), p.441-449

2012

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Extracellular Calcium Regulates Postsynaptic Efficacy through Group 1 Metabotropic Glutamate Receptors

The Journal of neuroscience, 2006-06, Vol.26 (23), p.6337-6345

2006

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Direct pharmacological monitoring of the developmental switch in NMDA receptor subunit composition using TCN 213, a GluN2A‐selective, glycine‐dependent antagonist

British journal of pharmacology, 2012-06, Vol.166 (3), p.924-937

2012

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Nuclear calcium signaling controls CREB-mediated gene expression triggered by synaptic activity

Nature neuroscience, 2001-03, Vol.4 (3), p.261-267

2001

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Retinoid-independent motor neurogenesis from human embryonic stem cells reveals a medial columnar ground state

Nature communications, 2011-03, Vol.2 (1), p.214-214, Article 214

2011

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A calcium microdomain near NMDA receptors: on switch for ERK-dependent synapse-to-nucleus communication

Nature neuroscience, 2001-06, Vol.4 (6), p.565-566

2001

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CBP: A Signal-Regulated Transcriptional Coactivator Controlled by Nuclear Calcium and CaM Kinase IV

Science (American Association for the Advancement of Science), 1998-09, Vol.281 (5382), p.1505-1509

1998

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Distinct functions of nuclear and cytoplasmic calcium in the control of gene expression

Nature (London), 1997-01, Vol.385 (6613), p.260-265

1997

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Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N -methyl- d -aspartate receptors

Neuroscience, 2009-01, Vol.158 (1), p.344-352

2009

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Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways

Nature neuroscience, 2002-05, Vol.5 (5), p.405-414

2002

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Pro-survival signalling from the NMDA receptor

Biochemical Society transactions, 2006-11, Vol.34 (Pt 5), p.936-938

Hardingham, G E

2006

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SynGAP isoforms exert opposing effects on synaptic strength

Nature communications, 2012-06, Vol.3 (1), p.900-900, Article 900

2012

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Human embryonic stem cell derived astrocytes mediate non-cell-autonomous neuroprotection through endogenous and drug-induced mechanisms

Cell death and differentiation, 2012-05, Vol.19 (5), p.779-787

2012

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NMDA receptor expression and activity in osteoarthritic human articular chondrocytes

Osteoarthritis and cartilage, 2008-12, Vol.16 (12), p.1576-1584

2008

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Control of Recruitment and Transcription-Activating Function of CBP Determines Gene Regulation by NMDA Receptors and L-Type Calcium Channels

Neuron (Cambridge, Mass.), 1999-04, Vol.22 (4), p.789-798

1999

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The dichotomy of NMDA receptor signaling

The Neuroscientist (Baltimore, Md.), 2007-12, Vol.13 (6), p.572-579

2007

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Preconditioning Doses of NMDA Promote Neuroprotection by Enhancing Neuronal Excitability

The Journal of neuroscience, 2006-04, Vol.26 (17), p.4509-4518

2006

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The Yin and Yang of NMDA receptor signalling

Trends in neurosciences (Regular ed.), 2003-02, Vol.26 (2), p.81-89

2003

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Nuclear Ca2+ and the cAMP Response Element-Binding Protein Family Mediate a Late Phase of Activity-Dependent Neuroprotection

The Journal of neuroscience, 2005-04, Vol.25 (17), p.4279-4287

2005

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Mechanisms controlling gene expression by nuclear calcium signals

Cell Calcium, 1998-02, Vol.23 (2), p.131-134

1998

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c-Jun functions as a calcium-regulated transcriptional activator in the absence of JNK/SAPK1 activation

The EMBO journal, 1999-03, Vol.18 (5), p.1335-1344

1999

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Nuclear calcium: a key regulator of gene expression

Biometals, 1998-12, Vol.11 (4), p.345-358

1998

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Gene Regulation by Nuclear and Cytoplasmic Calcium Signals

Biochemical and biophysical research communications, 1997-07, Vol.236 (3), p.541-543

1997

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Influence of GluN2 subunit identity on NMDA receptor function

In developing hippocampal neurons, NR2B-containing N -methyl- d -aspartate receptors (NMDARs) can mediate signaling to neuronal survival and synaptic potentiation, as well as neuronal death

TCN 201 selectively blocks GluN2A-containing NMDARs in a GluN1 co-agonist dependent but non-competitive manner

Extracellular Calcium Regulates Postsynaptic Efficacy through Group 1 Metabotropic Glutamate Receptors

Direct pharmacological monitoring of the developmental switch in NMDA receptor subunit composition using TCN 213, a GluN2A‐selective, glycine‐dependent antagonist

Nuclear calcium signaling controls CREB-mediated gene expression triggered by synaptic activity

Retinoid-independent motor neurogenesis from human embryonic stem cells reveals a medial columnar ground state

A calcium microdomain near NMDA receptors: on switch for ERK-dependent synapse-to-nucleus communication

CBP: A Signal-Regulated Transcriptional Coactivator Controlled by Nuclear Calcium and CaM Kinase IV

Distinct functions of nuclear and cytoplasmic calcium in the control of gene expression

Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N -methyl- d -aspartate receptors

Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways

Pro-survival signalling from the NMDA receptor

SynGAP isoforms exert opposing effects on synaptic strength

Human embryonic stem cell derived astrocytes mediate non-cell-autonomous neuroprotection through endogenous and drug-induced mechanisms

NMDA receptor expression and activity in osteoarthritic human articular chondrocytes

Control of Recruitment and Transcription-Activating Function of CBP Determines Gene Regulation by NMDA Receptors and L-Type Calcium Channels

The dichotomy of NMDA receptor signaling

Preconditioning Doses of NMDA Promote Neuroprotection by Enhancing Neuronal Excitability

The Yin and Yang of NMDA receptor signalling

Nuclear Ca2+ and the cAMP Response Element-Binding Protein Family Mediate a Late Phase of Activity-Dependent Neuroprotection

Mechanisms controlling gene expression by nuclear calcium signals

c-Jun functions as a calcium-regulated transcriptional activator in the absence of JNK/SAPK1 activation

Nuclear calcium: a key regulator of gene expression

Gene Regulation by Nuclear and Cytoplasmic Calcium Signals

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