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Altered energy metabolism in Fatal Familial Insomnia cerebral organoids is associated with astrogliosis and neuronal dysfunction

PLoS genetics, 2023-01, Vol.19 (1), p.e1010565-e1010565

2023

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Prion propagation and cellular dysfunction in prion disease: Disconnecting the dots

PLoS pathogens, 2023-10, Vol.19 (10), p.e1011714-e1011714

2023

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Cerebral organoids as a new model for prion disease

PLoS pathogens, 2021-07, Vol.17 (7), p.e1009747-e1009747

2021

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Stress and viral insults do not trigger E200K PrP conversion in human cerebral organoids

PloS one, 2022-10, Vol.17 (10), p.e0277051-e0277051

2022

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Human cerebral organoids as a therapeutic drug screening model for Creutzfeldt–Jakob disease

Scientific reports, 2021-03, Vol.11 (1), p.5165-5165, Article 5165

2021

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Lack of Transmission of Chronic Wasting Disease Prions to Human Cerebral Organoids

Emerging infectious diseases, 2024-06, Vol.30 (6), p.1193-1202

2024

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α-Synuclein seeding activity in duodenum biopsies from Parkinson's disease patients

PLoS pathogens, 2023-06, Vol.19 (6), p.e1011456-e1011456

2023

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Reduced SOD2 expression does not influence prion disease course or pathology in mice

PloS one, 2021-11, Vol.16 (11), p.e0259597-e0259597

2021

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Sporadic Creutzfeldt-Jakob disease prion infection of human cerebral organoids

Acta neuropathologica communications, 2019-06, Vol.7 (1), p.90-90, Article 90

2019

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Hereditary E200K mutation within the prion protein gene alters human iPSC derived cardiomyocyte function

Scientific reports, 2022-09, Vol.12 (1), p.15788-15788, Article 15788

2022

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Using our mini-brains: cerebral organoids as an improved cellular model for human prion disease

Neural regeneration research, 2020-06, Vol.15 (6), p.1019-1020

2020

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The Prion Protein N1 and N2 Cleavage Fragments Bind to Phosphatidylserine and Phosphatidic Acid; Relevance to Stress-Protection Responses

PloS one, 2015-08, Vol.10 (8), p.e0134680-e0134680

2015

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Neuronal maturation reduces the type I IFN response to orthobunyavirus infection and leads to increased apoptosis of human neurons

Journal of neuroinflammation, 2019-11, Vol.16 (1), p.229-229, Article 229

2019

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Neuronal excitatory-to-inhibitory balance is altered in cerebral organoid models of genetic neurological diseases

Molecular brain, 2021-10, Vol.14 (1), p.156-156, Article 156

2021

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Neural cell engraftment therapy for sporadic Creutzfeldt-Jakob disease restores neuroelectrophysiological parameters in a cerebral organoid model

Stem cell research & therapy, 2023-12, Vol.14 (1), p.348-348, Article 348

2023

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Temporary alteration of neuronal network communication is a protective response to redox imbalance that requires GPI-anchored prion protein

Redox biology, 2023-07, Vol.63, p.102733-102733, Article 102733

2023

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Sporadic Creutzfeldt-Jakob disease infected human cerebral organoids retain the original human brain subtype features following transmission to humanized transgenic mice

Acta neuropathologica communications, 2023-02, Vol.11 (1), p.28-28, Article 28

2023

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Neutron Reflectometry Studies Define Prion Protein N-terminal Peptide Membrane Binding

Biophysical journal, 2014-11, Vol.107 (10), p.2313-2324

2014

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Pathogenic Prion Protein Isoforms Are Not Present in Cerebral Organoids Generated from Asymptomatic Donors Carrying the E200K Mutation Associated with Familial Prion Disease

Pathogens (Basel), 2020-06, Vol.9 (6), p.482

2020

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Organoids for modeling prion diseases

Cell and tissue research, 2023-04, Vol.392 (1), p.97-111

2023

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The lymphatic route of TAU

Science translational medicine, 2019-03, Vol.11 (483)

Haigh, Cathryn L.

2019

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Endoproteolytic cleavage as a molecular switch regulating and diversifying prion protein function

Neural regeneration research, 2016-02, Vol.11 (2), p.238-239

2016

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A 3D cell culture approach for studying neuroinflammation

Journal of neuroscience methods, 2021-07, Vol.358, p.109201-109201, Article 109201

2021

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Cellular Analysis of Adult Neural Stem Cells for Investigating Prion Biology

Methods in molecular biology (Clifton, N.J.), 2017, Vol.1658, p.133

Haigh, Cathryn L

2017

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Dominant roles of the polybasic proline motif and copper in the PrP23-89-mediated stress protection response

Journal of cell science, 2009-05, Vol.122 (10), p.1518-1528

2009

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Altered energy metabolism in Fatal Familial Insomnia cerebral organoids is associated with astrogliosis and neuronal dysfunction

Prion propagation and cellular dysfunction in prion disease: Disconnecting the dots

Cerebral organoids as a new model for prion disease

Stress and viral insults do not trigger E200K PrP conversion in human cerebral organoids

Human cerebral organoids as a therapeutic drug screening model for Creutzfeldt–Jakob disease

Lack of Transmission of Chronic Wasting Disease Prions to Human Cerebral Organoids

α-Synuclein seeding activity in duodenum biopsies from Parkinson's disease patients

Reduced SOD2 expression does not influence prion disease course or pathology in mice

Sporadic Creutzfeldt-Jakob disease prion infection of human cerebral organoids

Hereditary E200K mutation within the prion protein gene alters human iPSC derived cardiomyocyte function

Using our mini-brains: cerebral organoids as an improved cellular model for human prion disease

The Prion Protein N1 and N2 Cleavage Fragments Bind to Phosphatidylserine and Phosphatidic Acid; Relevance to Stress-Protection Responses

Neuronal maturation reduces the type I IFN response to orthobunyavirus infection and leads to increased apoptosis of human neurons

Neuronal excitatory-to-inhibitory balance is altered in cerebral organoid models of genetic neurological diseases

Neural cell engraftment therapy for sporadic Creutzfeldt-Jakob disease restores neuroelectrophysiological parameters in a cerebral organoid model

Temporary alteration of neuronal network communication is a protective response to redox imbalance that requires GPI-anchored prion protein

Sporadic Creutzfeldt-Jakob disease infected human cerebral organoids retain the original human brain subtype features following transmission to humanized transgenic mice

Neutron Reflectometry Studies Define Prion Protein N-terminal Peptide Membrane Binding

Pathogenic Prion Protein Isoforms Are Not Present in Cerebral Organoids Generated from Asymptomatic Donors Carrying the E200K Mutation Associated with Familial Prion Disease

Organoids for modeling prion diseases

The lymphatic route of TAU

Endoproteolytic cleavage as a molecular switch regulating and diversifying prion protein function

A 3D cell culture approach for studying neuroinflammation

Cellular Analysis of Adult Neural Stem Cells for Investigating Prion Biology

Dominant roles of the polybasic proline motif and copper in the PrP23-89-mediated stress protection response

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