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Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons

Brain (London, England : 1878), 2005-08, Vol.128 (8), p.1847-1854

2005

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DagSim: Combining DAG-based model structure with unconstrained data types and relations for flexible, transparent, and modularized data simulation

PloS one, 2023-04, Vol.18 (4), p.e0284443-e0284443

2023

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Nav1.7-related small fiber neuropathy: impaired slow-inactivation and DRG neuron hyperexcitability

Neurology, 2012-05, Vol.78 (21), p.1635

2012

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Factors Associated with Adherence to the Mediterranean Diet and Dietary Habits among University Students in Lebanon

Journal of nutrition and metabolism, 2021, Vol.2021, p.6688462-7

2021

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Sodium Channels in Normal and Pathological Pain

Annual review of neuroscience, 2010-01, Vol.33 (1), p.325-347

2010

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Diversity in health professional education scholarship: a document analysis of international author representation in leading journals

BMJ open, 2020-11, Vol.10 (11), p.e043970-e043970

2020

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NaV1.7 Gain-of-Function Mutations as a Continuum: A1632E Displays Physiological Changes Associated with Erythromelalgia and Paroxysmal Extreme Pain Disorder Mutations and Produces Symptoms of Both Disorders

The Journal of neuroscience, 2008-10, Vol.28 (43), p.11079-11088

2008

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NaN, a Novel Voltage-Gated Na Channel, is Expressed Preferentially in Peripheral Sensory Neurons and Down-Regulated after Axotomy

Proceedings of the National Academy of Sciences - PNAS, 1998-07, Vol.95 (15), p.8963-8968

1998

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Reducing health provider stereotypes through undergraduate interprofessional education

Journal of Taibah University Medical Sciences, 2022-12, Vol.17 (6), p.991-999

2022

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Cost savings and cost avoidance with the inpatient clinical pharmacist interventions in a tertiary cancer care hospital

Journal of oncology pharmacy practice, 2023-12, Vol.29 (8), p.1935-1943

2023

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Distinct repriming and closed-state inactivation kinetics of Nav1.6 and Nav1.7 sodium channels in mouse spinal sensory neurons

The Journal of physiology, 2003-09, Vol.551 (3), p.741-750

2003

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Major clinical response to nivolumab in relapsed/refractory Hodgkin lymphoma after allogeneic stem cell transplantation

Bone marrow transplantation (Basingstoke), 2016-06, Vol.51 (6), p.850-852

2016

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A Novel Persistent Tetrodotoxin-Resistant Sodium Current In SNS-Null And Wild-Type Small Primary Sensory Neurons

The Journal of neuroscience, 1999-12, Vol.19 (24), p.43-RC43

1999

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Nav1.9 expression in magnocellular neurosecretory cells of supraoptic nucleus

Experimental neurology, 2014-03, Vol.253, p.174-179

2014

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Sodium Channels and Pain

Proceedings of the National Academy of Sciences - PNAS, 1999-07, Vol.96 (14), p.7635-7639

1999

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The immuneML ecosystem for machine learning analysis of adaptive immune receptor repertoires

Nature machine intelligence, 2021-11, Vol.3 (11), p.936-944

2021

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Rat NaV1.7 loss-of-function genetic model: Deficient nociceptive and neuropathic pain behavior with retained olfactory function and intra-epidermal nerve fibers

Molecular pain, 2019, Vol.15, p.1744806919881846-1744806919881846

2019

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Erythermalgia: molecular basis for an inherited pain syndrome

Trends in molecular medicine, 2005-12, Vol.11 (12), p.555-562

2005

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Physiological and genetic analysis of multiple sodium channel variants in a model of genetic absence epilepsy

Neurobiology of disease, 2014-07, Vol.67, p.180-190

2014

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HIGH SODIUM CONCENTRATION MODULATES DENDRITIC CELLS IMMUNE FUNCTIONS

Journal of hypertension, 2018-06, Vol.36 Suppl 1 - ESH 2018 Abstract Book (Supplement 1), p.e111-e111

2018

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Changes in Expression of Two Tetrodotoxin-Resistant Sodium Channels and Their Currents in Dorsal Root Ganglion Neurons after Sciatic Nerve Injury But Not Rhizotomy

The Journal of neuroscience, 2000-10, Vol.20 (19), p.7279-7289

2000

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Small-fiber neuropathy Nav1.8 mutation shifts activation to hyperpolarized potentials and increases excitability of dorsal root ganglion neurons

The Journal of neuroscience, 2013-08, Vol.33 (35), p.14087-14097

2013

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Can robots patch‐clamp as well as humans? Characterization of a novel sodium channel mutation

The Journal of physiology, 2010-06, Vol.588 (11), p.1915-1927

2010

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Plasticity of sodium channel expression in DRG neurons in the chronic constriction injury model of neuropathic pain

Pain (Amsterdam), 1999-12, Vol.83 (3), p.591-600

1999

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[OP.3D.01] EFFECT OF SODIUM CHRLORIDE ON HUMAN DENDRITIC CELL FUNCTIONS

Journal of hypertension, 2016-09, Vol.34 Suppl 2 - ESH 2016 Abstract Book (Supplement 2), p.e33-e33

2016

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Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons

DagSim: Combining DAG-based model structure with unconstrained data types and relations for flexible, transparent, and modularized data simulation

Nav1.7-related small fiber neuropathy: impaired slow-inactivation and DRG neuron hyperexcitability

Factors Associated with Adherence to the Mediterranean Diet and Dietary Habits among University Students in Lebanon

Sodium Channels in Normal and Pathological Pain

Diversity in health professional education scholarship: a document analysis of international author representation in leading journals

NaV1.7 Gain-of-Function Mutations as a Continuum: A1632E Displays Physiological Changes Associated with Erythromelalgia and Paroxysmal Extreme Pain Disorder Mutations and Produces Symptoms of Both Disorders

NaN, a Novel Voltage-Gated Na Channel, is Expressed Preferentially in Peripheral Sensory Neurons and Down-Regulated after Axotomy

Reducing health provider stereotypes through undergraduate interprofessional education

Cost savings and cost avoidance with the inpatient clinical pharmacist interventions in a tertiary cancer care hospital

Distinct repriming and closed-state inactivation kinetics of Nav1.6 and Nav1.7 sodium channels in mouse spinal sensory neurons

Major clinical response to nivolumab in relapsed/refractory Hodgkin lymphoma after allogeneic stem cell transplantation

A Novel Persistent Tetrodotoxin-Resistant Sodium Current In SNS-Null And Wild-Type Small Primary Sensory Neurons

Nav1.9 expression in magnocellular neurosecretory cells of supraoptic nucleus

Sodium Channels and Pain

The immuneML ecosystem for machine learning analysis of adaptive immune receptor repertoires

Rat NaV1.7 loss-of-function genetic model: Deficient nociceptive and neuropathic pain behavior with retained olfactory function and intra-epidermal nerve fibers

Erythermalgia: molecular basis for an inherited pain syndrome

Physiological and genetic analysis of multiple sodium channel variants in a model of genetic absence epilepsy

HIGH SODIUM CONCENTRATION MODULATES DENDRITIC CELLS IMMUNE FUNCTIONS

Changes in Expression of Two Tetrodotoxin-Resistant Sodium Channels and Their Currents in Dorsal Root Ganglion Neurons after Sciatic Nerve Injury But Not Rhizotomy

Small-fiber neuropathy Nav1.8 mutation shifts activation to hyperpolarized potentials and increases excitability of dorsal root ganglion neurons

Can robots patch‐clamp as well as humans? Characterization of a novel sodium channel mutation

Plasticity of sodium channel expression in DRG neurons in the chronic constriction injury model of neuropathic pain

[OP.3D.01] EFFECT OF SODIUM CHRLORIDE ON HUMAN DENDRITIC CELL FUNCTIONS

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