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PERK-dependent regulation of IAP translation during ER stress

Oncogene, 2009-02, Vol.28 (6), p.910-920

2009

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Polo-like kinases (Plks) and cancer

Oncogene, 2005-01, Vol.24 (2), p.287-291

2005

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Increased globotriaosylceramide levels in a transgenic mouse expressing human 1,4-galactosyltransferase and a mouse model for treating Fabry disease

Journal of biochemistry (Tokyo), 2011-02, Vol.149 (2), p.161-170

2011

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C107 MITOCHONDRIA AND METABOLIC ALTERATIONS IN LUNG DISEASE: Hif-1α Is Required For Flow-Mediated Regulation Of Endothelial Cell Metabolism

American journal of respiratory and critical care medicine, 2017-01, Vol.195

2017

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C73 OXIDANTS: Reactive Oxygen Species Are Required For Metabolic Reprogramming In Endothelial Cells Following Loss Of Laminar Shear Stress

American journal of respiratory and critical care medicine, 2016-01, Vol.193, p.1

2016

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A74 REGULATORY MECHANISMS OF THE MOLECULAR PATHWAYS IN FIBROSIS: Activation Of Adenylyl Cyclase/camp Pathway Attenuates Tgf-β Transcriptional Response In Human Lung Fibroblasts Via Epac

American journal of respiratory and critical care medicine, 2017-01, Vol.195

2017

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C103 OUTDOOR AIR POLLUTION: EPIDEMIOLOGY AND MECHANISMS: Short-Term Acute Exposure Of Healthy Humans To Particulate Matter Induces Differential Gene Expression In Lung Immune Cells

American journal of respiratory and critical care medicine, 2017-01, Vol.195

2017

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Endogenous basic fibroblast growth factor-dependent induction of collagenase and interleukin-6 in tumor necrosis factor-treated human microvascular endothelial cells

The Journal of biological chemistry, 1991-10, Vol.266 (29), p.19162-19165

1991

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C17 EMERGING MECHANISMS IN LUNG INJURY AND INFECTION: [Beta]2-Agonists Promote Influenza A-Induced Lung Injury Via Increased Il-6 Release From Alveolar Macrophages

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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C30 ACUTE LUNG INJURY: CELL FUNCTION, SIGNALING AND STRETCH: Activation Of The â2-Adrenergic Receptors On Monocytes Negatively Regulates Their Recruitment To The Injured Lung

American journal of respiratory and critical care medicine, 2016-01, Vol.193, p.1

2016

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PERK and GCN2 Contribute to eIF2α Phosphorylation and Cell Cycle Arrest after Activation of the Unfolded Protein Response Pathway

Molecular biology of the cell, 2005-12, Vol.16 (12), p.5493-5501

2005

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C107 MITOCHONDRIA AND METABOLIC ALTERATIONS IN LUNG DISEASE: De Novo Serine/glycine Synthesis Is Required For Tgf-β-Induced Collagen Production In Human Lung Fibroblasts

American journal of respiratory and critical care medicine, 2017-01, Vol.195

2017

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B46 HEALTH EFFECTS OF AIR POLLUTION AND NANOPARTICLES: Estrogen Inhibits Particulate Matter-Induced Il-6 Response

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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Interstitial Lung Disease (ILD) After Pleurodesis Against Malignant Pleural Effusion (MPE)

Annals of oncology, 2013-11, Vol.24, p.ix61

2013

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Induction of low density lipoprotein receptor and a transcription factor SP-1 by tumor necrosis factor in human microvascular endothelial cells

The Journal of biological chemistry, 1992-07, Vol.267 (19), p.13160-13165

1992

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C25 THUNDERBALL: SPEEDING FORWARD OUR UNDERSTANDING OF SARCOIDOSIS: Serum Levels Of Autoantibodies Against Granulocyte-Macrophage Colony-Stimulating Factor (gm-Csf) In Sarcoidosis

American journal of respiratory and critical care medicine, 2014-01, Vol.189, p.1

2014

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Expression of polo-like kinase in ovarian cancer is associated with histological grade and clinical stage

Cancer letters, 2001-03, Vol.164 (1), p.41-49

2001

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Tumor necrosis factor and epidermal growth factor modulate migration of human microvascular endothelial cells and production of tissue-type plasminogen activator and its inhibitor

Experimental cell research, 1991-02, Vol.192 (2), p.574-580

1991

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Polo-like kinase expression in normal human endometrium during the menstrual cycle

Reproduction fertility and development, 2000, Vol.12 (1-2), p.59

2000

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Malignant transformation of mammalian cells initiated by constitutive expression of the polo-like kinase

Biochemical and biophysical research communications, 1997-05, Vol.234 (2), p.397

1997

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Polo-like kinase (PLK) expression in endometrial carcinoma

Cancer letters, 2001-08, Vol.169 (1), p.41-49

2001

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Biomechanical aspects of segmented arch mechanics combined with power arm for controlled anterior tooth movement: A three-dimensional finite element study

Journal of dental biomechanics, 2015, Vol.6, p.1758736014566337-1758736014566337

2015

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Polo-like Kinase Is a Cell Cycle-regulated Kinase Activated during Mitosis (∗)

The Journal of biological chemistry, 1995-09, Vol.270 (36), p.21086-21091

1995

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The response to epidermal growth factor of human maxillary tumor cells in terms of tumor growth, invasion and expression of proteinase inhibitors

International journal of cancer, 1991-11, Vol.49 (5), p.738-743

1991

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PERK-dependent regulation of IAP translation during ER stress

Polo-like kinases (Plks) and cancer

Increased globotriaosylceramide levels in a transgenic mouse expressing human 1,4-galactosyltransferase and a mouse model for treating Fabry disease

C107 MITOCHONDRIA AND METABOLIC ALTERATIONS IN LUNG DISEASE: Hif-1α Is Required For Flow-Mediated Regulation Of Endothelial Cell Metabolism

C73 OXIDANTS: Reactive Oxygen Species Are Required For Metabolic Reprogramming In Endothelial Cells Following Loss Of Laminar Shear Stress

A74 REGULATORY MECHANISMS OF THE MOLECULAR PATHWAYS IN FIBROSIS: Activation Of Adenylyl Cyclase/camp Pathway Attenuates Tgf-β Transcriptional Response In Human Lung Fibroblasts Via Epac

C103 OUTDOOR AIR POLLUTION: EPIDEMIOLOGY AND MECHANISMS: Short-Term Acute Exposure Of Healthy Humans To Particulate Matter Induces Differential Gene Expression In Lung Immune Cells

Endogenous basic fibroblast growth factor-dependent induction of collagenase and interleukin-6 in tumor necrosis factor-treated human microvascular endothelial cells

C17 EMERGING MECHANISMS IN LUNG INJURY AND INFECTION: [Beta]2-Agonists Promote Influenza A-Induced Lung Injury Via Increased Il-6 Release From Alveolar Macrophages

C30 ACUTE LUNG INJURY: CELL FUNCTION, SIGNALING AND STRETCH: Activation Of The â2-Adrenergic Receptors On Monocytes Negatively Regulates Their Recruitment To The Injured Lung

PERK and GCN2 Contribute to eIF2α Phosphorylation and Cell Cycle Arrest after Activation of the Unfolded Protein Response Pathway

C107 MITOCHONDRIA AND METABOLIC ALTERATIONS IN LUNG DISEASE: De Novo Serine/glycine Synthesis Is Required For Tgf-β-Induced Collagen Production In Human Lung Fibroblasts

B46 HEALTH EFFECTS OF AIR POLLUTION AND NANOPARTICLES: Estrogen Inhibits Particulate Matter-Induced Il-6 Response

Metformin Targets Mitochondrial Electron Transport to Reduce Air-Pollution-Induced Thrombosis

Interstitial Lung Disease (ILD) After Pleurodesis Against Malignant Pleural Effusion (MPE)

Induction of low density lipoprotein receptor and a transcription factor SP-1 by tumor necrosis factor in human microvascular endothelial cells

C25 THUNDERBALL: SPEEDING FORWARD OUR UNDERSTANDING OF SARCOIDOSIS: Serum Levels Of Autoantibodies Against Granulocyte-Macrophage Colony-Stimulating Factor (gm-Csf) In Sarcoidosis

Expression of polo-like kinase in ovarian cancer is associated with histological grade and clinical stage

Tumor necrosis factor and epidermal growth factor modulate migration of human microvascular endothelial cells and production of tissue-type plasminogen activator and its inhibitor

Polo-like kinase expression in normal human endometrium during the menstrual cycle

Malignant transformation of mammalian cells initiated by constitutive expression of the polo-like kinase

Polo-like kinase (PLK) expression in endometrial carcinoma

Biomechanical aspects of segmented arch mechanics combined with power arm for controlled anterior tooth movement: A three-dimensional finite element study

Polo-like Kinase Is a Cell Cycle-regulated Kinase Activated during Mitosis (∗)

The response to epidermal growth factor of human maxillary tumor cells in terms of tumor growth, invasion and expression of proteinase inhibitors

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