UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Microglia in Alzheimer's disease

The Journal of cell biology, 2018-02, Vol.217 (2), p.459-472

2018

Volltextzugriff (PDF)

Diverse modes of NMDA receptor positive allosteric modulation: Mechanisms and consequences

Neuropharmacology, 2017-01, Vol.112 (Pt A), p.34-45

2017

Volltextzugriff (PDF)

Changes in the Synaptic Proteome in Tauopathy and Rescue of Tau-Induced Synapse Loss by C1q Antibodies

Neuron (Cambridge, Mass.), 2018-12, Vol.100 (6), p.1322-1336.e7

2018

Volltextzugriff (PDF)

Complement C3 Is Activated in Human AD Brain and Is Required for Neurodegeneration in Mouse Models of Amyloidosis and Tauopathy

Cell reports (Cambridge), 2019-08, Vol.28 (8), p.2111-2123.e6

2019

Volltextzugriff (PDF)

Higher ambient synaptic glutamate at inhibitory versus excitatory neurons differentially impacts NMDA receptor activity

Nature communications, 2018-10, Vol.9 (1), p.4000-13, Article 4000

2018

Volltextzugriff (PDF)

Positive Allosteric Modulators of GluN2A-Containing NMDARs with Distinct Modes of Action and Impacts on Circuit Function

Neuron (Cambridge, Mass.), 2016-03, Vol.89 (5), p.983-999

2016

Volltextzugriff (PDF)

SAHA enhances synaptic function and plasticity in vitro but has limited brain availability in vivo and does not impact cognition

PloS one, 2013-07, Vol.8 (7), p.e69964

2013

Volltextzugriff (PDF)

Complement C1q-dependent excitatory and inhibitory synapse elimination by astrocytes and microglia in Alzheimer's disease mouse models

Nature aging, 2022-09, Vol.2 (9), p.837-850

2022

Volltextzugriff (PDF)

Identifying faulty brain circuits

eLife, 2017-04, Vol.6

Hanson, Jesse E

2017

Volltextzugriff (PDF)

Targeting synapse function and loss for treatment of neurodegenerative diseases

Nature reviews. Drug discovery, 2024-01, Vol.23 (1), p.23-42

2024

Volltextzugriff (PDF)

GluN2A NMDA Receptor Enhancement Improves Brain Oscillations, Synchrony, and Cognitive Functions in Dravet Syndrome and Alzheimer’s Disease Models

Cell reports (Cambridge), 2020-01, Vol.30 (2), p.381-396.e4

2020

Volltextzugriff (PDF)

Protocol for detection of glial complement expression in relation to amyloid plaques in mouse brain with combined FISH and IHC

STAR protocols, 2024-12, Vol.5 (4), p.103388, Article 103388

2024

Volltextzugriff (PDF)

Author Correction: Higher ambient synaptic glutamate at inhibitory versus excitatory neurons differentially impacts NMDA receptor activity

Nature communications, 2018-11, Vol.9 (1), p.4887-1, Article 4887

2018

Volltextzugriff (PDF)

Dual leucine zipper kinase is required for excitotoxicity-induced neuronal degeneration

The Journal of experimental medicine, 2013-11, Vol.210 (12), p.2553-2567

2013

Volltextzugriff (PDF)

TPL2 kinase activity regulates microglial inflammatory responses and promotes neurodegeneration in tauopathy mice

eLife, 2023-08, Vol.12

2023

Volltextzugriff (PDF)

Altered GluN2B NMDA receptor function and synaptic plasticity during early pathology in the PS2APP mouse model of Alzheimer's disease

Neurobiology of disease, 2015-02, Vol.74, p.254-262

2015

Volltextzugriff (PDF)

β-Amyloid Inhibits E-S Potentiation through Suppression of Cannabinoid Receptor 1-Dependent Synaptic Disinhibition

Neuron (Cambridge, Mass.), 2014-06, Vol.82 (6), p.1334-1345

2014

Volltextzugriff (PDF)

Computationally Discovered Potentiating Role of Glycans on NMDA Receptors

Scientific reports, 2017-04, Vol.7 (1), p.44578-44578, Article 44578

2017

Volltextzugriff (PDF)

GluN2B Antagonism Affects Interneurons and Leads to Immediate and Persistent Changes in Synaptic Plasticity, Oscillations, and Behavior

Neuropsychopharmacology (New York, N.Y.), 2013-06, Vol.38 (7), p.1221-1233

2013

Volltextzugriff (PDF)

Histone deacetylase 2 cell autonomously suppresses excitatory and enhances inhibitory synaptic function in CA1 pyramidal neurons

The Journal of neuroscience, 2013-04, Vol.33 (14), p.5924-5929

2013

Volltextzugriff (PDF)

The functional nature of synaptic circuitry is altered in area CA3 of the hippocampus in a mouse model of Down's syndrome

The Journal of physiology, 2007-02, Vol.579 (1), p.53-67

2007

Volltextzugriff (PDF)

Therapeutic potential of N-methyl-D-aspartate receptor modulators in psychiatry

Neuropsychopharmacology (New York, N.Y.), 2024-01, Vol.49 (1), p.51-66

2024

Volltextzugriff (PDF)

Dendritic sodium channels regulate network integration in globus pallidus neurons: a modeling study

The Journal of neuroscience, 2010-11, Vol.30 (45), p.15146-15159

2010

Volltextzugriff (PDF)

Cognitive Deficits, Changes in Synaptic Function, and Brain Pathology in a Mouse Model of Normal Aging(1,2,3)

eNeuro, 2015-09, Vol.2 (5), p.ENEURO.0047-15.2015

2015

Volltextzugriff (PDF)

Comment on "Inflammasome inhibition prevents α-synuclein pathology and dopaminergic neurodegeneration in mice"

Science translational medicine, 2023-05, Vol.15 (696), p.eade8728

2023

Volltextzugriff (PDF)

1 – 25 von 119

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(116)
Mit Volltextzugriff
(103)

Publikationsform

Artikel
(117)
Bücher
(2)

Sprache

Englisch
(118)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 30 Tagen
(1)
... seit 90 Tagen
(4)

Menü

Weitere Dienste

Einstellungen

Microglia in Alzheimer's disease

Diverse modes of NMDA receptor positive allosteric modulation: Mechanisms and consequences

Changes in the Synaptic Proteome in Tauopathy and Rescue of Tau-Induced Synapse Loss by C1q Antibodies

Complement C3 Is Activated in Human AD Brain and Is Required for Neurodegeneration in Mouse Models of Amyloidosis and Tauopathy

Higher ambient synaptic glutamate at inhibitory versus excitatory neurons differentially impacts NMDA receptor activity

Positive Allosteric Modulators of GluN2A-Containing NMDARs with Distinct Modes of Action and Impacts on Circuit Function

SAHA enhances synaptic function and plasticity in vitro but has limited brain availability in vivo and does not impact cognition

Complement C1q-dependent excitatory and inhibitory synapse elimination by astrocytes and microglia in Alzheimer's disease mouse models

Identifying faulty brain circuits

Targeting synapse function and loss for treatment of neurodegenerative diseases

GluN2A NMDA Receptor Enhancement Improves Brain Oscillations, Synchrony, and Cognitive Functions in Dravet Syndrome and Alzheimer’s Disease Models

Protocol for detection of glial complement expression in relation to amyloid plaques in mouse brain with combined FISH and IHC

Author Correction: Higher ambient synaptic glutamate at inhibitory versus excitatory neurons differentially impacts NMDA receptor activity

Dual leucine zipper kinase is required for excitotoxicity-induced neuronal degeneration

TPL2 kinase activity regulates microglial inflammatory responses and promotes neurodegeneration in tauopathy mice

Altered GluN2B NMDA receptor function and synaptic plasticity during early pathology in the PS2APP mouse model of Alzheimer's disease

β-Amyloid Inhibits E-S Potentiation through Suppression of Cannabinoid Receptor 1-Dependent Synaptic Disinhibition

Computationally Discovered Potentiating Role of Glycans on NMDA Receptors

GluN2B Antagonism Affects Interneurons and Leads to Immediate and Persistent Changes in Synaptic Plasticity, Oscillations, and Behavior

Histone deacetylase 2 cell autonomously suppresses excitatory and enhances inhibitory synaptic function in CA1 pyramidal neurons

The functional nature of synaptic circuitry is altered in area CA3 of the hippocampus in a mouse model of Down's syndrome

Therapeutic potential of N-methyl-D-aspartate receptor modulators in psychiatry

Dendritic sodium channels regulate network integration in globus pallidus neurons: a modeling study

Cognitive Deficits, Changes in Synaptic Function, and Brain Pathology in a Mouse Model of Normal Aging(1,2,3)

Comment on "Inflammasome inhibition prevents α-synuclein pathology and dopaminergic neurodegeneration in mice"

Suchergebnisse filtern