UB Paderborn / Katalog / Suche / in CDI

1 – 21 von 21

Sortiert nach

Heparan Sulfate in the Developing, Healthy, and Injured Lung

American journal of respiratory cell and molecular biology, 2016-07, Vol.55 (1), p.5-11

2016

Volltextzugriff (PDF)

Redirecting valvular myofibroblasts into dormant fibroblasts through light-mediated reduction in substrate modulus

PloS one, 2012-07, Vol.7 (7), p.e39969

2012

Volltextzugriff (PDF)

Fibroblast Growth Factor Signaling Mediates Pulmonary Endothelial Glycocalyx Reconstitution

American journal of respiratory cell and molecular biology, 2017-06, Vol.56 (6), p.727-737

2017

Volltextzugriff (PDF)

A model-specific role of microRNA-223 as a mediator of kidney injury during experimental sepsis

American journal of physiology. Renal physiology, 2017-08, Vol.313 (2), p.F553-F559

2017

Volltextzugriff (PDF)

Circulating Heparan Sulfate Fragments Attenuate Histone-Induced Lung Injury Independently of Histone Binding

Shock (Augusta, Ga.), 2017-12, Vol.48 (6), p.666-673

2017

Volltextzugriff (PDF)

Loss of Transforming Growth Factor Beta Type II Receptor Increases Aggressive Tumor Behavior and Reduces Survival in Lung Adenocarcinoma and Squamous Cell Carcinoma

Clinical cancer research, 2012-04, Vol.18 (8), p.2173-2183

2012

Volltextzugriff (PDF)

More than a biomarker: the systemic consequences of heparan sulfate fragments released during endothelial surface layer degradation (2017 Grover Conference Series)

Pulmonary Circulation, 2018-01, Vol.8 (1), p.1-10

2018

Volltextzugriff (PDF)

Circulating heparin oligosaccharides rapidly target the hippocampus in sepsis, potentially impacting cognitive functions

Proceedings of the National Academy of Sciences - PNAS, 2019-05, Vol.116 (19), p.9208-9213

2019

Volltextzugriff (PDF)

Epithelial Heparan Sulfate Contributes to Alveolar Barrier Function and Is Shed during Lung Injury

American journal of respiratory cell and molecular biology, 2018-09, Vol.59 (3), p.363-374

2018

Volltextzugriff (PDF)

Smad4 loss promotes lung cancer formation but increases sensitivity to DNA topoisomerase inhibitors

Oncogene, 2016-02, Vol.35 (5), p.577-586

2016

Volltextzugriff (PDF)

Loss of endothelial sulfatase-1 after experimental sepsis attenuates subsequent pulmonary inflammatory responses

American journal of physiology. Lung cellular and molecular physiology, 2019-11, Vol.317 (5), p.L667-L677

2019

Volltextzugriff (PDF)

Hepcidin Removal during Continuous Renal Replacement Therapy

Blood purification, 2024, Vol.53 (1), p.23

2024

Volltextzugriff (PDF)

Epidermal Smad4 Deletion Results in Aberrant Wound Healing

The American journal of pathology, 2010, Vol.176 (1), p.122-133

2010

Volltextzugriff (PDF)

Role of PTEN in basal cell derived lung carcinogenesis

Molecular carcinogenesis, 2014-10, Vol.53 (10), p.841-846

2014

Volltextzugriff (PDF)

Post‐septic Down‐regulation of Sulfatase‐1 Suppresses Pulmonary Endothelial ICAM‐1 Expression

The FASEB journal, 2018-04, Vol.32 (S1), p.706.7-706.7

2018

Volltextzugriff (PDF)

Transforming Growth Factor-β Receptor Type II Deletion Increases the Multiplicity and Growth of K-ras–induced Lung Cancers

Proceedings of the American Thoracic Society, 2012-05, Vol.9 (2), p.80-81

2012

Volltextzugriff (PDF)

Ran-dependent docking of importin-beta to RanBP2/Nup358 filaments is essential for protein import and cell viability

The Journal of cell biology, 2011-08, Vol.194 (4), p.597-612

2011

Volltextzugriff (PDF)

Control of neural cell composition in poly(ethylene glycol) hydrogel culture with soluble factors

Tissue engineering. Part A, 2011-11, Vol.17 (21-22), p.2805

2011

Volltextzugriff (PDF)

Psychosoziale Belastungen während Corona: Ergebnisse eines Scoping Reviews zu Familien mit Kindern zwischen 0 und 5 Jahren

Prävention und Gesundheitsförderung, 2024-02

2024

Volltextzugriff (PDF)

Ran-dependent docking of importin-[Beta] to RanBP2/Nup358 filaments is essential for protein import and cell viability

The Journal of cell biology, 2011-08, Vol.194 (4), p.597

2011

Volltextzugriff (PDF)

1 – 21 von 21

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(19)
Mit Volltextzugriff
(19)

Publikationsform

Artikel
(21)

Sprache

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Heparan Sulfate in the Developing, Healthy, and Injured Lung

Redirecting valvular myofibroblasts into dormant fibroblasts through light-mediated reduction in substrate modulus

Fibroblast Growth Factor Signaling Mediates Pulmonary Endothelial Glycocalyx Reconstitution

A model-specific role of microRNA-223 as a mediator of kidney injury during experimental sepsis

Circulating Heparan Sulfate Fragments Attenuate Histone-Induced Lung Injury Independently of Histone Binding

Loss of Transforming Growth Factor Beta Type II Receptor Increases Aggressive Tumor Behavior and Reduces Survival in Lung Adenocarcinoma and Squamous Cell Carcinoma

More than a biomarker: the systemic consequences of heparan sulfate fragments released during endothelial surface layer degradation (2017 Grover Conference Series)

Circulating heparin oligosaccharides rapidly target the hippocampus in sepsis, potentially impacting cognitive functions

Epithelial Heparan Sulfate Contributes to Alveolar Barrier Function and Is Shed during Lung Injury

Smad4 loss promotes lung cancer formation but increases sensitivity to DNA topoisomerase inhibitors

Loss of endothelial sulfatase-1 after experimental sepsis attenuates subsequent pulmonary inflammatory responses

Hepcidin Removal during Continuous Renal Replacement Therapy

Epidermal Smad4 Deletion Results in Aberrant Wound Healing

Alveolar epithelial glycocalyx degradation mediates surfactant dysfunction and contributes to acute respiratory distress syndrome

Role of PTEN in basal cell derived lung carcinogenesis

Post‐septic Down‐regulation of Sulfatase‐1 Suppresses Pulmonary Endothelial ICAM‐1 Expression

Transforming Growth Factor-β Receptor Type II Deletion Increases the Multiplicity and Growth of K-ras–induced Lung Cancers

Ran-dependent docking of importin-beta to RanBP2/Nup358 filaments is essential for protein import and cell viability

Control of neural cell composition in poly(ethylene glycol) hydrogel culture with soluble factors

Psychosoziale Belastungen während Corona: Ergebnisse eines Scoping Reviews zu Familien mit Kindern zwischen 0 und 5 Jahren

Ran-dependent docking of importin-[Beta] to RanBP2/Nup358 filaments is essential for protein import and cell viability

Suchergebnisse filtern