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Flow Cytometric Analysis of Myeloid Cells in Human Blood, Bronchoalveolar Lavage, and Lung Tissues

American journal of respiratory cell and molecular biology, 2016-01, Vol.54 (1), p.13-24

2016

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Mechanical Stretch Induces Epithelial-Mesenchymal Transition in Alveolar Epithelia via Hyaluronan Activation of Innate Immunity

The Journal of biological chemistry, 2011-05, Vol.286 (20), p.17435-17444

2011

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Case series of acute appendicitis association with SARS-CoV-2 infection

BMC infectious diseases, 2021-02, Vol.21 (1), p.217-4, Article 217

2021

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Hyaluronan signaling during ozone-induced lung injury requires TLR4, MyD88, and TIRAP

PloS one, 2011-11, Vol.6 (11), p.e27137

2011

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The Role of Toll-like Receptor 4 in Environmental Airway Injury in Mice

American journal of respiratory and critical care medicine, 2004-07, Vol.170 (2), p.126-132

2004

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Hyaluronan Mediates Ozone-induced Airway Hyperresponsiveness in Mice

The Journal of biological chemistry, 2009-04, Vol.284 (17), p.11309-11317

2009

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Surfactant protein-A suppresses eosinophil-mediated killing of Mycoplasma pneumoniae in allergic lungs

PloS one, 2012-02, Vol.7 (2), p.e32436-e32436

2012

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Genetic variation in SP-A2 leads to differential binding to Mycoplasma pneumoniae membranes and regulation of host responses

The Journal of immunology (1950), 2015-06, Vol.194 (12), p.6123-6132

2015

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Ozone primes alveolar macrophage–derived innate immunity in healthy human subjects

Journal of allergy and clinical immunology, 2016-10, Vol.138 (4), p.1213-1215.e1

2016

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Clara Cells Attenuate the Inflammatory Response through Regulation of Macrophage Behavior

American journal of respiratory cell and molecular biology, 2010-02, Vol.42 (2), p.161-171

2010

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Differential regulation of the PGC family of genes in a mouse model of Staphylococcus aureus sepsis

PloS one, 2010-07, Vol.5 (7), p.e11606-e11606

2010

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Gene Expression Profiling of Familial and Sporadic Interstitial Pneumonia

American journal of respiratory and critical care medicine, 2007-01, Vol.175 (1), p.45-54

2007

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Retraction: Innate Immune Activation by Inhaled Lipopolysaccharide, Independent of Oxidative Stress, Exacerbates Silica-Induced Pulmonary Fibrosis in Mice

PloS one, 2016-05, Vol.11 (5), p.e0155388-e0155388

2016

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A toll-like receptor 2 pathway regulates the Ppargc1a/b metabolic co-activators in mice with Staphylococcal aureus sepsis

PloS one, 2011-09, Vol.6 (9), p.e25249-e25249

2011

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The effect of toll-like receptors and toll-like receptor genetics in human disease

Annual review of medicine, 2008-01, Vol.59 (1), p.343-359

2008

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Hyaluronan fragments contribute to the ozone-primed immune response to lipopolysaccharide

The Journal of immunology (1950), 2010-12, Vol.185 (11), p.6891-6898

2010

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Ambient Ozone Primes Pulmonary Innate Immunity in Mice

Journal of Immunology, 2007-10, Vol.179 (7), p.4367-4375

2007

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Innate immune activation by inhaled lipopolysaccharide, independent of oxidative stress, exacerbates silica-induced pulmonary fibrosis in mice

PloS one, 2012-07, Vol.7 (7), p.e40789

2012

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TLR4 Signaling Attenuates Ongoing Allergic Inflammation

Journal of Immunology, 2006-05, Vol.176 (10), p.5856-5862

2006

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In utero supplementation with methyl donors enhances allergic airway disease in mice

The Journal of clinical investigation, 2008-10, Vol.118 (10), p.3462-3469

2008

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Retraction: Hyaluronan activation of the Nlrp3 inflammasome contributes to the development of airway hyperresponsiveness

Environmental health perspectives, 2015-07, Vol.123 (7), p.A172-A172

Hollingsworth, John W

2015

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Comment on Expression of Concern: TLR4 Is Necessary for Hyaluronan-mediated Airway Hyperresponsiveness after Ozone Inhalation

American journal of respiratory and critical care medicine, 2017-07, Vol.196 (2), p.249-250

2017

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The Critical Role of Hematopoietic Cells in Lipopolysaccharide-induced Airway Inflammation

American journal of respiratory and critical care medicine, 2005-04, Vol.171 (8), p.806-813

2005

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Ozone activates pulmonary dendritic cells and promotes allergic sensitization through a Toll-like receptor 4–dependent mechanism

Journal of allergy and clinical immunology, 2010-05, Vol.125 (5), p.1167-1170

2010

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Flow Cytometric Analysis of Myeloid Cells in Human Blood, Bronchoalveolar Lavage, and Lung Tissues

Mechanical Stretch Induces Epithelial-Mesenchymal Transition in Alveolar Epithelia via Hyaluronan Activation of Innate Immunity

TLR5 participates in the TLR4 receptor complex and promotes MyD88-dependent signaling in environmental lung injury

Case series of acute appendicitis association with SARS-CoV-2 infection

Hyaluronan signaling during ozone-induced lung injury requires TLR4, MyD88, and TIRAP

The Role of Toll-like Receptor 4 in Environmental Airway Injury in Mice

Hyaluronan Mediates Ozone-induced Airway Hyperresponsiveness in Mice

Surfactant protein-A suppresses eosinophil-mediated killing of Mycoplasma pneumoniae in allergic lungs

Genetic variation in SP-A2 leads to differential binding to Mycoplasma pneumoniae membranes and regulation of host responses

Ozone primes alveolar macrophage–derived innate immunity in healthy human subjects

Clara Cells Attenuate the Inflammatory Response through Regulation of Macrophage Behavior

Differential regulation of the PGC family of genes in a mouse model of Staphylococcus aureus sepsis

Gene Expression Profiling of Familial and Sporadic Interstitial Pneumonia

Retraction: Innate Immune Activation by Inhaled Lipopolysaccharide, Independent of Oxidative Stress, Exacerbates Silica-Induced Pulmonary Fibrosis in Mice

A toll-like receptor 2 pathway regulates the Ppargc1a/b metabolic co-activators in mice with Staphylococcal aureus sepsis

The effect of toll-like receptors and toll-like receptor genetics in human disease

Hyaluronan fragments contribute to the ozone-primed immune response to lipopolysaccharide

Ambient Ozone Primes Pulmonary Innate Immunity in Mice

Innate immune activation by inhaled lipopolysaccharide, independent of oxidative stress, exacerbates silica-induced pulmonary fibrosis in mice

TLR4 Signaling Attenuates Ongoing Allergic Inflammation

In utero supplementation with methyl donors enhances allergic airway disease in mice

Retraction: Hyaluronan activation of the Nlrp3 inflammasome contributes to the development of airway hyperresponsiveness

Comment on Expression of Concern: TLR4 Is Necessary for Hyaluronan-mediated Airway Hyperresponsiveness after Ozone Inhalation

The Critical Role of Hematopoietic Cells in Lipopolysaccharide-induced Airway Inflammation

Ozone activates pulmonary dendritic cells and promotes allergic sensitization through a Toll-like receptor 4–dependent mechanism

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