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Endothelium-Neutrophil Interactions in ANCA-Associated Diseases

Journal of the American Society of Nephrology, 2012-09, Vol.23 (9), p.1449-1461

2012

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Inhibition of the mTORC Pathway in the Antiphospholipid Syndrome

The New England journal of medicine, 2014-07, Vol.371 (4), p.303-312

2014

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Complement alternative pathway acts as a positive feedback amplification of neutrophil activation

Blood, 2011-01, Vol.117 (4), p.1340-1349

2011

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Complement Factor B Mutations in Atypical Hemolytic Uremic Syndrome—Disease-Relevant or Benign?

Journal of the American Society of Nephrology, 2014-09, Vol.25 (9), p.2053-2065

2014

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Human neutrophil integrin α9β1: up-regulation by cell activation and synergy with β2 integrins during adhesion to endothelium under flow

Journal of leukocyte biology, 2010-08, Vol.88 (2), p.321-327

2010

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A prevalent C3 mutation in aHUS patients causes a direct C3 convertase gain of function

Blood, 2012-05, Vol.119 (18), p.4182-4191

2012

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The interaction between factor H and VWF increases factor H cofactor activity and regulates VWF prothrombotic status

Blood, 2014-01, Vol.123 (1), p.121-125

2014

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M-ficolin and leukosialin (CD43): new partners in neutrophil adhesion

Journal of leukocyte biology, 2012-03, Vol.91 (3), p.469-474

2012

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LPS-induced galectin-3 oligomerization results in enhancement of neutrophil activation

PloS one, 2011-10, Vol.6 (10), p.e26004

2011

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Functional complement C1q abnormality leads to impaired immune complexes and apoptotic cell clearance

The Journal of immunology (1950), 2011-10, Vol.187 (8), p.4369-4373

2011

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A large subset of neutrophils expressing membrane proteinase 3 is a risk factor for vasculitis and rheumatoid arthritis

Journal of the American Society of Nephrology, 1999-06, Vol.10 (6), p.1224-1233

1999

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Increased membrane expression of proteinase 3 during neutrophil adhesion in the presence of anti-proteinase 3 antibodies

Journal of the American Society of Nephrology, 2007-08, Vol.18 (8), p.2330-2339

2007

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TNF-Induced β2 Integrin Activation Involves Src Kinases and a Redox-Regulated Activation of p38 MAPK

The Journal of immunology (1950), 2004-07, Vol.173 (2), p.1313-1320

2004

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Complement System Part II: Role in Immunity

Frontiers in immunology, 2015-05, Vol.6, p.257-257

2015

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Distinct Signaling Pathways Are Involved in Leukosialin (CD43) Down-regulation, Membrane Blebbing, and Phospholipid Scrambling during Neutrophil Apoptosis

The Journal of biological chemistry, 2005-02, Vol.280 (7), p.5843-5853

2005

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Complement activation by heme as a secondary hit for atypical hemolytic uremic syndrome

Blood, 2013-07, Vol.122 (2), p.282-292

2013

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Redox regulation of β2‐integrin CD11b / CD18 activation

European journal of immunology, 1999-11, Vol.29 (11), p.3419-3431

1999

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Long-Term Reversal of Established Autoimmunity upon Transient Blockade of the LFA-1/Intercellular Adhesion Molecule-1 Pathway

The Journal of immunology (1950), 2002-04, Vol.168 (7), p.3641-3648

2002

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Alternative complement pathway assessment in patients with atypical HUS

Journal of immunological methods, 2011-02, Vol.365 (1-2), p.8-26

2011

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Neutrophils : Molecules, functions and pathophysiological aspects

Laboratory investigation, 2000-05, Vol.80 (5), p.617-653

2000

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Free heme transforms endothelial cells into alternative pathway activators. Implications for the hemolytic and uremic syndrome

Immunobiology (1979), 2012-11, Vol.217 (11), p.1172-1172

2012

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Circulating microparticles in renal diseases

Nephrology, dialysis, transplantation, 2008-07, Vol.23 (7), p.2129-2132

2008

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Leukosialin (CD43, sialophorin) redistribution in uropods of polarized neutrophils is induced by CD43 cross-linking by antibodies, by colchicine or by chemotactic peptides

Journal of cell science, 1997-07, Vol.110 ( Pt 13) (13), p.1465-1475

1997

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The Cleavage of Neutrophil Leukosialin (CD43) by Cathepsin G Releases Its Extracellular Domain and Triggers Its Intramembrane Proteolysis by Presenilin/Î³-Secretase

The Journal of biological chemistry, 2008-08, Vol.283 (35), p.23627

2008

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Endothelium-Neutrophil Interactions in ANCA-Associated Diseases

Inhibition of the mTORC Pathway in the Antiphospholipid Syndrome

Complement alternative pathway acts as a positive feedback amplification of neutrophil activation

Complement Factor B Mutations in Atypical Hemolytic Uremic Syndrome—Disease-Relevant or Benign?

Human neutrophil integrin α9β1: up-regulation by cell activation and synergy with β2 integrins during adhesion to endothelium under flow

A prevalent C3 mutation in aHUS patients causes a direct C3 convertase gain of function

The interaction between factor H and VWF increases factor H cofactor activity and regulates VWF prothrombotic status

M-ficolin and leukosialin (CD43): new partners in neutrophil adhesion

LPS-induced galectin-3 oligomerization results in enhancement of neutrophil activation

Hyperfunctional C3 convertase leads to complement deposition on endothelial cells and contributes to atypical hemolytic uremic syndrome

Functional complement C1q abnormality leads to impaired immune complexes and apoptotic cell clearance

A large subset of neutrophils expressing membrane proteinase 3 is a risk factor for vasculitis and rheumatoid arthritis

Increased membrane expression of proteinase 3 during neutrophil adhesion in the presence of anti-proteinase 3 antibodies

TNF-Induced β2 Integrin Activation Involves Src Kinases and a Redox-Regulated Activation of p38 MAPK

Complement System Part II: Role in Immunity

Distinct Signaling Pathways Are Involved in Leukosialin (CD43) Down-regulation, Membrane Blebbing, and Phospholipid Scrambling during Neutrophil Apoptosis

Complement activation by heme as a secondary hit for atypical hemolytic uremic syndrome

Redox regulation of β2‐integrin CD11b / CD18 activation

Long-Term Reversal of Established Autoimmunity upon Transient Blockade of the LFA-1/Intercellular Adhesion Molecule-1 Pathway

Alternative complement pathway assessment in patients with atypical HUS

Neutrophils : Molecules, functions and pathophysiological aspects

Free heme transforms endothelial cells into alternative pathway activators. Implications for the hemolytic and uremic syndrome

Circulating microparticles in renal diseases

Leukosialin (CD43, sialophorin) redistribution in uropods of polarized neutrophils is induced by CD43 cross-linking by antibodies, by colchicine or by chemotactic peptides

The Cleavage of Neutrophil Leukosialin (CD43) by Cathepsin G Releases Its Extracellular Domain and Triggers Its Intramembrane Proteolysis by Presenilin/Î³-Secretase

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