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Di(2-ethylhexyl) phthalate inhibits antral follicle growth, induces atresia, and inhibits steroid hormone production in cultured mouse antral follicles

Toxicology and applied pharmacology, 2015-04, Vol.284 (1), p.42-53

2015

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Loss of Fertility in the Absence of Progesterone Receptor Expression in Kisspeptin Neurons of Female Mice

PloS one, 2016-07, Vol.11 (7), p.e0159534

2016

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Coordinated Regulation Among Progesterone, Prostaglandins, and EGF-Like Factors in Human Ovulatory Follicles

The journal of clinical endocrinology and metabolism, 2017-06, Vol.102 (6), p.1971-1982

2017

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Granulosa cell endothelin-2 expression is fundamental for ovulatory follicle rupture

Scientific reports, 2017-04, Vol.7 (1), p.817-11, Article 817

2017

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Daily exposure to Di(2-ethylhexyl) phthalate alters estrous cyclicity and accelerates primordial follicle recruitment potentially via dysregulation of the phosphatidylinositol 3-kinase signaling pathway in adult mice

Biology of reproduction, 2014-06, Vol.90 (6), p.136

2014

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Acute Exposure to Di(2-Ethylhexyl) Phthalate in Adulthood Causes Adverse Reproductive Outcomes Later in Life and Accelerates Reproductive Aging in Female Mice

Toxicological sciences, 2016-03, Vol.150 (1), p.97-108

2016

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The effects of phthalates on the ovary

Frontiers in endocrinology (Lausanne), 2015, Vol.6, p.8

2015

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Mono(2-ethylhexyl) phthalate accelerates early folliculogenesis and inhibits steroidogenesis in cultured mouse whole ovaries and antral follicles

Biology of reproduction, 2015-05, Vol.92 (5), p.120

2015

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The effects of endocrine‐disrupting chemicals on ovarian‐ and ovulation‐related fertility outcomes

Molecular reproduction and development, 2022-12, Vol.89 (12), p.608-631

2022

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Ovulation is Inhibited by an Environmentally Relevant Phthalate Mixture in Mouse Antral Follicles In Vitro

Toxicological sciences, 2021-01, Vol.179 (2), p.195-205

2021

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Exposure to a phthalate mixture disrupts ovulatory progesterone receptor signaling in human granulosa cells in vitro

Biology of reproduction, 2023-10, Vol.109 (4), p.552

2023

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Di-n-Butyl Phthalate Disrupts the Expression of Genes Involved in Cell Cycle and Apoptotic Pathways in Mouse Ovarian Antral Follicles1

Biology of reproduction, 2013-01, Vol.88 (1)

2013

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Di-n-Butyl Phthalate Disrupts the Expression of Genes Involved in Cell Cycle and Apoptotic Pathways in Mouse Ovarian Antral Follicles

Biology of reproduction, 2013, Vol.88 (1), p.23

2013

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Di (2-ethylhexyl) phthalate (DEHP) alters proliferation and uterine gland numbers in the uteri of adult exposed mice

Reproductive toxicology (Elmsford, N.Y.), 2018-04, Vol.77, p.70-79

2018

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Neurotensin expression, regulation, and function during the ovulatory period in the mouse ovary

Biology of reproduction, 2023-01, Vol.108 (1), p.107-120

2023

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Ovulatory Induction of SCG2 in Human, Nonhuman Primate, and Rodent Granulosa Cells Stimulates Ovarian Angiogenesis

Endocrinology (Philadelphia), 2018-06, Vol.159 (6), p.2447-2458

2018

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Pregnenolone co-treatment partially restores steroidogenesis, but does not prevent growth inhibition and increased atresia in mouse ovarian antral follicles treated with mono-hydroxy methoxychlor

Toxicology and applied pharmacology, 2013-11, Vol.272 (3), p.780-786

2013

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Daily Exposure to Di(2-ethylhexyl) Phthalate Alters Estrous Cyclicity and Accelerates Primordial Follicle Recruitment Potentially Via Dysregulation of the Phosphatidylinositol 3-Kinase Signaling Pathway in Adult Mice1

Biology of reproduction, 2014-06, Vol.90 (6)

2014

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Mono(2-Ethylhexyl) Phthalate Accelerates Early Folliculogenesis and Inhibits Steroidogenesis in Cultured Mouse Whole Ovaries and Antral Follicles1

Biology of reproduction, 2015-05, Vol.92 (5)

2015

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Estrogen receptor alpha overexpressing mouse antral follicles are sensitive to atresia induced by methoxychlor and its metabolites

Reproductive toxicology (Elmsford, N.Y.), 2012-06, Vol.33 (3), p.353-360

2012

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Di(2-ethylhexyl) Phthalate Alters Mouse Ovarian Follicle Numbers and Atresia In Vivo

Biology of reproduction, 2012-08, Vol.87 (Suppl_1), p.260-260

2012

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The amount of rearfoot motion used during the stance phase of walking

Journal of the American Podiatric Medical Association, 2005-07, Vol.95 (4), p.376

2005

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Dibutyl Phthalate (DBP) Alters the Levels of Cell Proliferation and Apoptosis Genes in Mouse Granulosa Cells

Biology of reproduction, 2011-07, Vol.85 (Suppl_1), p.276-276

2011

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Dibutyl Phthalate (DBP) Suppresses Growth of Isolated Mouse Antral Follicles

Biology of reproduction, 2011-07, Vol.85 (Suppl_1), p.270-270

2011

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Dibutyl Phthalate (DBP) Induces Cell Cycle Arrest and Apoptosis in Ovarian Antral Follicles

Biology of reproduction, 2012-08, Vol.87 (Suppl_1), p.250-250

2012

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Di(2-ethylhexyl) phthalate inhibits antral follicle growth, induces atresia, and inhibits steroid hormone production in cultured mouse antral follicles

Loss of Fertility in the Absence of Progesterone Receptor Expression in Kisspeptin Neurons of Female Mice

Coordinated Regulation Among Progesterone, Prostaglandins, and EGF-Like Factors in Human Ovulatory Follicles

Granulosa cell endothelin-2 expression is fundamental for ovulatory follicle rupture

Daily exposure to Di(2-ethylhexyl) phthalate alters estrous cyclicity and accelerates primordial follicle recruitment potentially via dysregulation of the phosphatidylinositol 3-kinase signaling pathway in adult mice

Acute Exposure to Di(2-Ethylhexyl) Phthalate in Adulthood Causes Adverse Reproductive Outcomes Later in Life and Accelerates Reproductive Aging in Female Mice

The effects of phthalates on the ovary

Mono(2-ethylhexyl) phthalate accelerates early folliculogenesis and inhibits steroidogenesis in cultured mouse whole ovaries and antral follicles

The effects of endocrine‐disrupting chemicals on ovarian‐ and ovulation‐related fertility outcomes

Ovulation is Inhibited by an Environmentally Relevant Phthalate Mixture in Mouse Antral Follicles In Vitro

Exposure to a phthalate mixture disrupts ovulatory progesterone receptor signaling in human granulosa cells in vitro

Di-n-Butyl Phthalate Disrupts the Expression of Genes Involved in Cell Cycle and Apoptotic Pathways in Mouse Ovarian Antral Follicles1

Di-n-Butyl Phthalate Disrupts the Expression of Genes Involved in Cell Cycle and Apoptotic Pathways in Mouse Ovarian Antral Follicles

Di (2-ethylhexyl) phthalate (DEHP) alters proliferation and uterine gland numbers in the uteri of adult exposed mice

Neurotensin expression, regulation, and function during the ovulatory period in the mouse ovary

Ovulatory Induction of SCG2 in Human, Nonhuman Primate, and Rodent Granulosa Cells Stimulates Ovarian Angiogenesis

Pregnenolone co-treatment partially restores steroidogenesis, but does not prevent growth inhibition and increased atresia in mouse ovarian antral follicles treated with mono-hydroxy methoxychlor

Daily Exposure to Di(2-ethylhexyl) Phthalate Alters Estrous Cyclicity and Accelerates Primordial Follicle Recruitment Potentially Via Dysregulation of the Phosphatidylinositol 3-Kinase Signaling Pathway in Adult Mice1

Mono(2-Ethylhexyl) Phthalate Accelerates Early Folliculogenesis and Inhibits Steroidogenesis in Cultured Mouse Whole Ovaries and Antral Follicles1

Estrogen receptor alpha overexpressing mouse antral follicles are sensitive to atresia induced by methoxychlor and its metabolites

Di(2-ethylhexyl) Phthalate Alters Mouse Ovarian Follicle Numbers and Atresia In Vivo

The amount of rearfoot motion used during the stance phase of walking

Dibutyl Phthalate (DBP) Alters the Levels of Cell Proliferation and Apoptosis Genes in Mouse Granulosa Cells

Dibutyl Phthalate (DBP) Suppresses Growth of Isolated Mouse Antral Follicles

Dibutyl Phthalate (DBP) Induces Cell Cycle Arrest and Apoptosis in Ovarian Antral Follicles

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