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Redirecting valvular myofibroblasts into dormant fibroblasts through light-mediated reduction in substrate modulus

PloS one, 2012-07, Vol.7 (7), p.e39969

2012

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Heparan Sulfate in the Developing, Healthy, and Injured Lung

American journal of respiratory cell and molecular biology, 2016-07, Vol.55 (1), p.5-11

2016

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Fibroblast Growth Factor Signaling Mediates Pulmonary Endothelial Glycocalyx Reconstitution

American journal of respiratory cell and molecular biology, 2017-06, Vol.56 (6), p.727-737

2017

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Circulating heparin oligosaccharides rapidly target the hippocampus in sepsis, potentially impacting cognitive functions

Proceedings of the National Academy of Sciences - PNAS, 2019-05, Vol.116 (19), p.9208-9213

2019

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Loss of endothelial sulfatase-1 after experimental sepsis attenuates subsequent pulmonary inflammatory responses

American journal of physiology. Lung cellular and molecular physiology, 2019-11, Vol.317 (5), p.L667-L677

2019

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A model-specific role of microRNA-223 as a mediator of kidney injury during experimental sepsis

American journal of physiology. Renal physiology, 2017-08, Vol.313 (2), p.F553-F559

2017

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Epithelial Heparan Sulfate Contributes to Alveolar Barrier Function and Is Shed during Lung Injury

American journal of respiratory cell and molecular biology, 2018-09, Vol.59 (3), p.363-374

2018

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Smad4 loss promotes lung cancer formation but increases sensitivity to DNA topoisomerase inhibitors

Oncogene, 2016-02, Vol.35 (5), p.577-586

2016

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Circulating Heparan Sulfate Fragments Attenuate Histone-Induced Lung Injury Independently of Histone Binding

Shock (Augusta, Ga.), 2017-12, Vol.48 (6), p.666-673

2017

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Hepcidin Removal during Continuous Renal Replacement Therapy

Blood purification, 2024, Vol.53 (1), p.23

2024

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Loss of Transforming Growth Factor Beta Type II Receptor Increases Aggressive Tumor Behavior and Reduces Survival in Lung Adenocarcinoma and Squamous Cell Carcinoma

Clinical cancer research, 2012-04, Vol.18 (8), p.2173-2183

2012

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Epidermal Smad4 Deletion Results in Aberrant Wound Healing

The American journal of pathology, 2010, Vol.176 (1), p.122-133

2010

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More than a biomarker: the systemic consequences of heparan sulfate fragments released during endothelial surface layer degradation (2017 Grover Conference Series)

Pulmonary Circulation, 2018-01, Vol.8 (1), p.1-10

2018

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Hyponatremia vs. Pseudohyponatremia: Sodium Measurement in a Patient With a Hematologic Malignancy: SA-PO475

Journal of the American Society of Nephrology, 2022-11, Vol.33 (11S), p.737-738

2022

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Role of PTEN in basal cell derived lung carcinogenesis

Molecular carcinogenesis, 2014-10, Vol.53 (10), p.841-846

2014

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Post‐septic Down‐regulation of Sulfatase‐1 Suppresses Pulmonary Endothelial ICAM‐1 Expression

The FASEB journal, 2018-04, Vol.32 (S1), p.706.7-706.7

2018

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Experimental Degradation of the Alveolar Epithelial Glycocalyx Decreases Lung Compliance in Young and Old Mice, Potentially by Inducing Surfactant Dysfunction

The FASEB journal, 2021-05, Vol.35 (S1), p.n/a, Article fasebj.2021.35.S1.02042

2021

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Heparan Sulfate is Shed into the Alveolus after Intratracheal Lung Injury and Facilitates Alveolar Epithelial Growth Factor Signaling and Lung Repair

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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Heparanase contributes to sepsis‐associated encephalopathy and neurovascular glycocalyx degradation

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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Compartment‐Specific Effects of Alveolar Heparan Suflate Shedding after Intratracheal Lipopolysaccharide

The FASEB journal, 2017-04, Vol.31 (S1)

2017

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Circulating Endothelial Glycocalyx Fragments Impact Endothelial and Epithelial Repair after Septic Lung Injury

The FASEB journal, 2015-04, Vol.29 (S1)

2015

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HS‐FGF2‐FGFR1 signaling contributes to pulmonary endothelial glycocalyx reconstitution

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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Alveolar Epithelial Heparan Sulfate and Chondroitin Sulfate in the Healthy and Injured Lung

Haeger, Sarah Marie

2018

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Control of neural cell composition in poly(ethylene glycol) hydrogel culture with soluble factors

Tissue engineering. Part A, 2011-11, Vol.17 (21-22), p.2805

2011

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Redirecting valvular myofibroblasts into dormant fibroblasts through light-mediated reduction in substrate modulus

Heparan Sulfate in the Developing, Healthy, and Injured Lung

Fibroblast Growth Factor Signaling Mediates Pulmonary Endothelial Glycocalyx Reconstitution

Circulating heparin oligosaccharides rapidly target the hippocampus in sepsis, potentially impacting cognitive functions

Loss of endothelial sulfatase-1 after experimental sepsis attenuates subsequent pulmonary inflammatory responses

A model-specific role of microRNA-223 as a mediator of kidney injury during experimental sepsis

Epithelial Heparan Sulfate Contributes to Alveolar Barrier Function and Is Shed during Lung Injury

Smad4 loss promotes lung cancer formation but increases sensitivity to DNA topoisomerase inhibitors

Circulating Heparan Sulfate Fragments Attenuate Histone-Induced Lung Injury Independently of Histone Binding

Hepcidin Removal during Continuous Renal Replacement Therapy

Loss of Transforming Growth Factor Beta Type II Receptor Increases Aggressive Tumor Behavior and Reduces Survival in Lung Adenocarcinoma and Squamous Cell Carcinoma

Epidermal Smad4 Deletion Results in Aberrant Wound Healing

More than a biomarker: the systemic consequences of heparan sulfate fragments released during endothelial surface layer degradation (2017 Grover Conference Series)

Hyponatremia vs. Pseudohyponatremia: Sodium Measurement in a Patient With a Hematologic Malignancy: SA-PO475

Role of PTEN in basal cell derived lung carcinogenesis

Alveolar epithelial glycocalyx degradation mediates surfactant dysfunction and contributes to acute respiratory distress syndrome

Post‐septic Down‐regulation of Sulfatase‐1 Suppresses Pulmonary Endothelial ICAM‐1 Expression

Experimental Degradation of the Alveolar Epithelial Glycocalyx Decreases Lung Compliance in Young and Old Mice, Potentially by Inducing Surfactant Dysfunction

Heparan Sulfate is Shed into the Alveolus after Intratracheal Lung Injury and Facilitates Alveolar Epithelial Growth Factor Signaling and Lung Repair

Heparanase contributes to sepsis‐associated encephalopathy and neurovascular glycocalyx degradation

Compartment‐Specific Effects of Alveolar Heparan Suflate Shedding after Intratracheal Lipopolysaccharide

Circulating Endothelial Glycocalyx Fragments Impact Endothelial and Epithelial Repair after Septic Lung Injury

HS‐FGF2‐FGFR1 signaling contributes to pulmonary endothelial glycocalyx reconstitution

Alveolar Epithelial Heparan Sulfate and Chondroitin Sulfate in the Healthy and Injured Lung

Control of neural cell composition in poly(ethylene glycol) hydrogel culture with soluble factors

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