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NF-κB modifies the mammalian circadian clock through interaction with the core clock protein BMAL1

PLoS genetics, 2021-11, Vol.17 (11), p.e1009933-e1009933

2021

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NOX4 inhibition promotes the remodeling of dystrophic muscle

JCI insight, 2022-10, Vol.7 (20)

Hammers, David W

2022

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Increased collagen cross-linking is a signature of dystrophin-deficient muscle

Muscle & nerve, 2016-06, Vol.54 (1), p.71-78

2016

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Activin Receptor Type IIB Inhibition Improves Muscle Phenotype and Function in a Mouse Model of Spinal Muscular Atrophy

PloS one, 2016-11, Vol.11 (11), p.e0166803-e0166803

2016

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MYOD1 functions as a clock amplifier as well as a critical co-factor for downstream circadian gene expression in muscle

eLife, 2019-02, Vol.8

2019

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The skeletal muscle circadian clock regulates titin splicing through RBM20

eLife, 2022-09, Vol.11

2022

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Anti-inflammatory macrophages improve skeletal muscle recovery from ischemia-reperfusion

Journal of applied physiology (1985), 2015-04, Vol.118 (8), p.1067-1074

2015

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Muscle Contraction

Cold Spring Harbor perspectives in biology, 2018-02, Vol.10 (2), p.a023200

2018

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Filopodia powered by class x myosin promote fusion of mammalian myoblasts

eLife, 2021-09, Vol.10

2021

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Blocking muscle wasting via deletion of the muscle-specific E3 ligase MuRF1 impedes pancreatic tumor growth

Communications biology, 2023-05, Vol.6 (1), p.519-519, Article 519

2023

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Evaluation of the DBA/2J mouse as a potential background strain for genetic models of cardiomyopathy

Journal of Molecular and Cellular Cardiology Plus (Online), 2022-09, Vol.1, p.100012, Article 100012

2022

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Tadalafil Treatment Delays the Onset of Cardiomyopathy in Dystrophin‐Deficient Hearts

Journal of the American Heart Association, 2016-08, Vol.5 (8), p.n/a

2016

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The Nuclear Receptor PPARγ Controls Progressive Macrophage Polarization as a Ligand-Insensitive Epigenomic Ratchet of Transcriptional Memory

Immunity (Cambridge, Mass.), 2018-10, Vol.49 (4), p.615-626.e6

2018

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RevAMP(K)ing Mitochondria for Sarcoglycanopathy Therapeutics

Circulation. Heart failure, 2019-04, Vol.12 (4), p.e005873-e005873

Hammers, David W

2019

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The D2.mdx mouse as a preclinical model of the skeletal muscle pathology associated with Duchenne muscular dystrophy

Scientific reports, 2020-08, Vol.10 (1), p.14070-14070, Article 14070

2020

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Supraphysiological levels of GDF11 induce striated muscle atrophy

EMBO molecular medicine, 2017-04, Vol.9 (4), p.531-544

2017

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Functional deficits and insulin-like growth factor-I gene expression following tourniquet-induced injury of skeletal muscle in young and old rats

Journal of applied physiology (1985), 2008-10, Vol.105 (4), p.1274-1281

2008

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Evaluating Genetic Modifiers of Duchenne Muscular Dystrophy Disease Progression Using Modeling and MRI

Neurology, 2022-11, Vol.99 (21), p.e2406

2022

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Effects of PDE5 inhibition on dystrophic muscle following an acute bout of downhill running and endurance training

Journal of applied physiology (1985), 2019-06, Vol.126 (6), p.1737-1745

2019

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Functional assessment of skeletal muscle regeneration utilizing homologous extracellular matrix as scaffolding

Tissue engineering. Part A, 2010-04, Vol.16 (4), p.1395

2010

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Repair of traumatic skeletal muscle injury with bone-marrow-derived mesenchymal stem cells seeded on extracellular matrix

Tissue engineering. Part A, 2010-09, Vol.16 (9), p.2871

2010

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Functional muscle hypertrophy by increased insulin‐like growth factor 1 does not require dysferlin

Muscle & nerve, 2019-10, Vol.60 (4), p.464-473

2019

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Glucocorticoids counteract hypertrophic effects of myostatin inhibition in dystrophic muscle

JCI insight, 2020-01, Vol.5 (1)

2020

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Smooth muscle atrophy and colon pathology in SMN deficient mice

American journal of translational research, 2019-01, Vol.11 (3), p.1789-1799

2019

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Controlled release of IGF-I from a biodegradable matrix improves functional recovery of skeletal muscle from ischemia/reperfusion

Biotechnology and bioengineering, 2012-04, Vol.109 (4), p.1051-1059

2012

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NF-κB modifies the mammalian circadian clock through interaction with the core clock protein BMAL1

NOX4 inhibition promotes the remodeling of dystrophic muscle

Increased collagen cross-linking is a signature of dystrophin-deficient muscle

Activin Receptor Type IIB Inhibition Improves Muscle Phenotype and Function in a Mouse Model of Spinal Muscular Atrophy

MYOD1 functions as a clock amplifier as well as a critical co-factor for downstream circadian gene expression in muscle

The skeletal muscle circadian clock regulates titin splicing through RBM20

Anti-inflammatory macrophages improve skeletal muscle recovery from ischemia-reperfusion

Muscle Contraction

Filopodia powered by class x myosin promote fusion of mammalian myoblasts

Blocking muscle wasting via deletion of the muscle-specific E3 ligase MuRF1 impedes pancreatic tumor growth

Evaluation of the DBA/2J mouse as a potential background strain for genetic models of cardiomyopathy

Tadalafil Treatment Delays the Onset of Cardiomyopathy in Dystrophin‐Deficient Hearts

The Nuclear Receptor PPARγ Controls Progressive Macrophage Polarization as a Ligand-Insensitive Epigenomic Ratchet of Transcriptional Memory

RevAMP(K)ing Mitochondria for Sarcoglycanopathy Therapeutics

The D2.mdx mouse as a preclinical model of the skeletal muscle pathology associated with Duchenne muscular dystrophy

Supraphysiological levels of GDF11 induce striated muscle atrophy

Functional deficits and insulin-like growth factor-I gene expression following tourniquet-induced injury of skeletal muscle in young and old rats

Evaluating Genetic Modifiers of Duchenne Muscular Dystrophy Disease Progression Using Modeling and MRI

Effects of PDE5 inhibition on dystrophic muscle following an acute bout of downhill running and endurance training

Functional assessment of skeletal muscle regeneration utilizing homologous extracellular matrix as scaffolding

Repair of traumatic skeletal muscle injury with bone-marrow-derived mesenchymal stem cells seeded on extracellular matrix

Functional muscle hypertrophy by increased insulin‐like growth factor 1 does not require dysferlin

Glucocorticoids counteract hypertrophic effects of myostatin inhibition in dystrophic muscle

Smooth muscle atrophy and colon pathology in SMN deficient mice

Controlled release of IGF-I from a biodegradable matrix improves functional recovery of skeletal muscle from ischemia/reperfusion

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