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MYC cofactors: molecular switches controlling diverse biological outcomes

Cold Spring Harbor perspectives in medicine, 2014-09, Vol.4 (9), p.a014399-a014399

Hann, Stephen R

2014

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Egr1 mediates p53-independent c-Myc—induced apoptosis via a noncanonical ARF-dependent transcriptional mechanism

Proceedings of the National Academy of Sciences - PNAS, 2011-01, Vol.108 (2), p.632-637

2011

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Role of post-translational modifications in regulating c-Myc proteolysis, transcriptional activity and biological function

Seminars in cancer biology, 2006-08, Vol.16 (4), p.288-302

Hann, Stephen R.

2006

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Nucleophosmin Interacts Directly with c-Myc and Controls c-Myc-Induced Hyperproliferation and Transformation

Proceedings of the National Academy of Sciences - PNAS, 2008-12, Vol.105 (48), p.18794-18799

2008

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Phosphorylation by Glycogen Synthase Kinase-3 Controls c-Myc Proteolysis and Subnuclear Localization

The Journal of biological chemistry, 2003-12, Vol.278 (51), p.51606-51612

2003

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Thr 163 phosphorylation causes Mcl-1 stabilization when degradation is independent of the adjacent GSK3-targeted phosphodegron, promoting drug resistance in cancer

PloS one, 2012-10, Vol.7 (10), p.e47060

2012

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Oikos and Market: Explorations in Self-Sufficiency after Socialism

1, 2015

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MB0 and MBI Are Independent and Distinct Transactivation Domains in MYC that Are Essential for Transformation

Genes, 2017-05, Vol.8 (5), p.134

2017

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The Myc-Nucleophosmin-ARF network: A complex web unveiled

Cell cycle (Georgetown, Tex.), 2009-09, Vol.8 (17), p.2703-2707

2009

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MCL1 is phosphorylated in the PEST region and stabilized upon ERK activation in viable cells, and at additional sites with cytotoxic okadaic acid or taxol

Oncogene, 2004-07, Vol.23 (31), p.5301-5315

2004

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Targeting Nucleophosmin 1 Represents a Rational Strategy for Radiation Sensitization

International journal of radiation oncology, biology, physics, 2014-08, Vol.89 (5), p.1106-1114

2014

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Economy and ritual: studies of postsocialist transformations

1, 2015

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The Myc-ARF-Egr1 pathway: Unleashing the apoptotic power of c-Myc

Cell cycle (Georgetown, Tex.), 2011-07, Vol.10 (13), p.2043-2044

2011

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Small-molecule high-throughput screening utilizing Xenopus egg extract

Methods in molecular biology (Clifton, N.J.), 2015, Vol.1263, p.63

2015

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c-Myc Proteolysis by the Ubiquitin-Proteasome Pathway: Stabilization of c-Myc in Burkitt's Lymphoma Cells

Molecular and Cellular Biology, 2000-04, Vol.20 (7), p.2423-2435

2000

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Determination of Isotopologue and Tandem Mass Isotopologue Ratios Using Gas Chromatography Chemical Ionization Time of Flight Mass Spectrometry - Methodology and Uncertainty of Measurement

Methods in molecular biology (Clifton, N.J.), 2020, Vol.2088, p.1

2020

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c-Myc represses FOXO3a-mediated transcription of the gene encoding the p27(Kip1) cyclin dependent kinase inhibitor

Journal of cellular biochemistry, 2008-08, Vol.104 (6), p.2091-2106

2008

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Myc-mediated transformation: the repression connection

Oncogene, 1999-05, Vol.18 (19), p.2925-2933

1999

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Nucleophosmin is essential for c-Myc nucleolar localization and c-Myc-mediated rDNA transcription

Oncogene, 2013-04, Vol.32 (15), p.1988-1994

2013

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The c-Myc Transactivation Domain Is a Direct Modulator of Apoptotic versus Proliferative Signals

Molecular and Cellular Biology, 2000-06, Vol.20 (12), p.4309-4319

2000

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Transactivation-defective c-MycS retains the ability to regulate proliferation and apoptosis

Genes & development, 1998-12, Vol.12 (24), p.3803-3808

1998

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The ARF Tumor Suppressor: Keeping Myc on a Leash

Cell cycle (Georgetown, Tex.), 2005-02, Vol.4 (2), p.248-251

2005

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Identification of downstream-initiated c-Myc proteins which are dominant-negative inhibitors of transactivation by full-length c-Myc proteins

Molecular and Cellular Biology, 1997-03, Vol.17 (3), p.1459-1468

1997

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p19ARF directly and differentially controls the functions of c-Myc independently of p53

Nature (London), 2004-10, Vol.431 (7009), p.712-717

2004

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Robust Analytical Methods for the Accurate Quantification of the Total Biomass Composition of Mammalian Cells

Methods in molecular biology (Clifton, N.J.), 2020, Vol.2088, p.119

2020

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MYC cofactors: molecular switches controlling diverse biological outcomes

Egr1 mediates p53-independent c-Myc—induced apoptosis via a noncanonical ARF-dependent transcriptional mechanism

Role of post-translational modifications in regulating c-Myc proteolysis, transcriptional activity and biological function

Nucleophosmin Interacts Directly with c-Myc and Controls c-Myc-Induced Hyperproliferation and Transformation

Phosphorylation by Glycogen Synthase Kinase-3 Controls c-Myc Proteolysis and Subnuclear Localization

Thr 163 phosphorylation causes Mcl-1 stabilization when degradation is independent of the adjacent GSK3-targeted phosphodegron, promoting drug resistance in cancer

Oikos and Market: Explorations in Self-Sufficiency after Socialism

MB0 and MBI Are Independent and Distinct Transactivation Domains in MYC that Are Essential for Transformation

The Myc-Nucleophosmin-ARF network: A complex web unveiled

MCL1 is phosphorylated in the PEST region and stabilized upon ERK activation in viable cells, and at additional sites with cytotoxic okadaic acid or taxol

Targeting Nucleophosmin 1 Represents a Rational Strategy for Radiation Sensitization

Economy and ritual: studies of postsocialist transformations

The Myc-ARF-Egr1 pathway: Unleashing the apoptotic power of c-Myc

Small-molecule high-throughput screening utilizing Xenopus egg extract

c-Myc Proteolysis by the Ubiquitin-Proteasome Pathway: Stabilization of c-Myc in Burkitt's Lymphoma Cells

Determination of Isotopologue and Tandem Mass Isotopologue Ratios Using Gas Chromatography Chemical Ionization Time of Flight Mass Spectrometry - Methodology and Uncertainty of Measurement

c-Myc represses FOXO3a-mediated transcription of the gene encoding the p27(Kip1) cyclin dependent kinase inhibitor

Myc-mediated transformation: the repression connection

Nucleophosmin is essential for c-Myc nucleolar localization and c-Myc-mediated rDNA transcription

The c-Myc Transactivation Domain Is a Direct Modulator of Apoptotic versus Proliferative Signals

Transactivation-defective c-MycS retains the ability to regulate proliferation and apoptosis

The ARF Tumor Suppressor: Keeping Myc on a Leash

Identification of downstream-initiated c-Myc proteins which are dominant-negative inhibitors of transactivation by full-length c-Myc proteins

p19ARF directly and differentially controls the functions of c-Myc independently of p53

Robust Analytical Methods for the Accurate Quantification of the Total Biomass Composition of Mammalian Cells

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