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Antioxidant therapies in traumatic brain and spinal cord injury

Biochimica et biophysica acta, 2012-05, Vol.1822 (5), p.675-684

2012

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Antioxidant Therapies for Acute Spinal Cord Injury

Neurotherapeutics, 2011-04, Vol.8 (2), p.152-167

Hall, Edward D.

2011

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trait-based framework for predicting when and where microbial adaptation to climate change will affect ecosystem functioning

Biogeochemistry, 2012-07, Vol.109 (1-3), p.35-47

2012

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Chronic traumatic encephalopathy-integration of canonical traumatic brain injury secondary injury mechanisms with tau pathology

Progress in neurobiology, 2017-11, Vol.158, p.15-44

2017

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Antioxidant Therapies for Traumatic Brain Injury

Neurotherapeutics, 2010-01, Vol.7 (1), p.51-61

2010

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Linking microbial community structure and microbial processes: an empirical and conceptual overview

FEMS microbiology ecology, 2015-10, Vol.91 (10), p.1

2015

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Relationships between protein-encoding gene abundance and corresponding process are commonly assumed yet rarely observed

The ISME Journal, 2015-08, Vol.9 (8), p.1693-1699

2015

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The effects of a hydrolyzed protein diet on the plasma, fecal and urine metabolome in cats with chronic enteropathy

Scientific reports, 2023-11, Vol.13 (1), p.19979-19979, Article 19979

2023

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Methylprednisolone for the Treatment of Patients with Acute Spinal Cord Injuries: A Propensity Score-Matched Cohort Study from a Canadian Multi-Center Spinal Cord Injury Registry

Journal of neurotrauma, 2016-05, Vol.33 (10), p.972-974

Hall, Edward D.

2016

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The effect of a hydrolyzed protein diet on the fecal microbiota in cats with chronic enteropathy

Scientific reports, 2022-02, Vol.12 (1), p.2746-15, Article 2746

2022

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Re-engaging disengaged pupils in physical education: an appreciative inquiry perspective

Sport, education and society, 2019-03, Vol.24 (3), p.241-255

2019

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Lewis Acid Activation of a Hydrogen Bond Donor Metal–Organic Framework for Catalysis

ACS catalysis, 2016-05, Vol.6 (5), p.3248-3252

2016

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Is it time to resurrect “lazaroids”?

Journal of neuroscience research, 2017-01, Vol.95 (1-2), p.17-20

2017

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Lipid peroxidation in brain or spinal cord mitochondria after injury

Journal of bioenergetics and biomembranes, 2016-04, Vol.48 (2), p.169-174

2016

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Nrf2–ARE activator carnosic acid decreases mitochondrial dysfunction, oxidative damage and neuronal cytoskeletal degradation following traumatic brain injury in mice

Experimental neurology, 2015-02, Vol.264, p.103-110

2015

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Time courses of post-injury mitochondrial oxidative damage and respiratory dysfunction and neuronal cytoskeletal degradation in a rat model of focal traumatic brain injury

Neurochemistry international, 2017-12, Vol.111, p.45-56

2017

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Indoleamine-pyrrole 2,3-dioxygenase-1 (IDO-1) mRNA is over-expressed in the duodenal mucosa and is negatively correlated with serum tryptophan concentrations in dogs with protein-losing enteropathy

PloS one, 2019-06, Vol.14 (6), p.e0218218-e0218218

2019

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Administration of the Nrf2–ARE activators sulforaphane and carnosic acid attenuates 4-hydroxy-2-nonenal-induced mitochondrial dysfunction ex vivo

Free radical biology & medicine, 2013-04, Vol.57, p.1-9

2013

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Newer pharmacological approaches for antioxidant neuroprotection in traumatic brain injury

Neuropharmacology, 2019-02, Vol.145 (Pt B), p.247-258

2019

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Time course of post-traumatic mitochondrial oxidative damage and dysfunction in a mouse model of focal traumatic brain injury: implications for neuroprotective therapy

Journal of cerebral blood flow and metabolism, 2006-11, Vol.26 (11), p.1407-1418

2006

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effect of resource quantity and resource stoichiometry on microbial carbon-use-efficiency

FEMS microbiology ecology, 2010-09, Vol.73 (3), p.430-440

2010

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Role of peroxynitrite in secondary oxidative damage after spinal cord injury

Journal of neurochemistry, 2007-02, Vol.100 (3), p.639-649

2007

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Affect and emotions in sports work: a research agenda

Sport in society, 2023-07, Vol.26 (7), p.1161-1177

2023

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Neuroprotection and Acute Spinal Cord Injury: A Reappraisal

NeuroRx, 2004, Vol.1 (1), p.80-100

2004

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Synaptic Mitochondria are More Susceptible to Traumatic Brain Injury-induced Oxidative Damage and Respiratory Dysfunction than Non-synaptic Mitochondria

Neuroscience, 2018-08, Vol.386, p.265-283

2018

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Antioxidant therapies in traumatic brain and spinal cord injury

Antioxidant Therapies for Acute Spinal Cord Injury

trait-based framework for predicting when and where microbial adaptation to climate change will affect ecosystem functioning

Chronic traumatic encephalopathy-integration of canonical traumatic brain injury secondary injury mechanisms with tau pathology

Antioxidant Therapies for Traumatic Brain Injury

Linking microbial community structure and microbial processes: an empirical and conceptual overview

Relationships between protein-encoding gene abundance and corresponding process are commonly assumed yet rarely observed

The effects of a hydrolyzed protein diet on the plasma, fecal and urine metabolome in cats with chronic enteropathy

Methylprednisolone for the Treatment of Patients with Acute Spinal Cord Injuries: A Propensity Score-Matched Cohort Study from a Canadian Multi-Center Spinal Cord Injury Registry

The effect of a hydrolyzed protein diet on the fecal microbiota in cats with chronic enteropathy

Re-engaging disengaged pupils in physical education: an appreciative inquiry perspective

Lewis Acid Activation of a Hydrogen Bond Donor Metal–Organic Framework for Catalysis

Is it time to resurrect “lazaroids”?

Lipid peroxidation in brain or spinal cord mitochondria after injury

Nrf2–ARE activator carnosic acid decreases mitochondrial dysfunction, oxidative damage and neuronal cytoskeletal degradation following traumatic brain injury in mice

Time courses of post-injury mitochondrial oxidative damage and respiratory dysfunction and neuronal cytoskeletal degradation in a rat model of focal traumatic brain injury

Indoleamine-pyrrole 2,3-dioxygenase-1 (IDO-1) mRNA is over-expressed in the duodenal mucosa and is negatively correlated with serum tryptophan concentrations in dogs with protein-losing enteropathy

Administration of the Nrf2–ARE activators sulforaphane and carnosic acid attenuates 4-hydroxy-2-nonenal-induced mitochondrial dysfunction ex vivo

Newer pharmacological approaches for antioxidant neuroprotection in traumatic brain injury

Time course of post-traumatic mitochondrial oxidative damage and dysfunction in a mouse model of focal traumatic brain injury: implications for neuroprotective therapy

effect of resource quantity and resource stoichiometry on microbial carbon-use-efficiency

Role of peroxynitrite in secondary oxidative damage after spinal cord injury

Affect and emotions in sports work: a research agenda

Neuroprotection and Acute Spinal Cord Injury: A Reappraisal

Synaptic Mitochondria are More Susceptible to Traumatic Brain Injury-induced Oxidative Damage and Respiratory Dysfunction than Non-synaptic Mitochondria

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