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Paraoxonase 2 deficiency alters mitochondrial function and exacerbates the development of atherosclerosis

Antioxidants & redox signaling, 2011-02, Vol.14 (3), p.341-351

2011

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Chronic inflammatory disorders and accelerated atherosclerosis: chronic kidney disease

Current pharmaceutical design, 2011, Vol.17 (1), p.17-20

2011

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The paraoxonase gene family and atherosclerosis

Free radical biology & medicine, 2005-01, Vol.38 (2), p.153-163

2005

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Treatment of patients with cardiovascular disease with L-4F, an apo-A1 mimetic, did not improve select biomarkers of HDL function[S]

Journal of lipid research, 2011-02, Vol.52 (2), p.361-373

2011

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Inflammatory/antiinflammatory properties of high-density lipoprotein distinguish patients from control subjects better than high-density lipoprotein Cholesterol levels and are favorably affected by simvastatin Treatment

Circulation (New York, N.Y.), 2003-12, Vol.108 (22), p.2751-2756

2003

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A cell-free assay for detecting HDL that is dysfunctional in preventing the formation of or inactivating oxidized phospholipids

Journal of lipid research, 2001-08, Vol.42 (8), p.1308-1317

2001

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Anti-inflammatory apoA-I-mimetic peptides bind oxidized lipids with much higher affinity than human apoA-I

Journal of lipid research, 2008-11, Vol.49 (11), p.2302-2311

2008

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Hemoglobin and Its Scavenger Protein Haptoglobin Associate with ApoA-1-containing Particles and Influence the Inflammatory Properties and Function of High Density Lipoprotein

The Journal of biological chemistry, 2009-07, Vol.284 (27), p.18292-18301

2009

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Normal high density lipoprotein inhibits three steps in the formation of mildly oxidized low density lipoprotein: steps 2 and 3

Journal of lipid research, 2000-09, Vol.41 (9), p.1495-1508

2000

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Normal high density lipoprotein inhibits three steps in the formation of mildly oxidized low density lipoprotein: step 1

Journal of lipid research, 2000-09, Vol.41 (9), p.1481-1494

2000

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Intestine may be a major site of action for the apoA-I mimetic peptide 4F whether administered subcutaneously or orally[S]

Journal of lipid research, 2011-06, Vol.52 (6), p.1200-1210

2011

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Paraoxonase-2 Is a Ubiquitously Expressed Protein with Antioxidant Properties and Is Capable of Preventing Cell-mediated Oxidative Modification of Low Density Lipoprotein

The Journal of biological chemistry, 2001-11, Vol.276 (48), p.44444-44449

2001

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Oral administration of an apo A-I mimetic peptide synthesized from D-amino acids dramatically reduces atherosclerosis in mice independent of plasma cholesterol

Circulation (New York, N.Y.), 2002-01, Vol.105 (3), p.290-292

2002

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Increased Atherosclerosis in Mice Lacking Apolipoprotein A-I Attributable to Both Impaired Reverse Cholesterol Transport and Increased Inflammation

Circulation research, 2005-10, Vol.97 (8), p.763-771

2005

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Apolipoprotein A-I mimetic peptides

Arteriosclerosis, thrombosis, and vascular biology, 2005-07, Vol.25 (7), p.1325-1331

2005

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Heart Failure is Associated With Impaired Anti-Inflammatory and Antioxidant Properties of High-Density Lipoproteins

The American journal of cardiology, 2013-12, Vol.112 (11), p.1770-1777

2013

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Lipoprotein inflammatory properties and serum amyloid A levels but not cholesterol levels predict lesion area in cholesterol-fed rabbits

Journal of lipid research, 2007-11, Vol.48 (11), p.2344-2353

2007

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D-4F and statins synergize to render HDL antiinflammatory in mice and monkeys and cause lesion regression in old apolipoprotein E-null mice

Arteriosclerosis, thrombosis, and vascular biology, 2005-07, Vol.25 (7), p.1426-1432

2005

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High-density lipoprotein loses its anti-inflammatory properties during acute Influenza A infection

Circulation (New York, N.Y.), 2001-05, Vol.103 (18), p.2283-2288

2001

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HDL and the inflammatory response induced by LDL-derived oxidized phospholipids

Arteriosclerosis, thrombosis, and vascular biology, 2001-04, Vol.21 (4), p.481-488

2001

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A novel method for oral delivery of apolipoprotein mimetic peptides synthesized from all L-amino acids

Journal of lipid research, 2009-08, Vol.50 (8), p.1538-1547

2009

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Adenovirus mediated expression of human paraoxonase 2 protects against the development of atherosclerosis in apolipoprotein E-deficient mice

Molecular genetics and metabolism, 2006-12, Vol.89 (4), p.368-373

2006

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A new synthetic class A amphipathic peptide analogue protects mice from diet-induced atherosclerosis

Journal of lipid research, 2001-04, Vol.42 (4), p.545-552

2001

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Mice lacking serum paraoxonase are susceptible to organophosphate toxicity and atherosclerosis

Nature (London), 1998-07, Vol.394 (6690), p.284-287

1998

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An oral apoJ peptide renders HDL antiinflammatory in mice and monkeys and dramatically reduces atherosclerosis in apolipoprotein E-null mice

Arteriosclerosis, thrombosis, and vascular biology, 2005-09, Vol.25 (9), p.1932-1937

2005

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Paraoxonase 2 deficiency alters mitochondrial function and exacerbates the development of atherosclerosis

Chronic inflammatory disorders and accelerated atherosclerosis: chronic kidney disease

The paraoxonase gene family and atherosclerosis

Treatment of patients with cardiovascular disease with L-4F, an apo-A1 mimetic, did not improve select biomarkers of HDL function[S]

Inflammatory/antiinflammatory properties of high-density lipoprotein distinguish patients from control subjects better than high-density lipoprotein Cholesterol levels and are favorably affected by simvastatin Treatment

A cell-free assay for detecting HDL that is dysfunctional in preventing the formation of or inactivating oxidized phospholipids

Anti-inflammatory apoA-I-mimetic peptides bind oxidized lipids with much higher affinity than human apoA-I

Hemoglobin and Its Scavenger Protein Haptoglobin Associate with ApoA-1-containing Particles and Influence the Inflammatory Properties and Function of High Density Lipoprotein

Normal high density lipoprotein inhibits three steps in the formation of mildly oxidized low density lipoprotein: steps 2 and 3

Normal high density lipoprotein inhibits three steps in the formation of mildly oxidized low density lipoprotein: step 1

Intestine may be a major site of action for the apoA-I mimetic peptide 4F whether administered subcutaneously or orally[S]

Paraoxonase-2 Is a Ubiquitously Expressed Protein with Antioxidant Properties and Is Capable of Preventing Cell-mediated Oxidative Modification of Low Density Lipoprotein

Oral administration of an apo A-I mimetic peptide synthesized from D-amino acids dramatically reduces atherosclerosis in mice independent of plasma cholesterol

Increased Atherosclerosis in Mice Lacking Apolipoprotein A-I Attributable to Both Impaired Reverse Cholesterol Transport and Increased Inflammation

Apolipoprotein A-I mimetic peptides

Heart Failure is Associated With Impaired Anti-Inflammatory and Antioxidant Properties of High-Density Lipoproteins

Lipoprotein inflammatory properties and serum amyloid A levels but not cholesterol levels predict lesion area in cholesterol-fed rabbits

D-4F and statins synergize to render HDL antiinflammatory in mice and monkeys and cause lesion regression in old apolipoprotein E-null mice

High-density lipoprotein loses its anti-inflammatory properties during acute Influenza A infection

HDL and the inflammatory response induced by LDL-derived oxidized phospholipids

A novel method for oral delivery of apolipoprotein mimetic peptides synthesized from all L-amino acids

Adenovirus mediated expression of human paraoxonase 2 protects against the development of atherosclerosis in apolipoprotein E-deficient mice

A new synthetic class A amphipathic peptide analogue protects mice from diet-induced atherosclerosis

Mice lacking serum paraoxonase are susceptible to organophosphate toxicity and atherosclerosis

An oral apoJ peptide renders HDL antiinflammatory in mice and monkeys and dramatically reduces atherosclerosis in apolipoprotein E-null mice

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